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离体酶

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The young mycelia, plate-cultured for 5 days, were almost dissociated through myolysis in a mixed enzymic solution at 32℃ for 5-7hours.

六菌株平板培养五天的幼龄菌丝体,在混合酶液中32℃摇瓶酶解5~7小时,即可全部离解。

The infectivity of TMV recovered by dialysis from incubation with anthraquinone glycosides in vitro has been reduced in contrast to infectivity of TMV control, it seems that anthraquinone glycosides have direct viricidal effect on TMV. The anthraquinone glycosides may interfere recognization of TMV to host receptor when applied before TMV inoculation. The anthraquinone glycosides can inhibit multiplication of TMV in infected leaf discs but did not inhibit infectivity of TMV-RNA in vitro. The control of tobacco mosaic virus disease of N.

总蒽醌甙对TMV混合,对粒体形态无破坏作用,但经透析,其抑制作用仍较高,表明总蒽醌甙与TMV结合能力强不易解离或对粒体具有直接毒害作用;总蒽醌甙预先接种可能影响病毒对侵染位点的识别,对叶圆片中的病毒具有抑制复制的作用,但在体外混合不影响TMV-RNA的侵染活性,可能与总蒽醌甙干扰病毒的衣壳装配过程或影响复制酶的活性有关。

Methods The neuromuscular transmission in isolated rat phrenic diaphragm was inhibited completely by 2.5×10-3 mol/L DDVP,thenwashed out the toxicants and the neuromuscular transmission was recoverd by incubation with the above oximes respectively.

用2.5×10-3 mol/L敌敌畏完全抑制大鼠离体膈神经膈肌传导,然后洗掉毒剂,加药对抗:用外加5.6×10-5 mol/L乙酰胆碱的方法来检验药物是以中毒酶的重活化作用为主,还是以直接生理对抗作用为主。

Objective This paper is to study the mechanism of HI-6,PAM-Cl,PAM-I and obidoxime against the inhibition of DDVP on isolated rat phrenic diaphragm. Methods The neuromuscular transmission in isolated rat phrenic diaphragm was inhibited completely by 2.5×10-3 mol/L DDVP,thenwashed out the toxicants and the neuromuscular transmission was recoverd by incubation with the above oximes respectively.

用2.5×10-3 mol/L敌敌畏完全抑制大鼠离体膈神经膈肌传导,然后洗掉毒剂,加药对抗:用外加5.6×10-5 mol/L乙酰胆碱的方法来检验药物是以中毒酶的重活化作用为主,还是以直接生理对抗作用为主。

The coronary flow,troponin-T and adenosine of effuent,ecto-5′-nucleotidase activity and MDA in myocardial tissue were measured.

应用兔离体心肌缺血再灌注损伤模型,在Thomas 液中加入黄芪注射液,检测冠脉流量、冠脉回流液中肌钙蛋白-T和腺苷释放、心肌组织中的MDA及外部-5′-核苷酸酶的活性。

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然而,正如其名字所指出的那样,CD盘不能写,也不能用任何方式改变其内容。

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