神经细胞

Objective To investigate the mechanism of neurocytes' damage caused by Aβ25-35 and the change of enzyme in the signal transduction passway.

目的 探讨β淀粉样蛋白25-35 (Aβ25-35)导致神经细胞损伤的机制及在损伤过程中涉及的信号转导通路中相关酶caspase-3的变化。

Objective To establish a perineural invasion model of human bile duct carcinoma in vitro, and observe the growth of nerve fiber, proliferation of the human bile duct carcinoma cell and the invasion process.

目的构建人胆管癌细胞神经周围浸润模型，观察培养神经细胞的生长、人胆管癌细胞的增殖与神经周围浸润的过程的形态学变化。

The result showed that PSexternalization. This observation suggested that apoptotic neurons losed membranephospholipid asymmetry and exposed PS on the outer leaflet of the plasmamembrane. Macrophages then phagocytose apoptotic neurons after specific recognition of theexposed PS.

流式细胞术检测磷脂酰丝氨酸，结果表明凋亡的神经细胞PS外翻，证明AVM可使凋亡的神经细胞膜丧失不对称性，细胞膜中的PS由脂膜内侧翻向外侧，可被吞噬细胞识别后导致凋亡细胞的清除。

In the post-treatment groups, dl-NBP(12.5, 25 mg·kg-1 ip) induced dilation of the pial arterioles and the increase in BFV was in dose-dependent manner. The pial arteriolar response to MCAO was not affected by d-NBP and nimodipine.

在脑缺血时，脑微循环障碍是导致神经细胞损伤的主要病理基础之一，所以本研究通过观测NBP对MCAO后软脑膜微循环的影响，进一步分析其抗脑缺血的药理作用机制。

Pinal muscular atrophy is one of the most common inherited forms of neurological disease leading to infant mortality.

MA是一种遗传疾病，它会攻击脊髓中的运动神经细胞，人体内缺乏SMN蛋白（SMN蛋白是运动神经元生存蛋白，能够使肌肉活动）时会引发该疾病。

Outcome:(1) Acute ischemic damage may induce the expression of c-fos protein in Cerebral Cortex quickly. Also, the expression increases by the ischemia time delay. It may hint that ischemia injury can rise the ability of nerve cells to tolerate ischemia and anoxia. And the expression level of c-fos can be up-regulated by putting blood of 12 hand Jing Points. So we think that treated by this therapy can rise the irritability of brain to resist the development of ischemia and enhance the ability to plerosis.(2) Acute ischemic damage may induce the expression of HspVO in Cerebral Cortex quickly. Also, the expression increases by the ischemia time delay. It may hint that ischemia injury can rise the ability of nerve cells to tolerate ischemia and anoxia.

三 结果(1)急性缺血性脑损伤可快速诱导c-fos蛋白在皮层缺血区脑组织的表达，其表达有随缺血时间延长而逐渐增多的趋势，提示脑的缺血性损伤可应激性地增强神经细胞对缺血、缺氧的耐受和适应能力，而应用井穴放血的干预治疗可明显上调缺血区皮层脑组织c-fos的表达水平，进一步增强脑组织对抗缺血性损伤的应激能力，抵抗缺血性脑损害的进一步发展。

The cerebral cortex include three layers: outer plexiform layer,cell layer and inner plexiform layer. There are some small cells scatter in outer and inner plexiform layer.

扬子鳄大脑皮层可分为外网状层、细胞层和内网状层三层，神经细胞绝大多数集中于细胞层内，内、外网状层中零星分布一些小细胞。

In this study, the antioxidant components, antioxidative activity of five species of Davalliaceae plants and two polypodiaceae plants were investigated first by microtiter spectrophotometric and fluorescent methods. Then the protective effects of these plants on 6-OHDA-induced neuronal damage in B35 cells were investigated.

本实验采用俗名为骨碎补的五种骨碎补科与两种水龙骨科之近源植物作为实验材料，进行抗氧化成分含量与活性评估，并以离体方式探讨其对6-OHDA所诱导神经细胞损伤之保护作用与机制。

Neurotransmitters traverse the physical space between two neurons and bind to special protein receptors on the postsynaptic cell.

神经传感横贯两个神经细胞的物质空间，在突触后的细胞中绑定特殊的蛋白质受体。

Preinduction was performed using basic fibroblast growth factor, which could promote the differentiation from BMSCs into nerve precursor cells or neurons. The expression of neuroprotein molecule was coincided with the development of neural cells.

在添加化学诱导剂之前先使用成纤维生长因子进行预诱导，能促进骨髓间充质干细胞向神经前体细胞和神经元转化，且在诱导分化过程中神经蛋白分子的表达顺序与神经细胞发育过程一致。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。