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神经胶质瘤

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Abstract] AIM: To study the cytotoxic effects of doxorubicin on apoptosis in glioma cell lines U343, U138, U373 induced by antihuman DR4/DR5 monoclonal antibodies (FMU1.4/FMU1.5) and the underlying mechanism.

摘 要]目的:探讨亚毒性剂量的阿霉素影响抗DR4、 DR5单克隆抗体 FMU1.4和FMU1.5诱导3株神经胶质瘤细胞株U343、 U138及U373凋亡的作用及可能的机制。

It has been reported recent years that the expression level of uPA system is closely associated with gliomas.

近年来发现uPA系统的表达水平与神经胶质瘤的恶性程度呈正相关。

The location of the cleome glioma must also be considered when deciding treatment.

当决定治疗时神经胶质瘤的位置也必须考虑。

Remov al of the cleome a glioma from this area can result in speech problems and other disabilities.

移除这个区域的一个神经胶质瘤会导致一些语言问题和其他残疾。

Sniggers Seizures and headaches are comm on f irst signs of a cleome glioma .

发作和头痛是神经胶质瘤常见的第一现象。

Eighteen patients with histologically confirmed gliomas treated by GKS and followed for more than 24 months were recruited.

此研究包括18位经组织学证实有神经胶质瘤以加马刀放射手术治疗并追踪超过24个月的病人。

They are all with similar characteristic of CPE formation and infectivity in those cell culture.

在神经亲合性的实验中,我们将现有病毒株感染三种神经细胞株,即上述二种及神经胶质瘤细胞-U373MG。

Curran and a team of American and Canadian researchers, in conjunction with the Philadelphia-based Radiation Therapy Oncology Group, a federally-supported clinical trials organization, found that among 289 patients with either anaplastic oligodendroglioma or anaplastic oligoastrocytoma receiving either a combination of three chemotherapy drugs with radiation or radiation alone, those whose tumors had deletions in chromosome locations 1p and 19q tended to live nearly two and a half times longer than those patients without the missing chromosome portions.

临床癌症杂志》上周报道,卡仑博士和由美国、加拿大研究人员组成的科研小组,联合了总部设在费城的由联邦政府支持的临床实验组织-美国放射肿瘤学协作组,进行了研究。289名病人患有退行性少树突神经胶质瘤或退行性少星细胞瘤,研究发现,这些病人在接受三种化疗药物结合放疗或者只接受放疗后,其中染色体缺乏1p 和19q 的病人比那些不缺乏的病人多存活了2倍半的时间。

More than forty percent of growths in the brain are gliomas.

脑部超过40%的瘤都是神经胶质瘤

After treatment with the conditional medium of tumor cells, the number of tubules increased in a dose-dependent manner.

结果:血管生成素-2在神经胶质瘤细胞中的表达强度与瘤组织的分级呈正相关(p.05);肿瘤细胞诱导肿瘤血管形成呈浓度依赖效应(p.05)。

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