突变型

We compared the volume change of the plasmid control (C1), Bcl-2 wild-type (B6), and Bcl-2 mutant cells following superfusion with hypotonic solutions.

我们在低张溶液下比较了控制株C1、野生型B6 与突变型mI-2.4， mI-2.7 细胞的体积调控能力。

Thus, the mutant amplification products were made of mostly double-stranded DNA, while the wild-type products were made of mostly unconsumed single-stranded DNA.

突变型样品扩增产物中大部分是双链DNA；而野生型样品由于不能被顺利扩增，产物中大部分是单链DNA。

And large numbers of mutant p53 protein were expressed in liver, kidney, ventriculus glandularis, tumor, oviduct. Moderate expression were detected in heart, pancreas, bone marrow, spleen, lung, duodenum, thymus and bursa of fabricius.

结果表明：突变型p53在肝脏、肾脏、腺胃、肿瘤、输卵管中表达量最高，心脏、胰脏、骨髓、脾脏、肺脏、十二指肠、胸腺及法氏囊均可检出中等量的表达。

The development mode of this sudden change is different from that of the Wicker Inner.

边外城镇的这种突变型的发展模式是迥异于边内地区的。

A mutation type and a wideness type were established by clone and gene sequence detection.

基因测序技术对无缺失片段进行克隆、测序，筛选出突变型及野生型。

They were able to confirm which pairs of mutants were allelic and which were not by using the heterocaryon test.

他们通过异核体测验可以确定哪些突变型是等位的，哪些是非等位的。

Five missense mutations in the most commonly altered PTP were biochemically examined and found to reduce phosphatase activity.

在人类肿瘤细胞中表达野生型而不是突变型PTPRT可以抑制肿瘤生长。

Objective To study the relation between histopathologic grading and some of the cytogenetic and molecular biology characteristics of breast cacner.

目的探讨乳腺癌细胞增殖活性、bcl-2和突变型p53蛋白表达水平的临床意义。

In order to understand the drug resistance mechanism of HIV-1 integrase mutually existed for multiple drug-resistant strains to the most potent IN inhibitors diketo acids, three S-1360-resistant HIV-1 strains were selected and molecular docking and molecular dynamics simulations were performed to obtain the inhibitor binding modes.

为了解DKAs引起的多种耐药株共有的耐药性机理，选择3种S-1360引起的IN耐药突变体，用分子对接和分子动力学模拟，研究了野生型和突变型IN与S-1360的结合模式，基于该结合模式探讨了3种耐药突变体所共有的耐药性机理。

To investigate the relationship between Arg778Leu mutation of ATPTB gene and clinical manifestation in hepatolenticular degeneration.

目的：探讨肝豆状核变性（Hepatolenticular Degeneration，HLD）患者ATP7B基因Arg778Leu突变型与临床表现之间的相关性。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。