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The risk factors of breast carcinoma includes family history (The likelihood of developing breast cancer for a patient, if one of whose mother or sisters does, will increase by 1.5 to 3 times.), age, BMI, hereditary factors (The risk of developing breast cancer for a female whose BRCA1 gene has been mutated reaches 50-85% in her life.), female, contact history of the endogenous and exogenous estrogen, past history of chest being exposed to the radiation, dietary pattern, benign mastopathy and environmental factors.

发生乳腺肿瘤的危险因素包括:家族史(假如患者母亲或姐妹当中有一人发生乳腺癌,该患者乳腺癌发生的可能性增加1.5至3倍),年龄,肥胖,遗传性素因(BRCA1基因突变的女性一生中发生乳腺癌的可能性为50-85%),女性性别,内源性和外源性雌性激素接触,既往有过胸部放射性暴露史,膳食结构,良性乳腺疾病和环境因素。

We studied the mutation of PDS gene in children diagnosed to have prelingual non-syndromic hearing loss with concomitant bilateral large vestibular aqueduct and Mondini's dysplasia. We tried to find the correlation between non-syndromic hearing loss in local patients and PDS gene, and to establish the basic data of PDS gene mutations in Taiwan.

本篇研究之目的,乃对於患有非症候群学语前听障并合并双侧大前庭导水管及Mondini氏发育异常(Mondini's dysplasia)的儿童,作基因的分析,尝试找出本土非症候群听障与PDS基因的关连性,以建立台湾地区PDS基因突变的基本资料。

This represents the first demonstration of a genetic linkage between the pretibial phenotype and COL7A1 gene and supports that the Pasini, Cockayne-Touraine, and now the pretibial clinical variants of DEB are allelic.URI: http://grbsearch.stpi.org.tw/GRB/reportDetail.jsp?id=RB8

这是第一个显示胫前型EB与COL7A1基因有连锁的报告,进一步支持了显性DEB的三种主要的亚型,Pasini, Cockyane-Touraine及胫前型,都同样是COL7A1基因突变的表现。

Secret information embedded asymmetrically made the correlative degree of adjacent pixels sequence of carrier image reduced, especially the correlative degree of some local image may saltate. This phenomenon enhanced the risk of secret information being detected.

隐藏信息的非均匀嵌入使得载体图像的相邻像素序列相关度降低,而且图像局部有相关度突变的现象,增大了隐藏信息被检测到的风险。

"Our discovery that the mutant Huntington's disease protein derails the cholesterol delivery system and causes cholesterol accumulation in neurons provides us with key results and solid clues to the mechanism of this disease," says Dr. McMurray."Fully understanding the mechanism of toxicity is the key to developing treatments."

&我们发现突变的Huntington's疾病蛋白使胆固醇运输系统异常并造成神经元内胆固醇蓄积,这提供了该疾病的关键结果及重要线索,&McMurray博士说&全面理解该疾病的毒性机制有助于更进一步的临床治疗。&

Then they artificially supplied the cultured cells with one of two genes-either a functional copy of the WRN gene, which is mutant or nonfunctional in Werner Syndrome, or a gene encoding the protein telomerase, which elongates short or missing telomeres.

然后他们人为地在培养细胞中供给下述两种基因的其中之一,一种是WRN基因的功能性拷贝,它在Werner综合症患者中是突变的或无功能的。

With the aim of elucidating features which may help with differential diagnosis, this study reports the incidence and pathologic features of benign ovarian alterations, benign ovarian tumors, and occult primary and metastatic malignancies in prophylactic oophorectomies from 108 women with a BRCA mutation and from 35 women with other strong risk factors for hereditary breast/ovarian carcinoma.

为了阐明鉴别诊断的要点,本研究报道了卵巢良性病变、良性肿瘤和隐匿性原发性及转移性恶性病变的发生率和病理特征,所选病例都做了预防性卵巢切除术,包括108例伴有BRCA基因突变的女性和35例伴有其他遗传性乳腺/卵巢癌高危因素的女性。

In Crash's latest adventure, the villainous Cortex has teamed up with N. Brio and created a monstrous mind-controlling device that has taken over all the bandicoots and mutants on Wumpa Island.

在崩溃的最新冒险,在恶劣的Cortex已经与联合国 Brio ,创造了巨大的精神控制装置,已采取了所有狸和突变的Wumpa岛。

The adult stem cells in intestinal crypts, i.e., intestinal stem cells, may relate with the pathogenesis of colorectal cancer closely. This paper reviews the recent advances about intestinal stem cells and cancer stem cells of colorectal cancer.

近年较多的文献报道结直肠癌组织中存在肿瘤干细胞,而这些具有干细胞特性的瘤细胞可能来自突变的肠干细胞,本文就肠干细胞和结直肠癌肿瘤干细胞的研究进展作一综述。

Cybrid cells can be studied in th...

融合细胞具有一致的核背景,可以消除核基因的作用,因而有助于判断mtDNA突变的致病作用及机制和线粒体缺陷的致病作用及机制。

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This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。

There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.

双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。