神经鞘炎

Those with heterogeneous enhancement were often of acquired etiology, such as ranula, lymphoma, neurilemmoma, parotid adenoma, metastatic nodes and tubercubus adenitis, etc.

至於那些有异质性加强的病变，通常属於后天性疾病，例如：舌下囊肿、淋巴瘤、神经鞘瘤、腮腺瘤、转移性淋巴结、结核性淋巴腺炎等。

Results Among 85 cases, 46 cases were lipomas, Twenty-seven cases were sebaceous cysts, 19 cases were angeiomas, 8 cases were fibroids, 5 cases were ganglionic cysts, 3 cases were neurilemmomas,1 case was dermal nevus, 1 cases was fattey necrosis, 1 case was adipositis, 1 case was foreign-body granuloma,1 case was calcified epithelioma.

结果 85例中脂肪瘤46例，皮脂腺囊肿27例，血管瘤19例，纤维瘤8例，腱鞘滑液囊肿5例，神经鞘膜瘤3例，皮肤痣、脂肪坏死、脂膜炎、异物肉芽肿、钙化上皮瘤各1例。

The notable proliferation was not observed by eyes in the local of injection. The infiltration of inflammation cells and mild proliferation of fibrocyte around dura mater was observed by HE stained in 4 and 8 weeks after injection. Infiltration and exudation of inflammation cells was observed by HE stained in epidural nerve root. Compared with group A, no changes of group B, C and D were observed under specific stained. Proliferation of type Ⅱ collagen fibers around dura mater was seen under immunohistochemical stained in 4 and 8 weeks after injection. There is no significant demyelination changes under LFB stained. The thickness and shape of the myelin sheath in epidural nerve root was not regular under transmission electronic microscopy in 4 and 8 weeks after injection. Fibroblast was also seen there. In nerve endometrium, macrophage could be seen under TEM, myelinated nerve fiber changed significantly, but nonmyelinated nerve fiber changed mildly. When 8 weeks, the changes of group D is smaller than the group B and C.

给药局部肉眼观察未见明显的纤维组织增生；HE染色可见B、C、D三组给药后四周及八周时硬膜内外均有炎细胞浸润，纤维细胞轻度增生，硬膜外神经根内有炎细胞浸润及炎性渗出；特殊染色B、C、D三组同A组相比未见有脊髓及神经根的改变；免疫组化染色，给药后四周及八周时，硬膜内外均有Ⅱ型胶原纤维增生；固兰染色B、C、D三组未见有明显脱髓鞘改变，与A组相比无明显异常改变；电镜观察B、C、D三组在给药后的四周及八周时，表现为硬膜外神经根内髓鞘厚薄不一，形状不规则，可见成纤维细胞，神经内膜中可见有巨噬细胞；粗大的有髓神经纤维变化明显，无髓神经纤维受累较轻；八周时电镜下D组改变较B、C两组为轻。

It was considered an effective and ideal model of PM.2 uPA,PAI-1 and TNF-αwere all involved in the pathogenesis of PM and participated in the process of the degeneration,apoptosis and necrosis of the diseased muscle.3 MMP-9 and TIMP-2 immunoreactivity were positive in the capillary,venous wall,arteriola endotheliocyte and neurilemma in the normal group skeletal muscle,but more intensive in EAM.They may aggravate the pathological changes of the muscle in human PM.

结论1兔纯化的肌球蛋白加含卡介苗的完全弗氏佐剂可仿制出人类多发性肌炎的理想动物模型。2 UPA、PAI-1和TNF-α参与多发性肌炎的肌纤维坏变过程。3 MMP-9和TIMP-2在正常肌肉组织中的毛细血管、静脉管壁、动脉肉皮细胞和神经鞘膜表达，在多发性肌炎中，参与加重肌纤维的坏变过程。

"Myelin basic protein as an encephalitogen in encephalomyelitis and polyneuritis following rabies vaccination".

髓鞘碱性蛋白作为encephalitogen在脑脊髓炎和多发性神经炎后狂犬病疫苗接种。

There were infiltration of inflammatory cells inside nerve tissue and perivascular cuffings in HE staining. In trichrome staining, the axon with inhomogeneous chromatosis was swollen and beaded-discontinued; The myelin was porous and partly disaggregated.

HE染色发现神经组织内炎细胞浸润，血管&袖套&样病灶形成；三色染色可见轴突肿胀，呈串珠状，且不连续，着色不均匀；髓鞘结构层次紊乱，疏松，崩解。

Results Among 85 cases, 46 cases were lipomas, Twenty-seven cases were sebaceous cysts, 19 cases were angeiomas, 8 cases were fibroids, 5 cases were ganglionic cysts, 3 cases were neurilemmomas,1 case was dermal nevus, 1 cases was fattey necrosis, 1 case was adipositis, 1 case was foreign-body granuloma,1 case was calcified epithelioma.

回顾性分析85例体表良性肿块的超声表现，总结其声像图特征，所有病例均经手术病理证实。结果 85例中脂肪瘤46例，皮脂腺囊肿27例，血管瘤19例，纤维瘤8例，腱鞘滑液囊肿5例，神经鞘膜瘤3例，皮肤痣、脂肪坏死、脂膜炎、异物肉芽肿、钙化上皮瘤各1例。

Do you know, i need you to come back

你知道吗，我需要你回来

Yang yinshu、Wang xiangsheng、Li decang,The first discovery of haemaphysalis conicinna.

1〕 杨银书，王祥生，李德昌。安徽省首次发现嗜群血蜱。