神经元的

In the present study we transfected F-GFP into cultured hippocampal neurons at 5 days in vitro(DIV 5).Then ASICs antagonists were applied to the neuronal medium to inhibit the function of ASICs.Dendritic growth and arborization of cultured hippoeampal neuron were observed in DIV8 and DIV14. We attempted to determine whether and how ASICs regulate dendritic development.

在本研究中，我们在体外培养第五天(days in vitro，DIV5)的原代海马神经元中转染定位在膜上的GFP，随后加入ASICs拮抗剂抑制ASICs的功能，观察DIV8和DIV14这两个时间点海马神经元的树突生长、分支复杂程度，来研究酸敏感离子通道是否会影响海马神经元的树突发育。

Research concerning research status and development of CNTF was selected, including changes of CNTF following nerve injury; the repair mechanism and protection of CNTF on neurons; regulation on reparative process; the recovery of axoplasmic transport function of axoplasm, as well as the regeneration on injured nerves.

纳入与神经损伤与睫状神经营养因子研究现状与发展密切相关的研究，包括神经元损伤后的变化；睫状神经营养因子对神经元的修复机制及保护作用，对修复过程的调控，对神经元轴浆运输功能的恢复，对损伤神经的再生作用。

GDNF has been reported to exhibit trophic actions on various neurons of central nervous system and peripheral nervous system .

GDNF对中枢及外周的多种神经元有神经营养作用，与目前已知的神经营养因子相比，GDNF对受到损伤的多巴胺能神经元和运动神经元的促存活作用最为显著。

Compared with quinine stimulability, discharges of vagus efferent never in both CTA rats and control rats had no difference when sucrose stimulability was was given. 5 When the same sucrose stimulability was given, FLI neurons of SON and DMV in the CTA rats had much more expressions than that in the control rats.

5给予蔗糖刺激后，CTA组大鼠在SON和DMV内FLI神经元的表达较多，且表达远高于正常组大鼠；而且味觉厌恶建立后，蔗糖刺激和水刺激引起的SON核和DMV内FLI神经元的表达也有差异，蔗糖刺激引起的FLI神经元的表达要高于后者。

The plasticity of NSCs was detected by the differentiating test. HESCs were induced into dopaminergic neurons by means of adding Sonic Hedgehog Peptide and fibroblast growth factor eight (FGF8) early under the condition of mocked microenvironment and different development stage of dopaminergic neurons in vivo ,meanwhile made comparison with traditional method . The marker of dopaminergic neurons was tested by immunocytochemistry and RT-PCR, the content of dopamine and its derivation homovanillic acid were measured by HPLC-ECD.

模拟体内多巴胺神经元分化发育的不同阶段及微环境，采用早期添加音猥因子及成纤维细胞生长因子-8(FGF8)的方法，诱导hESCs定向生成多巴胺能神经元，并与传统的诱导方法相比较，免疫荧光细胞化学和RT-PCR法检测多巴胺能神经元的标志，高效液相色谱法检测诱导后细胞及细胞培养上清液中多巴胺及其代谢产物高香草酸的含量。

And the Akt of activation carries gene of phosphation Bad, GSK-3, transcribe familial, IB, Caspases make close to wither dies gene misses work, have inhibitory nerve thereby yuan wither dies reach stimulative nerve yuan surviving action, reduce AD nerve then yuan many and missing, the pathology that improves AD changes.

而活化的Akt通过磷酸化Bad、GSK-3、转录因子家族、IB、caspases等使促凋亡基因失活，从而起到抑制神经元凋亡及促进神经元存活的功能，进而减少AD神经元的大量丢失，改善AD的病理变化。

The present results showed that both cytoplasm and membrane of neuronal soma and dendrite expressed NKCC1, while KCC2 only expressed in membrane of soma and dendrite. The results also indicated that the dendrites rather than the somata of neurons expressed more NKCC1 in adult rat neocortex, while the level of KCC2 expression in the dendrite membrane was similar to that in the membrane of somata.

结果显示：成年大鼠皮质神经元的胞浆和细胞膜均有NKCC1的表达，而KCC2主要表达在神经元胞体和树突膜上，其中NKCC1在神经元树突上的表达水平比胞体高，而KCC2的表达水平在树突和胞体膜上没有明显差异。

Methods: The cerebral cortical astrocytes and neurons of rats were cultured and the model of simulated cerebral ischemia and reperfusion was set up in vitro. The activity, survival rate, death rate of the neurons, leakage rate of lactate dehydrogenase and the activity of nitric oxide synthase of cerebral cortical neurons were measured after 18 boors of reperfusion.

分离纯化培养大鼠大脑皮质Ast和神经元，体外模拟脑缺血再灌注损伤模型，用ACM培养损伤后的神经元，测定其神经元的活性、存活率、死亡率、培养液乳酸脱氢酶的漏出率和NOS强阳性细胞的表达量。

The mPSCs induced by GABA released from the presynaptic boutons could be completely and reversibly blocked by 5μM bicuculline, which is the specific antagonist of GABAA receptor. These results indicate that GABA released from the presynaptic boutons may produce modulatory effects through binding with GABAA receptor on postsynaptic Vme neurons.

以上结果表明：突触前终扣内释放的GABA与分布于突触后的GABAA受体结合从而发挥对Vme神经元的调控作用，GABAA受体激活后，氯离子通道开放，Vme神经元胞体内外氯离子流动构成了GABA对Vme神经元的作用基础。

This course deals with the specific functions of neurons, the interactions of neurons in development, and the organization of neuronal ensembles to produce behavior.

本课程旨在探讨神经元的特定功能、发育过程中神经元的相互作用和引发行为的神经元集合的组织状态。

But we don't care about Battlegrounds.

但我们并不在乎沙场中的显露。

Ah! don't mention it, the butcher's shop is a horror.

啊！不用提了。提到肉，真是糟透了。