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After embeding routingly,serial 10μm sections were prepared for modified trichromic staining with improve-ments to manifest axons and myelin sheaths simultaneously,immunohistochemistryand in situ hybridization.

组化染色用新的改良三色法同时显示神经轴突和神经髓鞘。

At present, SC could be obtained and cultured from infancy peripheral nerve or adult Wallerian degenerated nerve.

目前多从幼年动物周围神经或成年动物瓦勒变性神经获取、培养 SC,并与聚乙醇酸、聚乳酸等材料粘附、复合,预构成含 SC的人工神经导管,引导轴突再生。

Only 1 single 5-HT/TPH-like immunoreactive interneuron was localized near the dorsal antennal lobe, contributing to most immunoreactivity in the antennal lobe.

咽下神经节处5-HT/TPH阳性神经元数目相对较多,每侧大约有15个,主要分布于髓区侧细胞体层和腹侧细胞体层,轴突投射进入咽下神经节。

It is widely known that during the development of nervous system, the synapse between neurons and target tissues plays an important role in the survival and maturation of neurons because target tissues or target cells can produce and secrete many kinds of neurotrophic factors and neuroinductive factors which can promote the growth, differentiation and migration of innervating neurons.

在神经系统发育中,神经元与相应靶组织建立的突触联系,是神经元存活和分化的关键因素,因为靶组织或靶细胞可以合成、分泌多种神经营养因子和神经诱向因子,促进支配神经元的生长、分化和迁移。

Results Denatured nerve allografts and muscle autografts supported the higher rates and volumes of axonal regeneration.

结果 微波变性异体腓神经组和微波变性自体骨骼肌组轴突再生率和数量较高,与自体神经组比较差异有显著性。

The FM imaging technique is an advanced and effective method for analyzing synaptic vesicle exocytosis and neurotransmitter release, and can be applied in various synaptic functional studies.

FM 1-43成像技术能够检测钙离子介导的神经递质的释放过程,是一种非常先进并切实有效的检测神经元突触末端神经递质释放过程的手段和方法。

Neurology also influences the surgical approach when there is cord compression from a vertebral body fracture, anterior traumatic disc protrusion, or a facet fragment causing foraminal root compression.

当脊髓压迫来自骨折的椎体,前方破裂突出的椎间盘,或关节突的骨折片引起椎间孔的神经根受压时,神经状态同样能影响手术入路。

Axonal degeneration similar to that observed in KIF 1A mutants has also been reported for several neurodegenerative diseases such as senile dementia and most recently Charcot-Marie-Tooth disease type 2A by another synaptic vesicle precursors transporter KIF1B knockout mice. In some neurodegenerative diseases, neuronal cell death caused by defects in the transport of synaptic vesicle precursors by KIF1A may be involved.

和KIF1A基因剔除鼠相似的轴突变性在一些神经退行性疾病如老年性痴呆的研究中也有发现,特别是最近,与KIF1A一样运输突触小泡前体的KIF1B基因剔除鼠与Charcot-Marie-Tooth二型疾病的关系,更提示了KIF1A基因的功能缺失,与神经退行性病变及神经元死亡的发生有不可分割的关系。

The nerve axon regeneration length was measured 2 weeks after operation. The effects of peripheral nerve regeneration were evaluated by neural electrophysiology, the recovery rate of triceps surae muscular tension and weight and histological assessment 16 weeks after operation. Results All the animals survived till the end of experiment.

术后行大体观察;术后2 周测量神经轴突生长距离;术后16 周行电生理检测,计算术侧坐骨神经运动传导速度恢复率、术侧小腿三头肌收缩力恢复率及湿重恢复率,并行移植神经吻合中段HE 和半薄切片甲苯胺蓝染色观察。

Acute organophosphate poisoning ; Intermediate myasthenia syndrome ; Cranial nerves ; Neck flexor weakness ; Respiratory muscle paralysis ; Repetitive nerve stimulation electromyography ; Postsynaptic transmission block

急性有机磷中毒;中间期肌无力综合征;颅神经;屈颈肌无力;呼吸肌麻痹;重频刺激神经肌电图;突触后传导阻滞

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