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The FM imaging technique is an advanced and effective method for analyzing synaptic vesicle exocytosis and neurotransmitter release, and can be applied in various synaptic functional studies.

FM 1-43成像技术能够检测钙离子介导的神经递质的释放过程,是一种非常先进并切实有效的检测神经元突触末端神经递质释放过程的手段和方法。

Objective:To investigate the neurochemical mechanisms responsible for learning dependent long-term potentiation,the effect of CNQX on extracellular levels of glutamatewas measured in hippocampal dentate gyrusregion during the period of establishment and extinction of the conditioned reflex in free-moving conscious rats.

目的:观察条件反射的建立和消退过程中,ɑ-氨基羟甲基恶唑丙酸受体拮抗剂对自由活动大鼠海马齿状回细胞外液中Glu浓度的影响,并进一步探讨它们与习得性突触效应长时程增强和行为学习之间的关系。

The data showed that there was no difference in input-out curve and LTD between the two genotypes.

在本部分实验中,主要研究行为学改变的突触可塑性机制。

These data indicate that cortical neurons remain responsiveto synaptic input during isoflurane and halothane anesthesia.

这些数据显示皮层神经元在异氟烷和氟烷麻醉时对突触输入仍有反应。

Furthermore, marked neuronal degeneration and death occurred both in KIF 1A mutant mice and in cultures of mutant neurons.

在神经系统,突触小泡前体的运输水平大大地下降了,另外,在KIF1A基因剔除的小鼠及神经细胞培养中,均出现明显的神经元退行性改变及死亡现象。

Axonal degeneration similar to that observed in KIF 1A mutants has also been reported for several neurodegenerative diseases such as senile dementia and most recently Charcot-Marie-Tooth disease type 2A by another synaptic vesicle precursors transporter KIF1B knockout mice. In some neurodegenerative diseases, neuronal cell death caused by defects in the transport of synaptic vesicle precursors by KIF1A may be involved.

和KIF1A基因剔除鼠相似的轴突变性在一些神经退行性疾病如老年性痴呆的研究中也有发现,特别是最近,与KIF1A一样运输突触小泡前体的KIF1B基因剔除鼠与Charcot-Marie-Tooth二型疾病的关系,更提示了KIF1A基因的功能缺失,与神经退行性病变及神经元死亡的发生有不可分割的关系。

Theoretically, through blocking presynaptic serotonin reuptake, meperidine may induce or interact with other serotonergic agents and result in serotonin hyperstimulation.

理论上,透过阻止於突触间隙前的血清素再吸收,meperidine可能诱发或与其他促血清素药物互动导致血清素超量刺激。

Cocaine and amphetamines are powerful releasers of monoamines into the synapse as well as inhibitors of reuptake.

可卡因和苯乙丙胺强烈释放单胺进入突触同时抑制再摄取。

Increased levels of monoamines in the synapse affect the hypothalamic–pituitary–adrenal axis 56 and reverse some of the long-term effects of stress.

突触内单胺水平上升,影响下丘脑-垂体-肾上腺轴,逆转一些长期的紧张效应。

Both induced a series of physiological events such as increment of monoamines, PKC activity, synaptic effecacy and structureal plasticity, All of these events will finally lead to formation of LTP and facilitation of memory.

提示黄皮酰胺可促进突触形态可塑性的形成。

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She gently rebuff ed him, but agreed that they could be friends

她婉言拒绝了,但同意作为朋友相处。

If in the penal farm, you were sure to be criticized.

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