神经元的

2 The result of electron microscope cytochemistry stain: The positive production of ACPase was present black lead phosphate deposit with high electron-dense under light microscope, located in cytoplasm or apophysis mainly. Lysosome was located in cytoplasm in the earlier of differentiation, then was present in apophysis with it grew.

4.2 电镜酶细胞化学染色结果：电镜下ACPase阳性产物为电子致密度高的黑色磷酸铅沉淀，主要分布于神经元的胞浆中或突起内，神经元分化的初期，溶酶体集中分布于胞质内，随突起的长出，可见在突起内出现。

Objective By using Alzheimer's model rats induced by AlCl3,the effects and mechanism of Ginkgo flavone were investigated.Methods Ginkgo flavone were given to rats by oral for seven weeks.Then,the ability of study of mice were determined by step-down test and the number of neurons in the Bregma hippocampus containing β-APP proteins were counted.

目的 研究银杏黄酮对三氯化铝所致痴呆模型鼠学习记忆能力和海马内β-APP阳性神经元的影响，以期探讨银杏黄酮抗老年性痴呆的作用机制方法用三氯化铝致老年性痴呆模型大鼠实验对象，以其学习记忆能力、背海马和齿状回内β-APP阳性神经元的量为指标，全面考察银杏黄酮抗老年性痴呆的作用。

objective by using alzheimer's model rats induced by alcl3,the effects and mechanism of ginkgo flavone were investigated.methods ginkgo flavone were given to rats by oral for seven weeks.then,the ability of study of mice were determined by step-down test and the number of neurons in the bregma hippocampus containing β-app proteins were counted.results to the model rats,ginkgo flavone 100,200,400mg?

目的 研究银杏黄酮对三氯化铝所致痴呆模型大鼠学习记忆能力和海马内β-app阳性神经元的影响，以期探讨银杏黄酮抗老年性痴呆的作用机制。方法用三氯化铝致老年性痴呆模型大鼠为实验对象，以其学习记忆能力、背海马和齿状回内β-app阳性神经元的数量为指标，全面考察银杏黄酮抗老年性痴呆的作用。

Injury to the corticospinal tract, which has facilatory influence on motor neurons in the spinal cord, causes the "negative" manifestations including muscle weakness, fatigability, and decreased dexterity (Rossi, 1994). These features are the major determinant of motor disability associated with upper motor neuron lesions.

痉挛在极易影响运动神经元的脊髓灰质损伤中带来的阴性效应包括肌肉无力、易疲劳、灵巧性下降，这些特征成为与上运动神经元损伤并发的运动残疾的主要决定因素。

It was found that SO2 derivatives increase the amplitude of IA in CA3 neurons, and inhibit the inactivation of IA. After SO2 derivatives application, normal sensory physiology of hippocampal neurons was affected. This variation caused by SO2 could underlie the toxical modulation of sensory input to the central nervous system.

说明SO2代谢衍生物可增大大鼠海马CA3区神经元IA电流，延长A-电流的失活时间，从而影响海马神经元的膜生理感应，这可能是SO2影响神经细胞功能的机理之一。

The main results and conclusions are as follows: 1. Culrured neurons were damaged by simulated cerebral ischemia and reperfusion in vitro: Results:(1) The activity of the darnaged neurons was decreased, the survival rate was descent.(2) The transudation rate of LDH in cultured medium increased obviously.

体外模拟脑缺血再灌注造成了培养神经元的损伤：表现为：(1)受损神经元的活性降低，细胞存活率下降；(2)细胞培养液LDH的漏出率明显提高；(3)细胞死亡率显著上升；(4)NOS染色强阳性细胞数在缺血期和再灌注早期明显增多。

The steady-state inactivation curve for TTX-R sodium channel was shifted by SO_2 derivatives in the depolarizing direction as that for TTX-S sodium channel.

SO_2通过作用钠电流，增大了神经元的兴奋性，尤其是伤害性神经元的兴奋性，这可能是SO_2影响神经细胞功能的作用机理之一。

In order to explore the relationship between the echolocation call or signal of leaf-nosed bats and frequency tuning of inferior collicular neurons, the echolocation calls of the leaf-nosed bats were recorded using bat detector and frequency tuning curves of inferior collicular neurons were obtained by extracellular recording.

采用超声监测仪录制超声信号和细胞外电生理记录下丘神经元的频率调谐曲线(frequency tuning curves， FTCs)的方法，探讨了大蹄蝠回声定位信号与下丘(inferior colliculus， IC)神经元频率调谐之间的相关性。

In order to study the architectuer of human cardiac neurons, the location and mumber of human cardiac ganglionated plexuses and their nervous connections were investigated.

为了研究人心脏神经元的构筑，观察了人心脏神经节的分布数量和神经连接。光镜下发现大量神经节（550±149）或神经元分布在人的心房和心室表面。

Objective: To observe the distribution pattern and morphology of NOS positive neurons in the olfactory bulb of the wild hedgehog .

用依赖还原型辅酶Ⅱ的黄递酶组织化学方法显示NOS阳性神经元在刺猬嗅球的分布和形态，观察野生刺猬嗅球内一氧化氮合酶阳性神经元的分布和形态。

For a big chunk of credit-card losses; the number of filings (and thus charge-off rates) would be rising again, whether

年美国个人破产法的一个改动使得破产登记急速下降，而后引起了信用卡大规模的亏损。

Eph. 4:23 And that you be renewed in the spirit of your mind

弗四23 而在你们心思的灵里得以更新