突触

Deltamethrin was found to affect ATPase activity and protein phosphorylation of housefly brain synaptosome in vitro .

为材料，研究和比较了神经毒剂溴氰菊酯对其脑突触体蛋白磷酸化作用的影响。

The increased expression of NMDA-R1 plays an important role in the formation of visceral hypersensitivity.

内脏高敏感的形成与突触后NMDA-R1的高表达密切相关。

By using a carefully optimized form of iontophoresis, we are able to deliver specified profiles of neurotransmitter directly to single synapses.

并经由谨慎的利用离子电泳法，我们可以直接传送特定型式的神经传导物质到单一突触。

This in vitro study on toads provides the first electrophysiological evidence that electrical stimulation of the decussatio Veli in the midbrain tegmentum predominantly produced inhibition on neurons extracellularly recorded from the nucleus isthmi .

本文用离体脑片的方法直接研究了脑脚核团-峡核投射的突触连接性质，第一次证明，电刺激中华大蟾蜍中脑Veli交叉，在峡核内胞外记录，大多数细胞呈抑制反应，少数细胞呈兴奋反应。

The change of synaptophysin resulted from kindling, it also could result in the molecular elements of kindling maintenance.

突触体素含量的变化一方面是点燃的结果，另一方面也可能是导致点燃维持的分子学基础。

Objective To know the ultrastructure and function of synapse in mammal's medulla oblongata megacell reticular nucleus.

目的探讨哺乳动物延髓巨细胞网状核内突触的超微结构及其功能。

Our data has elucidated the mechanism of the modulatory effect of extracellular ATP (and its catabolic product adenosine) on synaptic transmission in the central nervous system.

我们以上的工作，不仅阐明了细胞外ATP对中枢神经系统神经元间突触传递的调控作用的机理，而且证明存在内源性ATP的紧张性调控作用。

How synapses are molecularly constructed is a fascinating question, which attracted scientists' attention for many decades.

突触的分子构建是一个引人入胜的问题，数十年来一直吸引着科学家们的注意。

Neurotrophin-3/neurotrophic factor 3 is the important member of neurotrophin family, which can prevent embryonic and postnatal motoneuron death, maintain neuron survival, promote reparation and regeneration after nervous injury, and regulate synapse activity of neuron.

神经元营养因子-3 （neurotrophin-3， NT-3），是神经营养因子家族的重要成员，具有阻止胚胎期及出生后运动神经元细胞死亡，维持神经元的存活及促进神经损伤后的修复与再生，并参与调节神经元突触活动的作用。

OLIG2 is a transcription factor which could regulate the transcription of many oligodendrocyte/myelination related genes, and its expression level also was down-regulated in schizophrenia subjects. Therefore, OLIG2 is mostly like to be a risk gene of schizophrenia.

OLIG2是一个能够调控下游许多与少突触细胞/髓鞘发生相关的基因表达的转录因子，且OLIG2的表达水平在精神分裂症患者体内也明显下降，因此，OLIG2极有可能是精神分裂症的致病基因。

She gently rebuff ed him, but agreed that they could be friends

她婉言拒绝了，但同意作为朋友相处。

If in the penal farm, you were sure to be criticized.

要是在劳改农场，你等着挨绳子吧！