神经元的

Light and electron microscopy showed central chromatolysis of motor neurons in the lumbar cord from 1 week after the start of compression.

研究结果：光学和电子显微镜检查表明从压迫1周后出现腰髓运动神经元的中心染色质溶解。3周后，在前角可见到一些神经元的凋亡。

An adaptive PID controller for thrust speed of a hydraulic thrust system in shield machine based on the single neuron structure was designed on the basis of the fact that the single neuron features the PID characteristics.

在分析单神经元PID特性的基础上，设计了基于单神经元的盾构推进液压系统速度自适应PID控制器。

The resting membrane potential and input resistance of DCN neurons were -60±6 mV (range -53 to-70 mV, n=19) and 484±298 MΩ(range 88-1635 MΩ, n=70), respectively.

DCN神经元的静息膜电位和输入阻抗分别为-60±6 mV（n=19）和484±298 MΩ（n=70）。大部分神经元的电流电压曲线呈现内向整流。

The probe was tone burst and its frequency was the same as neuronal characteristic frequency . The effects of different maskers on the minimum threshold, response latency, dynamic range and firing rate of the mouse inferior colliculus neurons were observed in the experiment.20 healthy adult mice 20-25 g.

探测声频率为神经元的特征频率(characteristic frequency， CF)，掩蔽声分别为和神经元CF相同的短纯音掩蔽声和上扫调频掩蔽声(up-sweep frequency modulation masker)。

Change of sodium pump activity is involved in hypoxic injury in rat cortical neurons. High affinity sodium pump is re lated to the protection of hypoxic injury and low affinity sodium pump is related to the hypoxic injury.

Na，K-ATP酶活性改变参与了皮质神经元的缺氧性损伤，其中高汞和力Na，K-ATP酶与皮质神经元缺氧性损伤保护作用有关，而低亲和力Na，K-ATP酶则与其缺氧性损伤有关。

Result and Conclusion: TrkA was expressed and distributed ubiquitously in both eytoplasm and dendrites of spinal cord.

结果与结论：TrkA广泛分于脊髓各层神经元及灰质内，并且在神经元的胞体和树突中均有表达。

Methods Three groups of spinal cord paraffin specimens of macaca mulatta were collected, every group including 4 cases.

目的 探讨GDNF与MSCs源性神经元样细胞联合移植对猕猴脊髓损伤后前角运动神经元的协同保护作用及机制。

Among 24 excitatory neurons of NRM, the spontaneous discharges of most of them were obviously enhanced by nociceptive stimulation.

在24例兴奋型神经元中，刺激尾核头部能够激活大多数NRM神经元的自发放电和抑制其伤害感受性反应。

It can be concluded that theophylline can prevent cerebellar granule neurons from apoptosis induced by depolarizing concentration of KCL deprivation in culture.

认为茶碱对撤去极化浓度氯化钾诱发小脑颗粒神经元的凋亡有保护作用，此作用产生除与其升高小脑颗粒神经元内cAMP有关外，还有其它机理。

GHBA can protect hypoxia-reoxygenation neurons through upregulating GABA and GABARα1 positive neurons.

GHBA可通过上调GABA和GABARα1阳性神经元的表达水平对缺氧复氧皮层神经元发挥保护作用。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。