神经胶质的

There was an obvious increase of insulin receptor gene and protein in the cerebral ischemia reperfusion tissues in rats. The positive cells such as neuron, neuroglia cells, vascular endothelium cells possibly participated in regulating the processes of energy metabolism of cells and neovascularization. It implied that the high expressions of insulin receptors in vascular endothelium cells in ischemic zones possibly participated in regulating the pathophysiological changes of the blood vessels in ischemic zones; The higher expressions of insulin receptors in different cells were possibly one of the self-protective machanisms in which the ischemic tissues confront the anoxemia injury.

脑缺血再灌注大鼠脑组织胰岛素受体基因和蛋白水平表达增加；半暗带区神经元、部分神经胶质细胞及血管内皮细胞等阳染细胞可能参与脑缺血后组织细胞的能量代谢、促新生细胞形成等病理生理调节过程；胰岛素受体在缺血区血管内皮细胞高表达提示其可能在缺血区血管的病理生理变化中起某种调节作用；胰岛素受体在不同细胞中表达增高可能是缺血脑组织对抗缺血缺氧损伤的自身保护机制之一。

Abstract］ AIM: To study the cytotoxic effects of doxorubicin on apoptosis in glioma cell lines U343, U138, U373 induced by antihuman DR4/DR5 monoclonal antibodies (FMU1.4/FMU1.5) and the underlying mechanism.

摘 要］目的：探讨亚毒性剂量的阿霉素影响抗DR4、 DR5单克隆抗体 FMU1.4和FMU1.5诱导3株神经胶质瘤细胞株U343、 U138及U373凋亡的作用及可能的机制。

We aim to show that, concerning glia as well as neurons, Cajal was far ahead of his time.

我们的目的在于向世人表明：Cajal在涉及神经胶质和神经元的研究中，远远领先于他的时代。

This Manichean finding came after Dr Wigertz and her team interviewed 1,527 people with gliomas in Denmark, Finland, Norway, Sweden and the south-east of England.

威格茨博士与她的研究小组是在丹麦、芬兰、挪威、瑞典和英格兰东南部调查了 1527名神经胶质瘤患者之后得出这一模棱两可的结论的。

Meanwhile, activatedNF-κB also appeared in vascular endothelial cell in brain for more than 1 year. Long-term activation of NF-κB in neuroglia cells induced atrophy of involved brain.

同时，激活的NF-κB在创伤1小时后出现于脑血管内皮细胞内，持续时间也在1年以上，神经胶质细胞内NF-κB的长期激活可引起该部位的脑萎缩。

Neuroglia, the basic integrant of blood-brain-barrier is defective in brain metastatic tumor tissue, thus there is no blood-brain-barrier existing in brain metastatic tumor lesions.

脑转移癌组织缺乏构成血脑屏障的神经胶质膜，血管内皮也非均一的紧密连接，所以脑转移癌组织不存在完整的血脑屏障。

Methods: Oxygen–glucose deprivation injury was induced in pure neuronal or neuronal–glial cocultures 24 h after preconditioning with xenon and/or sevoflurane.

缺氧缺糖损伤是预处理氙气和/或七氟醚24 h后纯粹的神经元或神经胶质联合培养所致的。

Using an in itro bioassay, we demonstrated that cytokine-actiated microglia release cytotoxins that kill retinal neurons.

应用体外生物测定技术，我们证明细胞因子活化的小神经胶质细胞释放杀死视网膜神经元的细胞毒素。

Phagocytic microglia release cytokines and cytotoxins that regulate the survival of astrocytes and neurons in culture.

酒精进入神经系统后可对各种细胞产生直接毒性作用，包括减少大脑某些部位神经元细胞的数目和破坏神经元与神经胶质细胞之间的正常关系。

After treatment with the conditional medium of tumor cells, the number of tubules increased in a dose-dependent manner.

结果：血管生成素-2在神经胶质瘤细胞中的表达强度与瘤组织的分级呈正相关(p.05)；肿瘤细胞诱导肿瘤血管形成呈浓度依赖效应（p.05）。

Breath, muscle contraction of the buttocks; arch body, as far as possible to hold his head, right leg straight towards the ceiling (peg-leg knee in order to avoid muscle tension).

呼气，收缩臀部肌肉；拱起身体，尽量抬起头来，右腿伸直朝向天花板（膝微屈，以避免肌肉紧张）。

The cost of moving grain food products was unchanged from May, but year over year are up 8%.

粮食产品的运输费用与5月份相比没有变化，但却比去年同期高8％。