神经元的

Nowadays, as more and more attention are paid to glia, which is considered to play an important role in epileptic pathogenesis,In this study, we chose fluoroacetate, a reversible neuroglia metabolic inhibitor as well as a rodenticide that can induce epileptic seizures, to set up a new epileptic animal model by acting on neuroglia to induce seizures.

目前常用的急性化学致痫模型其致痫机制均是作用于神经元，通过对CNS递质的作用来诱发癫痫。虽然，随着胶质细胞在癫痫的发病机制中的作用日益受到关注，但由于缺乏作用于胶质细胞的癫痫模型，使研究胶质细胞在癫痫发病机制中的作用及其与神经元的关系带来了障碍。

Through immunocytochemistry study, we found that the α3 and/or α5 subunits of nicotinic acetylcholine receptor are predominantly expressed in the oxytocinergic neurons. Our finding is quite different from the conclusion of the British scholar Dr. Mason's work in 1980s. We also found in this study that, the α3 and/or α5 subunits of N-AChR can also be expressed on the axons of oxytocinergic neurons, and these immunoreactive axons could be seen projecting to the median eminence and further to the posterior end of neurohypophysis. In the hypothalamus of the 7-day postnatal pup rats, the expression patterns of α3 and/or α5 subunits of N-AChR are similar to that in adult rats.

我们用免疫组化的方法发现，α3和/或α5型烟碱型乙酰胆碱受体(Nicotinic acetylcholine receptor，N-AChR)亚单位在大鼠下丘脑OT能神经元中呈优势性的表达，这与上世纪八十年代英国学者Mason类似工作的结论不一致；而且我们还发现α3和/或α5型N-AChR亚单位还表达在OT能神经元的轴突，此阳性免疫反应的轴突一直延续到正中隆起和神经垂体的末端；在新生7天大鼠的下丘脑，α3和/或α5型N-AChR亚单位的分布类似成年鼠。

In order to improve the arithmetic, a king of self - correcting gain K adaptive PID control way is presented by using recursion formula of gain K in adaptive PSD controller.

并推导了为改进其应用上的不足，将自适应PSD控制器中递推计算增益K的方法应用到单神经元的自适应PID控制，构成具有自动调整增益K值的单神经元自适应PID控制器的学习算法。

It has been found that ectogenic GDNF has nervous protective effec,but ectogenic GDNF cannot enter blood brain barrier.

传统观点认为，星形胶质细胞是神经系统的支持细胞，构成神经组织的网架而起支持作用，与神经元的解剖位置关系紧密，参与中枢神经系统的免疫反应，参与构成血脑屏障，目前研究发现，脑缺血时星形胶质细胞异常活跃，通过合成多种蛋白质、细胞因子和神经营养因子等对神经元起保护作用，对缺血性脑损伤的发展和转归具有重要意义。

It's shown that GIF could not inhibit neuromata extendedly.

表明GIF并不具有广谱的抑制神经细胞瘤株的作用；GIF抑制PC12可能是针对它所具有的胆碱能神经元的某些特性，这一结果对于揭示GIF抑制神经元生长的机制探讨将有所裨益。

The differentiation of dopaminergic neurons of substantial nigra completes under the induction of striatum and its function realizes by synapse with striatal neurons.

黑质中多巴胺能神经元的分化是在纹状体的诱导下完成的，其功能活动也是通过与纹状体神经元建立的突触连接而实现的。

METHODS: Silicon chambers were sutured respectively to the distal stumps of motorial branch of femoral nerve and saphenous nerve to collect NRCF, namely MD-NRCF and SD-NRCF. The rats cortex motoneuron were divided into 4 groups and cocultured with MD-NRCF, SD-NRCF, b-FGF and serum-free medium respectively. The cultured cells were photoed under phase-contrast microscope, their longest neurites and cell-body areas were measured by cell image processing computer system.

建立运动及感觉神经再生室模型，收集术后7天的运动及感觉神经再生条件液(MD-NRCF，SD-NRCF)，将两者以及bFGF分别加入分散培养的SD大鼠运动神经元的DMEM无血清培养基中，在倒置相差显微镜下观察并摄片，运用细胞图像分析系统测量各组细胞的胞体面积及最长突起长度；并采用MTT法检测各组神经元的细胞活性。

Methods: The study was designed to observe SS and influence of serum CD_4~+、CD_8~+、IL-2、IL-6、TNF-α, splenic lymphocyte transformation rate, the expression of SS、SSTR-2 in spinal cord、PAG、NRM and hypothalamus the coexistence of SS and enkephalinergic neuron in back corner of spinal cord, the coexistence of SS and 5-HT neuron in NRM by using methods of behavioral nociceptive threshold test, measure of foot's cubage, flowcytometry, MTT, radioimmunity, HPLC-ECD, immunohistochemistry, laser confocal microscope on FAC-induced hyperalgesia.

以弗氏完全佐剂诱发的炎症痛模型大鼠为研究对象，应用行为学痛阈观察、足跖容积测定、流式细胞检测法、MTT法、放射免疫法、高效液相电化学法、免疫组织化学技术法、激光共聚焦技术等方法，观察SS和电针对痛阈、大鼠CD_4~+、CD_8~+、IL-2、IL-6、TNF-α水平和脾淋巴细胞转化率等指标的影响，以及SS、SSTR-2在脊髓和PAG、NRM及下丘脑部位的表达、在脊髓SS与脑啡肽能神经元的共存现象及在NRM中与5-HT能神经元的共存情况。

The characteristics of recovery cycles in inferior collicular neurons of leaf-nosed bat and effect of the recovery cycle on the following pulse repetition rate were studied using mimic CF-FM sound stimuli emitted by free flying bat. 93 IC neurons were obtained from IC of five bats with normal hearing.

为探究恒频-调频蝙蝠下丘神经元恢复周期特点及其对声脉冲跟随率的影响，实验采用模拟的大蹄蝠自然状态下的恒频-调频发声信号为声刺激，在5只听力正常的大蹄蝠上记录了下丘神经元的声反应和恢复周期（n = 93）。

Factors that influence the evoked activity of RVLM neurones may affect the results of integration.

既然RVLM神经元对信息的整合取决于信息的性质和信息到达的时间，那么影响这两者的因素都可能影响RVLM神经元的最后整合结果。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。