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Focal cytopathic areas were observed in cerebral cortical area and so did neuron necrosis around by gliacytes formed stellitorsis, neuronophagia and glial nodule.

RVJR2 3 株在人脑神经细胞和Balb/c小鼠脑组织中均能形成感染,感染为非杀细胞性

NGF is one of the neurotrophic factors.

NGF是神经营养因子的一种,具有促进交感神经细胞和感觉神经细胞生长与发育的作用。

These growth factors have the effects of neurogenesis, angiogenesis, neurotrophy, inhibitory excitoxity induced by glutamate and NMDA, and anti-oxidative, by these ways they can prevent neural cells from death.

这些生长因数均有保护神经细胞避免因为脑损伤而造成的死亡,其作用有神经生长性、血管生长性、神经滋养性、抑制麸氨酸盐与NMDA所引发的兴奋性毒性及抗氧化作用,但是这些仅限於动物模式,应用於临床还有一段艰辛的历程。

Results B, C can cause the hypomnesia of the Wistar rats(P<0.05) and result in decreasing amounts of pyramidale in CA1 sector of hippocampus. The neurotropic and glial nodule were observed.

结果 氯胺酮和D-半乳糖分别可致大鼠记忆功能明显下降(P<0.05)和海马CA1区锥体细胞减少,并可见嗜神经细胞现象和胶质细胞小结。

The human bile duct carcinoma cell line QBC_(939) was neurotropic. The co-culture model of DRG/nerve cell and carcinoma cells could simulate the process of the tumor cell going through the matrix and invading the nerve fiber.

结论DRG/神经细胞-肿瘤细胞共培养模型较好的模拟了肿瘤细胞穿过细胞基质,浸润神经纤维的过程,人胆管癌细胞QBC939细胞对神经纤维具有亲嗜性。

Neurovascular unit was an integral one that consisted of neuron,vascular endothelial cells,et al.

神经血管单元是指由神经细胞、血管内皮细胞、星形胶质细胞及其周围的细胞外基质组成的一个整体。

Objective To investigate the interrelationship between the expression of VEGF and the oncosis of neuron after ICH by establishing the rat ICH model through the expression of oncosis of neuron after ICH and the cell location of VEGF-positive cells in perihematoma and the expression of VEGF. Methods The rat ICH model was induced by two-step.

目的 通过建立大鼠脑出血动物模型观察血管内皮生长因子在出血灶周的细胞定位和动态表达变化和脑出血后神经细胞胀亡时程表达以探讨两者之间的相关性。

These results show that overexpress the Tra2β1 protein may influence the propotion of the flip/flop of the AMPA receptors, and change the kinetic and ion selective of the receptors. So it can drive the development of the neural synapses.

此实验表明:过表达Tra2β1蛋白可能影响了神经细胞谷氨酸AMPA型受体flip/flop型剪接的比例,并且改变了AMPA受体的动力学和离子选择特性,从而加速了神经突触形成的发育进程。

Suyu capsule can improve all the symptoms of the depression model mice and protect injury of hippocampal neurons in the depression model mice. The possible mechanism of action is to restrict Ca2+ ion overfreight.

苏郁胶囊能改善小鼠的抑郁样行为;保护抑郁模型小鼠海马神经元损伤,其可能与苏郁胶囊抑制海马神经细胞外Ca2+内流,阻止Ca2+超载相关。

METHODS: Model of oxygenic injury of PC12 nerve cells was established.

制备神经细胞氧化损伤模型。

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