神经病理学的

The ubiquitin positive dystrophic neurites and neuronal intranuclear inclusions in frontal and temporal cortex as well as in limbic system might be a pathological basis in the development of psychiatric symptoms.

额叶、颞叶和扣带回皮层出现神经细胞核内包涵体或营养不良性神经突起可能是精神异常的病理学基础。

Despite similar neuropathological characteristics , the behavioral phenotype of Lurcher is less severe than that of staggerer , possibly caused by the slower onset of their neuronal degeneration .

尽管类似的神经病理学特点，小偷的行为是那么严重型比 staggerer 认为，可能由他们的神经元变性发病缓慢造成的。

On the other hand, both these factors in PD and DLB appear to be largely independent from coexistent vascular pathology, except in cases with severe cerebrovascular lesions or those related to neuritic AD pathology.

帕金森病和路易体痴呆的以上影响因素似乎独立于共存的血管病理学改变，除了合并严重脑血管损害或与神经炎性阿尔茨海默病病理学变化相关的患者。

We discuss the definition and limits of the Marburg entity with reference to acute disseminated encephalomyelitis, impact of pregnancy, unusual MRI features, neuropathology and treatment.

我们讨论了急性播散性脑脊髓炎相关的 Marburg 实体的定义和局限，妊娠的影响，罕见的核磁共振成像特征，神经病理学和相关治疗。

Objective: To study the effect of "Yiqitiaoxue, fubenpeiyuan" Acup Therapy on selective neuron lost of Senile Accelerated Mouse P8, a model of AD, treated with acupuncture. Observe the number and sharp of neuron in the hippocampus and the cortex of temporal lobe, activation and accrementition of horizontal cell, as well as the expression of apoptosis relative protein. Discusse the possible mechanism of acupuncture in neuroprotective effect.

目的：以快速老化模型小白鼠SAMP8为阿尔茨海默病(Alzheimer&s disease， AD，老年性痴呆)动物模型，通过观察针刺对该小鼠海马和颞叶皮质神经元数量以及神经元形态、星形胶质细胞的活化增生、凋亡相关蛋白的表达的影响，研究&益气调血，扶本培元针法对AD选择性神经元丢失的干预作用及其机制，为针刺治疗AD提供神经病理学实验依据。

Chapters on symptom management, the molecular genetics and neuropathology of intracranial tumors, the leptomeningeal dissemination of systemic cancer, the epidemiology of brain tumors, and innovative treatment strategies round out the volume.

关于症状管理的章， intracranial 瘤的分子遗传学和神经病理学，系统的癌症的leptomeningeal 传播，大脑瘤的流行病学，和革新的治疗策略圆满体积。 DsyHealth Medicine medicine 健康医学医药

This therapy significantly improved survival rate, induced long-lasting systemic anti-tumor immunity, and resolved the neuropathological abnormalities caused by the tumors, which has been a stumbling block to many promising treatments.

& &这一疗法的显着改善存活率，诱导长期持久的系统性抗肿瘤免疫，并解决了神经病理学异常所造成的肿瘤，已成为绊脚石，许多有前途的治疗方法。

Objectives In order to investigate how does King's Brain pill work or mechanism of King's Brain pill effective in treating AD clinically, we observed effects of extract of King's Brain on neuronal and vascular lesions induced by Aβ combining with praxiology and neuropathology.

目的 运用现代分子神经病理学和行为学技术，围绕炎症假说、淀粉样蛋白瀑布流假说，以神经元和脑血管为保护目标，着眼于AD的关键病理机制Aβ（β淀粉样蛋白沉积和淀粉样血管病变）及其代谢，开展中药补肾活血化痰复方制剂金思维对损伤的神经元和血管的保护作用机理研究，从而探讨其防治老年性痴呆的机理。

Results: Dynamic observation with histopathology and immunohistochemical staining showed that, after the nerve root nerve root compression, the myelinated nerve fibers decreased and there was ultrastructure changes of the nerve fibers, such as Schwann cell cytoplasm swelling and edema, Wallerian degeneration, and demyelinated degeneration of axons.

结果：通过对压迫神经根及神经根节大体观，组织病理学和免疫组织化学的光学显微镜的动态观察，可见神经根压迫后其有随神经纤维数目减少和超微结构的改变，如神经模细胞胞质肿胀和细胞水肿、华勒变性、神经轴突脱髓鞘变等。

It is an important regalator for brain development, nerve location, and synaptic transmission.

过度磷酸化的tau蛋白组成的神经原纤维缠结是AD的主要神经病理学特征之一。

Do you know, i need you to come back

你知道吗，我需要你回来

Yang yinshu、Wang xiangsheng、Li decang,The first discovery of haemaphysalis conicinna.

1〕 杨银书，王祥生，李德昌。安徽省首次发现嗜群血蜱。