神经细胞

Results: The brain fabric was normal without edema, hemorrhage and necrosis in groupⅠ. The brain parenchyma were loose, perivascular canal became widen, some neurocytes bulk and degenerate in groupⅡ. In groupⅢ, the brain injury aggravateed accompanying the lapse of survival time with extreme cerebral edema at 24 h, and colliquative necrosis in the brain parenchyma aparted at 40 h.

结果：Ⅰ组脑组织结构清晰，无水肿、出血、坏死病灶；Ⅱ组脑实质疏松，小血管周隙增宽，部分神经细胞体积增大变性；Ⅲ组随溺水后存活时间的推移，脑损伤程度逐渐加重，其中24h脑水肿最重，40h个别脑实质小灶性液化性坏死。

Uch var iatio i ize, hape,chemistry, conductio eed, excitatio threshold,and the like a had ee demo trated i erve cell remained egligible i ignificance for any o ible correlatio with the manifold dime io of mental experience.

类似于这些已经被在神经细胞中证明的在大小、形状、化学过程、产生的速度、兴奋阈值及其类似的方面上所发生变化，当他们被用来与大脑的体验以可能的方式联系起来的时候，他们在重要性上仍然是微不足道的。

Objective to evaluate the functional recovery of acute spinal cord injuried rats treated with exogenous wnt-3a signal protein administration and to explore its mechanism.methods moderate spinal cord contusion injury was made in 40 adult sprague dawley rats at t10.twenty rats served as contusion controls(group 1).twenty rats were treated with wnt-3a for three days after injury (group 2).the functional recovery of the rats was observed through basso,beattie,bresnahan open field locomotor score.rats were killed at 14 or 28 days after injury,then spinal cords were removed for histopathological examinations,and the expression of the bromodeoxyuridine plus neural cell markers was stained with immunohistochemical method.results rats of two groups receiving a contusive injury recovered substantial function within 1 week.by 28 days,rats in groups 2 scored 7.0 points better on the bbb scores than rats in group 1 group 2=16.94,after 28 days vs.

目的 研究外源性wnt-3a信号蛋白对脊髓损伤的修复作用，并探讨其作用机制。方法取40只成年雌性sd大鼠在t10节段制备适度脊髓挫伤模型。随机从中取20只为损伤对照组(group 1)，另外20只为损伤治疗组(group 2)。脊髓损伤3天后用wnt-3a蛋白治疗。这些大鼠的功能恢复通过basso、beattie、bresnahan开放视野运动评分来观察。这些大鼠分批在损伤后14天或28天被处死，取出损伤节段脊髓用来组织病理学检查，同时用5-溴脱氧尿嘧啶核苷和神经细胞标记物进行免疫组化染色。结果两组大鼠在伤后一周运动功能有明显的恢复。不过，到损伤后28天，我们观察发现，损伤治疗组中的大鼠bbb评分比损伤对照组中的评分高出7.0分左右(group 2∶16.94±1.18，group 1∶9.89±1.29；p.05)，光镜和电镜检查发现wnt-3a蛋白对髓鞘形成和轴突再生有一定的修复效果。

Methods①The animal model of braincontusion caused by free drop hammer was established.②The injuredtissue of rat brain were stained by TUNEL for apoptosis,immunohistochemistry for Caspase-3 and Feulgen"s for DNA. Image analysistechnique and the statistical method were employed to explorate thetemporal changes of injury time.③The DNA was extracted from ratcontusive tissue of brain and assayed by gel electrophoresis toinvestigate the relationship between the DNA fragmentation and injurytime.④One handred and seventeen cases of death from traumatic braininjury were retrospective researched to investigate the characteristicof TBI in forensic medicine.⑤The contusive tissue of human brain werestained by TUNEL, Caspase-3 immunohistochemistry and Feulgen"s methodsin the same way to analyze and disclose the linear relationship betweentemporal changes with injury time.

方法①建立大鼠自由落体打击脑损伤模型；②对不同损伤时间组的大鼠脑挫伤组织进行TUNEL、Caspase-3免疫组化、Feulgen's DNA染色，结合图像分析技术和统计学分析，探讨脑挫伤后神经细胞凋亡、DNA片段化和含量的时序性变化；③提取大鼠脑挫伤组织DNA进行琼脂糖凝胶电泳分析，观察DNA片段化随损伤时间的变化特点；④对117例颅脑损伤致死案例进行回顾性分析，探讨其法医学特点；⑤选择不同损伤时间的人脑挫伤组织同样进行TUNEL、Caspase-3免疫组化、Feulgen's DNA染色和分析，观察上述指标与损伤时间的关系。

Reverisible middle cerebral artery occlusion without craniectomy in rats.

电针对大鼠脑缺血后脑内神经细胞凋亡的影响。

For the purpose of exploiting the affluent resource of traditional Chinese medicines, rat pheochromocytoma PC 12 cells were used as a model to screen the neuroactive components from natural products. Most of all, the effect of it on inducing the differentiation of PC 12 cells and/or execute the neuroprotective effect challenged by serum-deprivation and oxidative pressure, and its mechanism were studied in this paper.Powered seeds of Cuscuta Chinensis Lam.

本文以利用我国丰富的天然药物资源为目的，用大鼠嗜铬神经瘤细胞PC12作为模型，筛选能模拟NGF的功能诱导PC12细胞分化、对神经细胞有一定保护作用的天然药物，并对其作用机制进行初步探索。

Mn increases free Ca2+ in neurons , Tau treatment could decline significantly the cytoplastic Ca2+ of neurons induced by Mn.

锰可升高胞浆钙离子浓度，加入牛磺酸后可明显降低锰造成的培养神经细胞升高的钙离子浓度。

The neuroprotective mechanism of Davallia mariesii might be due to its phenolic antioxidative activity, and efficiently decreased intracellular oxidative stress and damage induced by 6-OHDA.

结果显示，以海州骨碎补提高6-OHDA诱导B35神经细胞损伤后之存活率最好。其作用机制可能与其总酚类抗氧化活性及能有效地降低细胞内氧化压力及细胞损伤有关。

Davallia mariesii has the hightest protective effects among the aqueous extracts of these three plants on the neuronal oxidative damage induced by 6-OHDA in B35 neuroblastoma cell.

因此，进一步探讨海州骨碎补、鳞轴小膜盖蕨及槲蕨之水抽取物对6-OHDA诱发B35神经细胞氧化损伤之保护效果。

Centrophenoxine combined with point thread embedding protects the nerve cells in the dentale gyrus area of the hippocampus better and improves the learning and memory of the VD model rats more effectively.

在2006年3～8月采用药物和埋线结合的方法，用盐酸甲氯芬酯联合穴位埋线治疗VD大鼠，观察该方法对VD大鼠海马神经细胞及学习记忆的影响，以期为中西医结合治疗VD提供实验依据。

But we don't care about Battlegrounds.

但我们并不在乎沙场中的显露。

Ah! don't mention it, the butcher's shop is a horror.

啊！不用提了。提到肉，真是糟透了。