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神经压迫

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Waist leg pain and sciatic nerve are painful , receive because of waist leg muscle ligament, tendon membrane and nerve not proper besides put violence or have borne a heavy burdon for a long time, chill wet heat soaks to enter, make its organization tear to raid sprain , oppression or chronic strain, fiber venereal disease changes and agedness degraded venereal disease changes , bone hyperplasia makes its interverbebral disc outstanding, leg joint inflames , is injured position hyperemia, air Zhi blood Yu, oedema, inflammation and adhesion, and deliver spine nerve root and sciatic nerve to receive press pain.

腰腿痛和坐骨神经痛,多因腰腿肌肉韧带、筋膜和神经受不适当的外置暴力或负重过久,风寒湿热浸入,使其组织撕袭扭伤,压迫或慢性劳损,纤维性病变及老年性退化性病变,骨质增生,使其椎间盘突出,腿关节发炎,受伤部位充血,气滞血瘀,水肿,炎症及粘连,而致脊神经根、坐骨神经受压疼痛。

Methods:The nucleus pulposus of coccygeal vertebra was applyed to the L 4 and L 5 nerve roots of the rat without compression and the mechanical withdrawal threshold and thermal withdrawal latency were observed and CSEP of hindlimbs were recorded in 3 days, 1 week,2 weeks and 4 weeks respectively after surgery in order to show ...

取大鼠自体尾部的髓核无压迫下放置在L4 和L5神经根上,制成髓核突出动物模型,分别在术后3d、1、2及 4周观察大鼠术侧肢体机械刺激敏感性和热刺激敏感性的变化,并引出大鼠后肢CSEP ,观察术侧肢体CSEP的变化。

Autogenous nucleus pulposus was removed from the coccygeal vertebral and placed into the back of left L(5~6) nerve root,The modle of the no...

结论杜仲腰痛丸有减轻非机械压迫性髓核对神经根损伤后所导致的机械痛觉过敏,提高痛阈的作用。

Methods: 50 normal Wistar male rats were randomly divided into five geoups: the sham operated group, injury model group, shenjindan capsule treat group, high dose and low dose group (duzhongyaotong pill treat, D and E)(n=10each). Autogenous nucleus pulposus was removed from the coccygeal vertebral and placed into the back of left L5~6 nerve root, The modle of the non-compressive nucleus pulposus protrusion was created by transplanting autogenous nucleus pulposus in the back of left L5~6 nerve roots. At tow weeks, the SP in dorsal root ganglion was determined by immunohistochemistry.

取健康Wistar大鼠50只,随机分为假手术组、模型组、对照组、高剂量组(D组、腰腿理痛散高剂量组)、低剂量组(E组、腰腿理痛散低剂量组),每组10只,将大鼠自身的尾椎髓核取出移植于左侧硬膜外腔L5、L6神经根背侧,造成非压迫性大鼠自体髓核移植硬膜外模型,通过免疫组化的方法测定大鼠2周时DRG中CGRP的变化。

Genetic factors,obesity, high salt consumption, and alcohol may be involved in the pathogenesis of essential hypertension. Jannetta et al. suggested that essential hypertension might be associated with vascular compression of the left rostral ventrolateral medulla and the root entry zone of cranial nerves Ⅸ and Ⅹ by ectatic loops of posterior fossa arteries,described as neurogenic hypertension.

原发性高血压的发生可能与遗传、肥胖、高盐饮食、饮酒等综合因素有关。1979年Jannetta等提出由于后颅窝异常血管袢压迫延髓左侧腹外侧、舌咽神经与迷走神经根入脑干区(Root entry zone, REZ)而引起高血压即神经源性高血压的假说以来,不少学者从临床及实验研究证实了Jannetta的假说。

Methods Rat spinal cord injury models were established by aneurysm clip compression, and nestin expression and proliferation of ependymal cells at different times were shown with pathological and immuno-histochemical staining.

应用动脉瘤夹压迫建立大鼠脊髓损伤模型,通过组织病理学及免疫组织化学方法检测不同时段室管膜细胞的反应性增生和神经外胚层多潜能细胞特异性抗原nestin的表达。

Objective: To evaluate the MRI findings and diagnostic value of neurovascular compression in patients with hemifacial spasm.

目的:探讨面肌痉挛患者神经血管压迫病因的MR表现及其诊断价值。

MRI had higher diagnostic value of vascular compression or contact in patients with hemifacial spasm.

MR能较清晰显示面听神经与毗邻血管之间的关系,为临床医师提供术前评估和指导治疗有极其重要的意义,MRI在面肌痉挛神经血管压迫病因诊断中具有较高的价值。

Results:There were 12 cases of peripheral sensory neuropathy,12 cases of myasthenic syndrome,6 cases of spinal cord compression,4 cases of brain stem encephalitis,3 cases of subacute cerebellar degeneration.3 cases were diagnosed in 1 week after appearing neurological symptoms,and 3 cases were diagnosed in 1 month,the other 16 cases were diagnosed after many months or years while the neuropathy was existing.

结果:癌性感觉性周围神经病12例,肌无力综合征8例,脊髓压迫症6例,脑干脑炎4例,亚急性小脑变性3例。3例在出现神经系统症状后1周内确诊,3例在1月内确诊,其余16例均在神经病变持续存在数月至数年后经多次检查确诊为肿瘤。

"Tuina Manipulation combined with Exercise Therapy" treating the lumbar disc herniation cases has a significant long residual effect. Analying the anatomy, physiological characteristics, mechanics analysis of the waist and LDH's modern research, its mechanism may adjust to lumbar compliance; ease cramps; change the relative positions of disc and nerve roots, reduce or lift the oppression; correct facet dislocation; release adhesions, improve circulation and promote absorption and repair of the inflammation and edema; analgesic pain threshold or enhance the organization; improve the waist myodynamia; reconstruct the balance of the waist and so on.

结合腰部椎体、肌肉及韧带等组织的解剖、生理特点、力学分析和腰椎间盘突出症的现代研究分析,其机制可能是调整腰椎顺应性,缓解痉挛;改变突出髓核与神经根的相对位置,减轻或解除压迫;纠正小关节错位及滑膜崁顿;松解粘连,改善循环,促进炎症、水肿的吸收和损伤修复;镇痛与提高组织痛阈;改善腰部肌力;促进人体生物力学结构重构;恢复腰椎及其周围力学平衡等几个方面。

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