神经元

BACKGROUND: After amyotrophic lateral sclerosis, motor neurons have changes in neural degeneration, but the precise mechanism is not clear.

摘要背景：肌萎缩侧索硬化后运动神经元存在神经变性改变，但其确切机制不明，内源性神经干细胞被认为是未来能有效解决此病的治疗方法之一。

Methods Amyotrophy, somatasthenia, fasciculation, dysarthria and acataposis were the major clinical symptom of MND patients. The conclusion suggested that MND must be treated mainly by reinforcing the kidney and invigorating the spleen and invigorating spleen to replenish qi, removing blood stasis dredge collaterals as adjunctive therapy.

结果运动神经元疾病临床以肌肉萎缩、肢体无力、肌束震颤、构音困难、吞咽困难为主要表现，治疗时应以健脾补肾、补中益气为主，加用活血通络之品。

These anaesthetics block sodium channels and thereby the excitability of all neurons, not just sensory neurons.

它们的亲油性使它们能进入几乎任何神经元，在那里，它们会不加甄别地阻断细胞膜中的钠通道。

After administration of Panax Tonifying Soup Mix with different doses, the number of neurons in brain cells was approximate normal value, and the anaplasia was also notably improved; hepatocyte proliferation, degeneration and necrosis were evidently suppressed; the effects in high dose group were most remarkable; and noticeable dose-effect relationship was observed.

给予不同剂量人参养荣汤干预治疗后，脑细胞神经元数目接近正常，退行性变化明显改善；肝细胞增生、变性及坏死显著被抑制；其中以高剂量作用显著，并且具有显著的量效关系。

Aim: To observe the changes of the expression of NSE, COX-2 in cerebral tissue in rats withgas toxicosis of acute Anchovy degeneration, and to discuss its molecular mechanism.

目的：观察鱼腐败气体中毒后大鼠脑组织神经元特异性烯醇化酶、环氧合酶(COX-2)表达的变化，探讨中毒的分子机制。

Objective: To study the effects of androgen on the apoptosis of the neuron and astrocytes after cerebral ischemia injury.

目的： 研究睾丸酮对小鼠脑缺血损伤后神经元和星形胶质细胞凋亡的影响。

However, the effects of prolonged exposure to local anesthetics at a small concentration have never been studied.

能而，长时间暴露在低浓度的局麻药对神经元的影响未见道。

There was an obvious increase of insulin receptor gene and protein in the cerebral ischemia reperfusion tissues in rats. The positive cells such as neuron, neuroglia cells, vascular endothelium cells possibly participated in regulating the processes of energy metabolism of cells and neovascularization. It implied that the high expressions of insulin receptors in vascular endothelium cells in ischemic zones possibly participated in regulating the pathophysiological changes of the blood vessels in ischemic zones; The higher expressions of insulin receptors in different cells were possibly one of the self-protective machanisms in which the ischemic tissues confront the anoxemia injury.

脑缺血再灌注大鼠脑组织胰岛素受体基因和蛋白水平表达增加；半暗带区神经元、部分神经胶质细胞及血管内皮细胞等阳染细胞可能参与脑缺血后组织细胞的能量代谢、促新生细胞形成等病理生理调节过程；胰岛素受体在缺血区血管内皮细胞高表达提示其可能在缺血区血管的病理生理变化中起某种调节作用；胰岛素受体在不同细胞中表达增高可能是缺血脑组织对抗缺血缺氧损伤的自身保护机制之一。

Objective To study the effects of GDNF and HSV-GDNF on Bcl-2 expression of spinal cord motoneurons of rats after anoxia in vitro.

目的 观察缺氧时GDNF和HSV-GDNF对体外培养的大鼠脊髓运动神经元Bcl-2表达的影响。

Key words: anthropotomy and histoembryology; fornix transection; paraventricular nucleus; arginine vasopressin; hypothalamic-pituitary-adrenal axis; neuron

关键 词：人体解剖学与组织胚胎学；穹窿切断；室旁核；精氨酸加压素；丘脑-垂体-肾上腺轴；神经元

But we don't care about Battlegrounds.

但我们并不在乎沙场中的显露。

Ah! don't mention it, the butcher's shop is a horror.

啊！不用提了。提到肉，真是糟透了。