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This paper reviews the GluR2-lacking AMPARs and their roles in the synaptic function and plasticity.

本文对GluR2缺失的AMPARs及其对突触功能和可塑性的作用作一综述。

Results: The model illuminated the influence to the change of synaptic plasticity.

结果:该数学模型深刻阐述了Ca^2+浓度变化对突触可塑性的影响。

People who seem to have a good ability to talk to spirit, actually have an ability to quickly build new synaptic pathways.

似乎有很好的能力与精神体谈话的人们,实际上是有一种能力能够很快地建立新的突触小路。

They are also looking at the number of synaptic connections between nerve cells in the two groups, as this could also provide useful information.

他们还研究了神经细胞间突触连接的数量,这也会提供有用的信息。

These results indicate that spike trains with various temporal structures will be synaptically transmitted between primary afferent fiber and spinal dorsal horn neuron.

这些结果表明具有各种时间结构的动作电位序列会在初级传入纤维与脊髓背角神经元之间进行突触传递。

In ER-containing spines, we frequently observed synaptically triggered calcium release events of very large amplitudes.

当神经细胞受到刺激时,含有内质网的棘会释放大量的钙,从而引发突触功能的变化。

METHODS: In rat hippocampal slices, whole cell patch clamprecordings were used to study anesthetic effects on CA1 neuronintrinsic excitability, and population spike recordings wereused to investigate effects on synaptically evoked discharge.

在大鼠海马脑片上采用全细胞膜片钳记录来研究麻醉药对CA1神经元自发兴奋性的作用;采用群峰电位记录来研究对突触诱发放电的作用。

Key words Voltage-dependence miniature inhibitory postsynaptic current tectal neuron slice depolarization membrane potential Xenopus.

电压依赖性;微突触后电流;视顶盖神经元;脑片;去极化;膜电位;非洲爪蟾

Visual responses of 80% tectal cells were reversibly blocked by NMDA antagonist CPP, and those of 20% cells by muscarinic antagonist atropine, suggesting that retinotectal pathway in toads may contain two groups of optic fibers that use glutamate and acetylcholine as transmitters, respectively.

视网膜-顶盖是视觉信息进入脑的第一站,但是对两栖类视网膜-顶盖投射所用递质却长期含混不清;另一方面,峡核是次级视觉中枢,与顶盖间存在着密切的神经联系,虽有实验对峡核-顶盖投射的突触连接性质进行了研究,但却未见峡核-顶盖投射所用递质方面的报道。

With regard to the second hypothesis, a great deal of evidence has been collcted to support the idea thad PAD is mediated by gamma-aminobutyric acid.

目前公认:r-氨基丁酸是引起粗纤维突触前抑制的主要递质。

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