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At first, a model of a pair of excitable neurons coupled via gap junction is built by Chay model, the effect of the strength of gap junction on the synchronous oscillation is studied;then, the synchronous oscillations of a 2-D network are also studied;at last, the Lyapunov exponent and phase portrait are utilized in the analysis of nonlinear, the approximate entropy is utilized to measure the complexity.

首先以Chay模型为基础建立了两个神经元电突触耦合模型,研究了不同电突触耦合强度对两个神经元同步振荡的影响;其次,研究了可兴奋神经元组成的二维网络的同步振荡;最后,应用李雅普诺夫指数和相图分析了同步振荡的非线性,应用近似熵分析了同步振荡的复杂性。

Computer image analysis was used to measure the average optical density of synaptophysin.

方法采用免疫组织化学方法,观察突触素在不同周龄的人胎儿海马中的表达水平,利用计算机图像分析技术测量不同周龄阶段海马突触素表达的平均光密度。

In recent years,along with the research technology and experimental condition\'s development,researchers overseas got certain achievement in form,molecular dissection,electricity physiology etc in normal physiology condition which enables us to research ribbon synapses\' structure and the function under the pathological state.

近年来随着研究技术和实验条件的发展,国外的研究者对内毛细胞带状突触正常生理条件下的形态、分子解剖、电生理等方面的研究取得了一定进展,使我们得以对病理状态下的内毛细胞带状突触的结构和功能进行研究。

Results: Among 32 tested MNs, depolarizing responses(excitatory postsynaptic potential, EPSP) were elicited by cVLF stimulation in 21 MNs, hyperpolarizing response (inhibitory postsynaptic potential, IPSP) in 1 MN, and IPSP preceded by EPSP in 4 MNs. The cVLFEPSPs were stimulus intensity-dependent, and could be abolished by low Ca(superscript 2+)/high Mg(superscript 2+) solution. compared with iVLF-EPSPs, cVLF-EPSPs showed longer latency (P.001). The cVLF-IPSP presented with membrane potential-dependent property and was eliminated by the perfusion of picrotoxin (30 μmol/L) and strychnine (1.0 μmol/L).

结果:在32个测试的MNs,观察到cVLF电刺激可在21个MNs上诱发去极化反应(即cVLF性兴奋性突触后电位,cVLF-EPSP),在1个MN上诱发超极化反应(即cVLF性抑制性突触后电位,cVLF-IPSP),在4个MNs上诱发cVLF-EPSP后复合有cVLF-IPSP的反应。cVLF-EPSP具有刺激强度依赖性、被低钙高镁溶液取消的特性,与i VLF性EPSP相比,有潜伏期较长的特点(P.001)。cVLF-IPSP呈膜电位依赖性,并被印防己毒素(30μmol/L)及士的宁(1.0μmol/L)取消。

Propofol induced Cl- currents at concentrations of 10-5 and10-4 M, which were sensitive to picrotoxin and, to a lesserextent, to strychnine. Propofol (10-6 M) enhanced -aminobutyricacidA (GABAA; 10-6 M)-induced current synergistically. Moreover,propofol (10-5 and 10-4 M) significantly increased the decaytime of evoked-inhibitory postsynaptic currents, which suggestsa postsynaptic modulation of GABAA receptors.

异丙酚在浓度10-5~10-4M 时,産生 Cl-电流,对印防己毒素敏感,在更小浓度范围内,对番木鼈碱敏感。10-6M 的异丙酚加强 GABAA 産生的电流,另外,异丙酚(10-5、10-4M )显著地增加了突触后诱发-抑制电流的衰退时间,提示了 GABAA 受体的突触后调节作用。

Here, temporal synaptic stability and the functional consequences for presynaptic operation will be considered.

应当考虑突触前的手术对临时突触的稳定性和相应功能的影响。

In terms of expression site, synaptic plasticity can be divided into presynaptic and postsynaptic.

突触前可塑性是指通过对神经递质释放过程的干预、修饰,调节突触强度的过程。

The results showed that GABA was the excitatory transmitter of presynaptic inhibition.

以上结果表明:GABA是突触前抑制环路中,最后一级突触的兴奋性递质。

The release is triggered by Ca〓 and completed by the fusion of neurotransmitter containing synaptic vesicles with the presynaptic membrane.

神经递质的释放是由钙引发的,由含神经递质的突触囊泡与突触前膜融合完成的。

Maturation of presynaptic function, assayed with endocytotic markers, followed accumulation of synapsin I.

成熟的前突触,以内细胞标记技术,显示累积的突触素I。

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