神经细胞

CD4+T cells directly injected into the endoneurium caused conduction block followed by macrophage infiltration and demyelination.

CD4+T细胞直接注射到神经细胞内，引起巨噬细胞浸润和脱髓鞘，随后引起神经传导阻滞。

This multitude of neurons in the enteric nervous system enables us to "feel" the inner world of our gut and its contents.

这种神经细胞的多种肠神经系统，使我们能够&感觉&我们的肠道，其内容的内心世界。

Spinal cord injury models were made in 60 wistar rats and the dynamic expression of bromodeoxyuridine and polysialylated ependymal cells adhesion molecule were determined by immuno-histochemisty.

方法］Wistar大鼠60只，制作大鼠脊髓全横断损伤模型，根据伤后不同时间点分组，应用免疫组织化学方法动态检测脊髓内5-溴脱氧尿嘧啶、多唾液酸神经细胞黏附分子的表达。

Multiple factors participated in the process of secondary brain edema and nerve injuries,such as the hydrostatic pressure during formation of the hematoma and clot retraction, activation of blood coagulation cascade, generation of thrombin,erythrocytolysis and the toxicity of hemoglobin, the activation of the complement system, space-occupying effects, rupture of the blood-brain barrier,secondary ischemia around the hematoma, inflammatory reaction of the neurons around hematoma and cell apoptosis.

脑出血后继发水肿及神经损害有多种因素参与，血肿形成过程中的流体静力压和血凝块的回缩、凝血瀑布的激活、凝血酶的产生、红细胞溶解和血红蛋白的毒性、补体的激活、占位效应、血脑屏障的破坏、血肿周围继发缺血、血肿周围组织神经细胞炎症反应及细胞凋亡等。

Aβ（β-amyloid peptide） induce neuronal cell damage by increasing the generation of ROS. Therefore, we further assessed the effects of test compounds on living cells. Esculetin was the most potent agent at protecting cells against Aβ induced cell cytotoxisity via inhibiting the generation of ROS or ROS scavenging among the tested coumarin derivatives.

由於Aβ肽造成神经细胞伤害涉及自由基的过度生成，因此我们将上述结构-活性关系结论进一步应用在活体细胞实验的表现，结果显示esculetin可以透过抑制自由基生成或自由基清除作用而降低Aβ所引发的细胞毒性。

Objective To examine the value of Edaravone in the treatment of vascular dementia through ethologic and pathologic changes induced by chronic cerebral ischemiaMethods Wistar rats were randomly divided into sham operated group,model group,edaravone group and duxil groupThe chronic cerebral hypoperfusion models were made by permanent bilateral occlusion of both common carotid arteriesThe memory ability and ultramicrostructure of brain tissue were observedResults The escape latencies in model group were more significantly prolonged than that in the other groups (P＜005)But there was no significant difference between edaravone and duxil groupAnd pathologic changes observed by electron microscope in nerve cell of edaravone group and duxil group and model group were fairly light than that in sham operated groupEdaravone group had increased mitochondrium and frontal membranous synaptic vesicle and more productive metabolic activityConclusions Edaravone can lessen injures of hippocampal neurons loss to improve the spatial learning and memory abilitiesAnd it is proved further that edaravone can treat VD

目的 观察依达拉奉对慢性缺血致血管性痴呆大鼠行为学及形态学的影响，探讨依达拉奉在治疗VD中的应用价值。方法实验分假手术组、模型组及都可喜对照组及依达拉奉治疗组，应用双侧颈总动脉结扎方法制备慢性脑缺血大鼠痴呆模型，观察各组大鼠的记忆功能及脑组织超微结构改变。结果缺血组水迷宫表现同对照组、都可喜组及依达拉奉组相比有显著差异(P＜005)，依达拉奉组与都可喜组相比未见显著差异。同时电镜观察结果也发现依达拉奉组及都可喜组与模型组及假手术组相比神经细胞病理改变较轻，且依达拉奉组线粒体及前膜突触小泡增多，代谢活动较都可喜组旺盛。结论依达拉奉可能通过减轻海马神经元的损伤来改善慢性脑缺血大鼠空间学习记忆障碍，证明依达拉奉对慢性缺血致VD大鼠具有治疗作用。

Adipose-derived mesenchymal stem cells can be induced to exhibit several phenotypic,morphologic,and excitory characteristics consistent with developing neuronal and glial tissue.

脂肪间充质干细胞在体外经诱导，能分化出类似神经元和神经胶质细胞，且具有类神经细胞的功能。

It was found that exogenic gene could start-up the expression of neural stem cell specific marker, nestin, and raise neural polar changes on ES cell.

发现外源性ibeB基因的转染能启动ES细胞表达神经干细胞标志蛋白nestin，并使ES细胞发生神经细胞样极性变化。

It may be the cause for celebration for the 'innovator', but brought about as the result of plugging in or dialling up the living neuronal processes of an enforced experimentee.

它可能是为&发明者&而庆祝的原因，但却是作为对于被强迫的被实验者的活体神经细胞活动过程接上电源和进行刺激的结果而产生的。

It's shown that GIF could not inhibit neuromata extendedly.

表明GIF并不具有广谱的抑制神经细胞瘤株的作用；GIF抑制PC12可能是针对它所具有的胆碱能神经元的某些特性，这一结果对于揭示GIF抑制神经元生长的机制探讨将有所裨益。

But we don't care about Battlegrounds.

但我们并不在乎沙场中的显露。

Ah! don't mention it, the butcher's shop is a horror.

啊！不用提了。提到肉，真是糟透了。