神经元

Between P14 and P21,CIAPIN1 immunoreaction in the brain,heart and liverbecame much lower. However,between P21 and P28,CIAPIN1 immunoreactionin the heart,brain,liver and skeletal muscle became much lower,while with thekidney development,CIAPIN1 immunoreaction in the kidney became higher. Invarious tissues from adult mouse,CIAPIN1 immunoreaction could be seen incardiac muscle cell,brain,hepatocyte,epithelium of renal tubule,skeletal muscle,lung tissue,gastric mucosa and gland,acinus lienalis.2. Distribution of CIAPIN1 in normal fetal and adult human tissuesTo reveal the possible physiological role of CIAPIN1,we examined theexpression and distribution of CIAPIN1 in fetal and adult human tissues usingimmunohistochemistry. We found that CIAPIN1 was ubiquitously distributed infetal and adult tissues,and was localized in both the cytoplasm and the nucleus.

然而，在3个月大的成年鼠中，CIAPIN1阳性反应物在心、脑、肝和肾小管中的表达强度要低于P28小鼠；但CIAPIN1阳性反应物在成年鼠骨骼肌中较P28小鼠高。2、CIAPIN1蛋白在人5个月胚胎及成人多器官组织内的表达在人5月胚胎多器官组织中，CIAPIN1阳性反应物见于心脏、胆囊单层柱状上皮和粘膜、结肠粘膜、小肠粘膜和绒毛、肝脏、直肠腺体、胃粘膜、肾上腺束状带、甲状腺滤泡、脾索、胸腺小叶间隔、皮肤真皮层和汗腺、睾丸白膜和间质、脑组织内神经元和神经胶质、肺小支气管和肺泡、骨骼肌、肾脏皮髓质和肾小管、子宫内膜、胰腺腺泡和胰岛、卵巢、输卵管粘膜等绝大多数组织细胞。

In 1997, Baulieu named these steroids as "neurosteroids". In general, through the genomic and nongenomic effects within the CNS, neurosteroids exert a broad spectrum of action on neuronal function and plasticity.

一般说来，通过基因水平和非基因水平两方面途径，神经甾体调节了神经元的功能及其可塑性，在中枢神经系统中起到复杂多样的作用。

The more active a region of the brain, the more active its neurons, and in turn, the more blood will travel to that region.

大脑中越是活跃的区域，它的神经元也就越活跃，为此，更多的血液会流经这一区域。

The primary noxious afferent neurotransmitters excitatory glutamate and substance P play a pivotal role in the development and maintenance of chronic inflammatory pain.

初级伤害性传入神经元的兴奋性递质谷氨酸和P物质对慢性炎性痛的形成和维持起到非常关键的作用。

A mathematical model of dynamic neural networks is built, which is shown to be equivalent to an affine nonlinear control system.

引 言 神经网络系统是在现代神经生物学和神经心理学的研究基础上，模仿人的大脑神经元的结构特征和功能特征而建立起来的一种非线性系统。

Results: T-588 administration successfully preented motoneuron loss and ameliorated the choline acetyltransferase immunoreactiity in the ipsilateral nucleus ambiguus after agal nere aulsion.

结果：T-588给药成功的阻止了运动神经元的丢失，并且改善了迷走神经撕脱伤后同侧疑核胆碱乙酰基转移酶免疫反应性。

The neuroprotectie effects of T-588 administration after agal nere aulsion and neurofunctional recoery after recurrent laryngeal nere crush were studied using motoneuron cell counting, ealuation of choline acetyltransferase immunoreactiity, the electrophysiologic examination, and the re-mobilization of the ocal fold.

本文采用运动神经元细胞记数、胆碱乙酰基转移酶免疫反应性、电生理学检查和声带再运动情况的方法来研究在迷走神经撕脱伤后T-588给药的神经保护作用和喉返神经挤压损伤后神经功能的恢复。

Results. Perfusion with ISM generated an agonal depolarization in all H and DH neurons recorded in current-clamp mode.

结果：所有腹侧和背侧神经元在电流箝记录中均出现濒死的去极化改变。

Results. Perfusion with ISM generated an agonal depolarization in all VH and DH neurons recorded in current-clamp mode.

结果：所有腹侧和背侧神经元在电流箝记录中均出现濒死的去极化改变。

Following ISM superfusion, an agonal inward current was produced at a holding potential of -70 m in all H and DH neurons tested in oltage-clamp mode.

在电位箝模式中所有神经元在域电位－70m时可见电流内流现象。

But we don't care about Battlegrounds.

但我们并不在乎沙场中的显露。

Ah! don't mention it, the butcher's shop is a horror.

啊！不用提了。提到肉，真是糟透了。