神经细胞

In 1994 and again, in 2002, Dr. Leif Salford from Lund University in Stockholm, Sweden found in his studies that rats exposed to cell phone radiation showed a breakdown in the blood brain barrier, as well as areas of shrunken, damaged neurons .

在1994年再次在2002年，雷夫索尔福德博士从瑞典隆德大学在瑞典首都斯德哥尔摩发现在他的研究，老鼠暴露于手机辐射的细目显示，在血脑屏障，以及领域的萎缩，神经细胞受损。

Recent studies found that ischemic brain damage is resulted from many reasons such as functional rearrangement of glia cells, an ion balance disorder, lipide peroxidation induced by free radicals, and changes of neurotransmitters.

许多继发性因素，如缺血后神经胶质细胞功能的重新安排，离子平衡紊乱，自由基引起的脂质过氧化损伤，以及神经递质的改变等可诱导神经细胞死亡，继而导致脑损伤，但有关缺血性细胞死亡机制目前还无定论。

Bone marrow mesenchymal stem cells could differentiate into osteoblasts, adipocytes and neural like cells with osteoblast inductor (β-sodium glycerophosphate, dexamethasone, vitamin C), lipoblast inductor (dexamethasone, 3-isobutyl-1-methylxanthine, bovine insulin, indometacin) and serum-free medium inductor (dimethyl sulphoxide, butylated hydroxyanisole) respectively. Osteoblast marker (alkaline phosphatase, osteocalcin mRNA, calcium node), adipocyte marker (lipid droplet, PPAR γ-2mRNA) and neural cell-like marker (nissl body, neuron specific enolase, neurofilament protein) were respectively determined by the immunohistochemical method, polymerase chain reaction and immunocytochemical method.

分别采用成骨细胞诱导剂（β-甘油磷酸钠，地塞米松，维生素C）、成脂肪细胞诱导液（地塞米松，甲基异丁酸黄嘌呤，牛胰岛素，吲哚美辛）及二甲基亚砜和羟基丁酸苯甲醚无血清培养基诱导剂干预细胞向成骨、脂肪、神经细胞分化，经免疫组织化学染色、PCR、免疫细胞染色方法检测成骨标志物（碱性磷酸酶、骨钙素mRNA、钙结节）、脂肪标志物（脂滴、PPARγ-2mRNA）、以及类神经标志物（尼克氏体、神经烯醇化酶、神经丝蛋白）。

Hours after operation, most nerve cells disappeared and the glial cells were degenerated. There were many mono-nucleus cells infiltrating in transplantation group while a few in control group. One week after operation, tissues in the infarction zone were liquescent with many monocaryons and lymphocytes infiltrating around them in control group.

术后48 h神经细胞大片消失，胶质细胞变性，自体骨髓干细胞移植组有大量单个核细胞浸润，对照组相对较少。1周后对照组梗死区脑组织液化变软，坏死灶周围可见较多的单核和淋巴细胞浸润；自体骨髓干细胞移植组大鼠部分坏死灶被新生毛细血管和胶质细胞增生修复，炎性细胞浸润不明显。

The astrocyte swelling appeared as early as 30 mins and indented the neuron at 6h after MACO.

缺血30分钟后，〓ATP酶及〓-ATP酶反应产物在神经细胞中即减少到消失。

In the experiment, depended on the traditional ischemia model MCAO, we both use the clot embolus model to study the edema of the neuron cell. We found that the hyperintense signal was seen in the DWI in few minutes by using the MACO models after the ischemia and reperfusion.

本实验在利用传统缺血模型MCAO基础上，结合与人类缺血卒中病程较接近的自体血栓脑栓塞模型，模拟急性脑卒中的过程，研究急性期缺血后神经细胞水肿发展的规律，了解神经元的生物代谢物质变化趋势，评价早期溶栓治疗的实验意义。

This study was designed to investigate the effects of microglia condition medium on neurons during kainate-induced excitotoxicity.

本论文为了探讨kainate所引发的兴奋性毒性过程中，小神经胶细胞条件培养液对神经细胞的影响。

So far the role of microglia to neural cells is still undefined.

因此小神经胶细胞对神经细胞的作用角色至今尚无定论。

In H/R injury of mixedly cultured cerebral cortex neuron and glia, the increase of NO production which was mainly generated by glia, participated in oxidative insult.

在混合培养的大鼠大脑皮质神经细胞和神经胶质细胞

This review focused on the biological significance of matrix metalloproteinases in its regulation when mesenchymal stem cells differentiate into myoblastic, osteoblastic, chondroblastic, neural, and apidoblastic lineages.

文章详细阐述了基质金属蛋白酶在间充质干细胞分化为成肌细胞、成骨细胞、破骨细胞、神经细胞、脂肪细胞过程中所起的重要生物学作用，并对基质金属蛋白酶在间充质干细胞分化中的调控作用进行分析。

But we don't care about Battlegrounds.

但我们并不在乎沙场中的显露。

Ah! don't mention it, the butcher's shop is a horror.

啊！不用提了。提到肉，真是糟透了。