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神经膜细胞

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Results Postoperative pathological diagnosis showed 13 ependymomas, 2 astrocytomas, 2 oligodendroglioma, 2 lipoma, 1 myeloma, 1 keratose, 1 metastatic tumor, 1 angeioma, and 1 neurinoma.24 cases were treated by surgeon.

结果 病理证实室管膜瘤13例,星形细胞瘤2例,少枝胶质细胞瘤2例,脂肪瘤2例,骨髓瘤、转移瘤、血管瘤、角化物、神经鞘瘤各1例。24例行手术治疗。

The thickness of myelin sheath of experimental group was inferior to that of group B (P.05) at the third month, while the number and diameter of axons in both groups was similar.

这种无细胞基膜管移植体能支持轴突的生长和雪旺细胞的迁移,是一种良好的神经移植替代材料。

The blood-brain barrier was composed of capillary endothelial cells, basement membrane and neuroglia membrane.

血脑屏障由毛细血管内皮细胞、基膜和神经胶质膜构成。

After every 60μl of 30% HRP were injected into abdominal section of the esophagus of 20 rats respectively,the labelled neurons were found bilaterally within nodose ganglions of vagus nerve,dorsal root ganglia(T9~L3),thoracic sympathetic ganglions and coeliac ganglion .

将30%辣根过氧化酶溶液注入大鼠食管腹段近贲门处外膜下,切片用四甲基联苯胺法成色,结果如下:标记细胞主要分布于双侧结状神经节,双侧的脊神经节(主要在T9~L3)、双侧的胸交感节(T9~L1)和腹腔节,在延髓的标记细胞见于疑核全长内,主要分布于疑核咀侧端。

Results Denatured necrosis occurred on the surface of hypothalamic periosteum with no cells and there was a large amount of starchy bodies and hydrops in foci with angiectasis of choroid plexus and necrosis and calcification in tunicaintima of vesicles,and mitochondria in the nerve synapse disappeared and part of anteroposterior membranes ...

结果 慢性SZ患者下丘脑部室管膜表面变性坏死、无细胞,病灶中有大量淀粉样小体和水肿,脉络丛血管扩张,管壁内膜有坏死和钙化;神经突触内大部分突触小泡和线粒体消失,部分前后膜模糊不清。

A total of 150 mesenchymal neoplasms of the GI tract, abdominal cavity, and retroperitoneum were retrieved from consult and surgical pathology archives, including 54 GISTs (8 KIT-negative; 13 desmin-positive), 17 GI leiomyosarcomas, 11 GI mural leiomyomas, 13 leiomyomas of the muscularis mucosae, 12 gastric schwannomas, 15 inflammatory myofibroblastic tumors, 9 cases of mesenteric desmoid fibromatosis, 10 dedifferentiated liposarcomas, and 9 malignant peripheral nerve sheath tumors.

我们从会诊病例及活检病例中找出150例间叶性肿瘤(来自胃肠道、腹腔和后腹膜),其中包括54例GISTs(8例KIT阴性、13例desmin阳性)、17例GI道平滑肌肉瘤、11例GI道肌壁间平滑肌瘤、13例GI道粘膜肌层平滑肌瘤、12例胃的神经鞘瘤、15例炎性肌纤维母细胞瘤、9例肠系膜韧带样纤维瘤、10例去分化型脂肪肉瘤和9例恶性外周神经鞘膜瘤。

Aim:To explore the feasibility of neural stem cells derived from both fetal rat ependyma and adult rat bone marrow.

目的 :探寻胚鼠室管膜和成鼠骨髓分离培养神经干细胞的可行性。方法:将分离的胚脑室管膜组织或成年骨髓细胞分别特殊培养。

Results The neurological defects score was singificiant lower in D group (41.50±8.18) than that in group A(82.20±8.08), B(63.60±6.22) and C (61.00±6.58)(all P.01). Compared with groups A, C, group D showed that the cellular nucleus was shrinkaged slightly, the gap of cells was nomal roughly and the degree of rarefaction in neurofibra net was not serious under the light microscope. Under the electron microscope, the neuron was degenerated, the cellular membrane was integrated and the plastosome was swelled lightly in the group D. The physiological demage were slighter in D group.

结果 D组神经功能缺损评分(41.50±8.18)明显低于A组(82.20±8.08)、B组(63.60±6.22)和C组(61.00±6.58)(均P.01);光镜下D组神经细胞核轻度皱缩,细胞周围间隙大致正常,神经纤维网疏程度不严重;电镜下D组神经元轻度变性,细胞核无皱缩,核膜完整,线粒体轻度肿胀;脑组织病理损害程度较B组和C组明显减轻。

The distribution of AQP1 consisted of cells lining the bony labyrinth, fibrocytes lining the endolymphatic duct and sac, cells under the basilar membrane, fibrocytes of the spiral ligament and the spiral limbus, Cortirs organ, inner and outer spiral sulcus, stria vascularis, saccular and utricular wall, and spiral ganglion.

AQP3、7、8的分布类似,在螺旋神经节和包绕膜迷路的组织中均有表达,其中Corti器、内外螺旋沟、血管纹、螺旋神经节表达较强,在螺旋韧带、螺旋缘纤维细胞表达较弱。

Activation of the surface DORs causes a slow Ca〓 elevation by both inositol 1, 4, 5-trisphosphate receptor-mediated intracellular Ca〓 release and extracellular Ca〓 entry, resulting in a slow and long-lasting increase in intracellular Ca〓, exocytosis, DOR insertion and CGRP release.

激活DOR引起的DOR的上膜是依赖于LDCV与细胞膜的融合和细胞内的钙离子升高。激活DORs可以引起胞内IP3钙库的释放,和一种不通过电压依赖性钙通道的外钙内流;从而引起DOR的上膜和神经肽的分泌。

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This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

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There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.

双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。