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Of interest, all 6 TSAs contained a KRAS mutation confirming that SSAs and TSAs are genetically distinct entities.

有趣的是,所有6例TSAs均发生了KRAS基因突变,这证明SSAs和TSAs在基因水平上属不同实体。

Objective To discuss the relationship between Kras gene activation and MTS1/p16 gene mutation/deletion and the progression of endometrial carcinoma.

目的 探讨Kras激活与MTS1/p16基因突变/缺失在子宫内膜癌中的发生发展关系。

KRAS status should be considered in selecting patients with mCRC as candidates for panitumumab monotherapy.

KRAS突变状态应考虑作为转移性结肠癌panitumumab单药治疗筛选患者的一个候选标记。

ConclusionThe point mutation rate of codon 12 of Hras and Kras is low in gastric cancer, which suggestive of little role in the genesis of the cancer.

胃癌组织中Hras、Kras基因第12密码子点突变率低,在胃癌的发生中不起主要作用。

Typical late-flowering mutants had more leaves than wild type, but the Atdfc mutants plants contain the same leaf number as the wild type .

Atdfc突变体在营养生长阶段与野生型并无明显差异,但其抽薹的时间要比野生型晚。

A late-flowering Arabidopsis mutant flx was isolated.

突变体是研究基因表达和功能的良好实

At present, the late-flowering mutants are fallen into four pathways: photoperiod pathway, vernalization pathway, autonomous pathway and GA pathway according to factors described above.

目前,一般按照对以上因素的反应将晚花突变体归于四条开花调控途径:光周期途径、春化途径、自主途径和GA途径。

Methods: Mutations of FX Leiden in 103 samples of DVT and 106 samples of control were examined with PCR-RFLP.

利用聚合酶链反应和限制性片段长度多态性方法,检测103例深静脉血栓形成患者与106例正常对照的FV Leiden突变,并进行对比分析。

Objective: To explore the relationship between various mitochondrial DNA tRNA Leu gene/ND-1 gene mutations and type 2 diabetes. Methods: Case-control study was designed.

目的:探讨DNA tRNA Leu基因及ND-l基因3316G/A、3394T/C等4个突变位点与2型糖尿病的关系。

He is liable to sudden changes of mood and would sulk and cry for no apparent reason.

他的情绪容易突变,且会无缘无故地愠怒和哭泣。

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This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。

There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.

双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。