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Our work is concentrated on the possible mechanism of the formation and development of short-term plasticity of thalamocortical visual pathway in rats.

本工作对大鼠视通路短时程突触可塑性的形成机制和发育机制进行了研究。

Similar as in major thalamocortical visual pathway, short-term synaptic depression was also prominent in extrageniculate pathway.

与丘脑皮层主要视觉通路相似,膝体外通路也具有显著的短时程突触抑制特性。

The thalamocortical connection, which is mainly involved in mediation of upward information flow across layers II-IV, was also characterized by ES at layer IV.

首先,丘脑皮层投射的激活可以诱发更多的有效场电位,并且与S1区更多的神经元建立突触联系。

However in the preparation of ganglion-body wall, when the mechanical'toucher'was still on the body wall, the high frequence activity of AP eells could make the T and P cells discharge, but no synaptic potential could be found in the T and P cells at the same time. When the contralatcral root was cut, this kind of effects disappeared.

然而,在体壁-神经节标本上,当机械刺激物仍接触表皮时,AP细胞的高频发放使T和P细胞产生动作电位,但此时在T和P细胞上未见有突触后效应,切断神经根这种影响消失。

Suprachiasmatic nucleus; Synapse ; Subjunctional body; 5-HT ; Degeneration ; Ultrastructure ; Immunoelectromicroscope ; Tupaia belangeri chinensis

视交叉上核;突触节下小体; 5-羟色胺;溃变;超微结构;免疫电镜;树鼠句

METHODS: The maximum number of platelet binding sites and their affinity for [3H]-rauwolscine, a selectie alpha 2-AR antagonist, were measured in unmedicated and medicated major depressed patients and in normal olunteers.

背景:曾认为抑郁症可以伴随中枢α2肾上腺素受体密度的降低,继发突触前儿茶酚胺神经元负性反馈的损害,导致刺激后去甲肾上腺素输出和释放去抑制功能的减弱。

ResultsThere was no expression of Caspase-3 in group A. The expression of Caspase-3 in group C was decreased compared with that in group B(P<0.01. In groups B and C, expansion and vacuolization of mitochondria, swollen neurons and apoptotic bodies appeared, which suggested that WBH could induce neuron injury, and the neuron injury in group C was more severe than that in group B.

结果A组大鼠海马区神经元无Caspase-3表达,B、C两组大鼠海马区神经元有Caspase-3表达,B组较C组Caspase-3表达平均灰度值减小。B、C两组大鼠海马神经元超微结构发生改变、细胞器水肿,部分线粒体空化,内质网稀疏,突触前膜、后膜结构不完整,可见自噬体结构;B组大鼠的神经元超微结构损害较C组轻。

Post mortem studies of suicide victims have shown a diffuse reduction in serotonin receptor transporter sites in the ventromedial PFC, and an increase in postsynaptic 5-HT (1A) receptors in localized regions of the PFC.

自杀死亡的尸检研究发现,腹内侧 PFC 的 5-HT 受体转运体位点普遍减少,并且 PFC 的部分区域突触后 5-HT (1A)受体数增加。

BACKGROUND: We studied the effects of isoflurane on hippocampalsynaptic transmission and paired-pulse plasticity, under invivo intact interneuron circuitry.

背景:作者在体研究具有完整的中间神经电路中异氟醚对海马突触传递和双脉冲可塑性的影响。

Chronic psychological stress can increase GC in Plasma and brain by HPAA. There are plentiful GR on Hippocam--pal cell membranes. It promote presynapitic membrane assoiling EAAs when GC combines with GR.

心理应激通过 HPAA 使血浆及脑组织中的 GC 升高,海马神经元细胞膜上存在丰富的 GR , GC 与 GR 结合后,可促进神经元突触前膜释放兴奋性氨基酸。

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