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However, the advance of intracellular labeling techniques enables us not only to visualize more complete dendritic arbor for qualitative ahalysis, but also to examine the relation between changes in the dendritic arborization and the evoked fast postsynaptic curents in the same neurons during the postnatal development.

但是,细胞内染色技术的进步使我们不仅能观察到更完整的树突分支来用于定性研究,而且也可以在同一神经元上研究在发育过程中树突分支的变化与诱发的快突触后电流(fast postsynaptic currents,fPSCs)之间的关系。

Tetanic synaptic stimulation induced a rapid delivery of tagged receptors into dendritic spines as well as clusters in dendrites.

强的突触刺激诱导标记受体迅速转移到树突棘和树突簇上。

Furthermore, we found synaptic structures near the neuron body, which is the evidence of connection between axons and dendrites.

进一步的观察中还看到了神经元胞体附近的突触结构,这是图案化网络中的神经元之间通过轴突和树突建立了相互联系的重要证据。

The stage of neural development during which axons and dendrites grow and establish synaptic contacts.

轴突和树突成长与建立突触连接的神经发展阶段。

We show gain of function of excitatory neurotransmission due to increased action-potential-evoked Ca(2+) influx and increased probability of glutamate release at pyramidal cell synapses but unaltered inhibitory neurotransmission at fast-spiking interneuron synapses.

我们发现兴奋性神经传递的功能增益来自于诱发Ca(2+)内流动作电位的增加,并可能与谷氨酸盐在椎体细胞突触中的释放增加有关,但与快速成峰中间神经元突触的抑制性神经传递无关。

Exercise increases synaptic plasticity by directly affecting synaptic structure and potentiating synaptic strength, and by strengthening the underlying systems that support plasticity including neurogenesis, metabolism and vascular function.

锻炼能增加突触的可塑性,主要通过直接影响突触的结构、增强神经键强度、加强支持可塑性的背景(包括神经发生、代谢和脉管功能)。

The range of synaptic plasticity is defined as the sum of amplitudes of LTP and LTD.

突触可塑性范围可以定量的表征突触可塑性的变化。

The mPSCs induced by GABA released from the presynaptic boutons could be completely and reversibly blocked by 5μM bicuculline, which is the specific antagonist of GABAA receptor. These results indicate that GABA released from the presynaptic boutons may produce modulatory effects through binding with GABAA receptor on postsynaptic Vme neurons.

以上结果表明:突触前终扣内释放的GABA与分布于突触后的GABAA受体结合从而发挥对Vme神经元的调控作用,GABAA受体激活后,氯离子通道开放,Vme神经元胞体内外氯离子流动构成了GABA对Vme神经元的作用基础。

Musk withBorneol can increase the quantity of the synapses in neuropil and protect the Synapseultrastructure after focal cerebral ischemia with reperfusion.

麝香配伍冰片可有效防止脑缺血再灌注导致的皮质内突触数量的减少,对神经元及神经元突触超微结构有一定的保护作用。

MAIN OUTCOME MEASURES: Comparisons of numeric density,surface density and average size of synaptic conjunction.

主要观察指标:各组大鼠突触数密度、面密度、突触连接带的平均面积比较。

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