神经病理学

The ubiquitin positive dystrophic neurites and neuronal intranuclear inclusions in frontal and temporal cortex as well as in limbic system might be a pathological basis in the development of psychiatric symptoms.

额叶、颞叶和扣带回皮层出现神经细胞核内包涵体或营养不良性神经突起可能是精神异常的病理学基础。

On the other hand, both these factors in PD and DLB appear to be largely independent from coexistent vascular pathology, except in cases with severe cerebrovascular lesions or those related to neuritic AD pathology.

帕金森病和路易体痴呆的以上影响因素似乎独立于共存的血管病理学改变，除了合并严重脑血管损害或与神经炎性阿尔茨海默病病理学变化相关的患者。

Pathological examination showed the obvious edema in myelin sheath,axons and perineurium in 2 w group,and disintegrated myelin sheath,degenerated axons and hyperplastic fibrous connective tissues in the other model groups.

病理学观察：卡压2周组神经束内髓鞘、轴索和神经束膜纤维出现明显水肿，余卡压组可见髓鞘崩解、轴索变性，纤维结缔组织增生逐渐明显。

At histopathologic analysis, neuronal tumors are usually classified as pure neuronal cell tumors (gangliocytoma, Lhermitte-Duclos disease , central neurocytoma) and mixed neuronal-glial tumors (ganglioglioma, desmoplastic infantile ganglioglioma, dysembryoplastic neuroepithelial tumor, ganglioneuroma).

根据组织病理学的分析，神经元肿瘤通常被分为：单纯的神经元细胞肿瘤（神经节细胞瘤，Lhermitte-Duclos 病〔发育不良性小脑神经节细胞〕瘤），中枢神经细胞瘤）和混合性的神经元-胶质肿瘤（神经节神经胶质瘤，成结缔组织性婴儿节细胞胶质瘤，胚胎发育不良性神经上皮瘤，神经节瘤）。

DESIGN: Senile plaques and neurofibrillary tangles were assessed quantitatively in 97 cases from 7 Alzheimer's Disease Centers. Neuropathological diagnoses of AD were also made with four sets of criteria.

设计：评估来源于7个阿尔茨海默氏病中心的97名患者的老年斑和神经纤维缠结情况，之后进行四套标准阿尔茨海默病的神经病理学诊断评估。

This therapy significantly improved survival rate, induced long-lasting systemic anti-tumor immunity, and resolved the neuropathological abnormalities caused by the tumors, which has been a stumbling block to many promising treatments.

& &这一疗法的显着改善存活率，诱导长期持久的系统性抗肿瘤免疫，并解决了神经病理学异常所造成的肿瘤，已成为绊脚石，许多有前途的治疗方法。

Objectives In order to investigate how does King's Brain pill work or mechanism of King's Brain pill effective in treating AD clinically, we observed effects of extract of King's Brain on neuronal and vascular lesions induced by Aβ combining with praxiology and neuropathology.

目的 运用现代分子神经病理学和行为学技术，围绕炎症假说、淀粉样蛋白瀑布流假说，以神经元和脑血管为保护目标，着眼于AD的关键病理机制Aβ（β淀粉样蛋白沉积和淀粉样血管病变）及其代谢，开展中药补肾活血化痰复方制剂金思维对损伤的神经元和血管的保护作用机理研究，从而探讨其防治老年性痴呆的机理。

Results: Dynamic observation with histopathology and immunohistochemical staining showed that, after the nerve root nerve root compression, the myelinated nerve fibers decreased and there was ultrastructure changes of the nerve fibers, such as Schwann cell cytoplasm swelling and edema, Wallerian degeneration, and demyelinated degeneration of axons.

结果：通过对压迫神经根及神经根节大体观，组织病理学和免疫组织化学的光学显微镜的动态观察，可见神经根压迫后其有随神经纤维数目减少和超微结构的改变，如神经模细胞胞质肿胀和细胞水肿、华勒变性、神经轴突脱髓鞘变等。

It is an important regalator for brain development, nerve location, and synaptic transmission.

过度磷酸化的tau蛋白组成的神经原纤维缠结是AD的主要神经病理学特征之一。

Since those early days, the discipline has taken full advantage of each new wave of technology, with neuropathologists always being well-represented among the earliest adopters of novel technical innovations.

从此，神经肿瘤病理学得到了长足的进步，这是因为神经病理学家通常是早期接受新技术改革者中的最好代表。

Do you know, i need you to come back

你知道吗，我需要你回来

Yang yinshu、Wang xiangsheng、Li decang,The first discovery of haemaphysalis conicinna.

1〕 杨银书，王祥生，李德昌。安徽省首次发现嗜群血蜱。