神经原的

Firstly, we introduce the basic theory and methods for realization of SOFM. Subsequently we improve the arithmetic and implement the feature extraction of raw data using K-L translation, select the eigenvector. Combining C-avarage and ISODATA arithmetic, classify the eigenvector, according to the methods of this dissertation, in lower layer, set nerve cell and unite or delete nerve cells in middle layer, to improve the anti-huise- and robust.

论文首先介绍了SOFM的基本原理和实现方法，接着在其基础上对具体算法进行改进，用K-L变换对原始数据进行特征提取，选取出特征变量，结合C-均值和ISODATA算法对特征变量进行预分类，按照本文中介绍的方法在底层预置神经元，在中间层对神经原进行合并和删除，加强了该网络的抗干扰性和稳定性。

It was the patho-physiologic progress of neurocytic degeneration and necrosis that the change of protein and phospholipid constituent in neurocytic membrane and the decrease of fluidity of neurocytic membrane in chicken with encephalomalacia

发病雏鸡脑神经细胞膜蛋白组分、磷脂组分的改变和膜流动性的降低是神经原变性和坏死的病理生理过程。

The identity of these genes also shed light on the mechanisms of motoneuron degeneration and revealed that overlapping motoneuron phenotypes might be caused by heterogeneous molecular mechanisms.

确认这些基因也揭示了运动神经元退行性变的机制，异型的分子机制可能引起运动神经原表型的重叠。

Alzheimer disease: A primary degenerative cerebral disease of unknown etiology with characteristic neuropathologcal and neurochemical features, including cortical atrophy with neurofibrillary tangles and neuritic argentophilic plaques, and a marked reduction on the enzyme choline acetyltransferase, in acetylcholine itself, and in other neurotransmitters and neuromodulators.

阿尔茨海默病：一种病因未明的原发退行性大脑疾病，具有特征性的神经病理和神经生化改变，包括伴有神经原纤维缠结和神经炎性嗜银斑的皮层萎缩，胆碱乙酰转移酶、乙酰胆碱本身、以及其它神经递质和神经调质的明显减少。

Hyperphosphorylation of tau protein composition of the original nerve fibers tangle AD is the main feature of neuropathology.

过度磷酸化的tau蛋白组成的神经原纤维缠结是AD的主要神经病理学特征之一。

BACKGROUND: One of the key neuropathological changes in Alzheimer disease is that neurofibrils over phosphorylated cytoskeletal protein composed of entwist together, and the phosphorylation of τ protein can be catalyzed by cyclin dependent kinase 5 (CDK5),however whether the phosphorylation of neurofilaments can be catalyzed by CDK5, as well as its role in the pathogenesis of Alzheimer diseases is less acknowledged.

背景：阿尔茨海默病的主要神经病理学特征之一是由过度磷酸化的细胞骨架蛋白如(o，神经细丝组成的神经原纤维缠结，细胞周期蛋白依赖性蛋白激酶5可催化蛋白的磷酸化，但神经细丝是否可被细胞周期蛋白依赖性蛋白激酶5磷酸化及其在阿尔茨海默病发病中的作用却知之甚少。

It is an important regalator for brain development, nerve location, and synaptic transmission.

过度磷酸化的tau蛋白组成的神经原纤维缠结是AD的主要神经病理学特征之一。

PART Ⅱ IMMUNOPATHOLOGICAL EVIDANCE OF SIALIC ACID STRUCTURE IN CAMPYLOBACTER JEJUNI AS THE CRITICAL ANTIGEN TO INDUCE PERIPHERAL NEUROPATHY To demonstrate the critical role of NANA structure in the pathogenesis of allergic peripheral neuropathy induced by Campylobacter jejuni and provide immunopathological evidence to confirm the supposition of molecular mimicry and cross-immunity between CJ LPS and gangliosides in nerve.

LPS免疫后第2周实验豚鼠的免疫血清中，抗LPS IgG抗体滴度均明显增高，第3周达高峰，第5周仍维持在峰水平，野生株和突变株LPS免疫血清中的抗-野生株LPS IgG抗体滴度较各自免疫前分别增高8倍和4倍，抗-突变株LPS IgG抗体滴度则较各自免疫前分别增高6.5倍和15倍；(2)全身免疫后第3周和第5周，野生株LPS免疫血清中检测到较免疫前增高6倍的抗-GM1 IgG抗体，而NANA缺失的突变株LPS免疫血清中一直未检测到抗-GM1 IgG抗体；(3)野生株LPS免疫组中有17.3％的坐骨神经原纤维发生以轴索变性为主（占65％）的免疫性损伤，与突变株LPS免疫组和对照组比较均有显著性差异。

In the negative and interrogative forms, of course, this is identical to the non-emphatic forms.

。但是，在否定句或疑问句里，这种带有"do"的方法表达的效果却没有什么强调的意思。

Go down on one's knees;kneel down

屈膝跪下。。。下跪祈祷