神经元

It was found that SO2 derivatives increase the amplitude of IA in CA3 neurons, and inhibit the inactivation of IA. After SO2 derivatives application, normal sensory physiology of hippocampal neurons was affected. This variation caused by SO2 could underlie the toxical modulation of sensory input to the central nervous system.

说明SO2代谢衍生物可增大大鼠海马CA3区神经元IA电流，延长A-电流的失活时间，从而影响海马神经元的膜生理感应，这可能是SO2影响神经细胞功能的机理之一。

The main results and conclusions are as follows: 1. Culrured neurons were damaged by simulated cerebral ischemia and reperfusion in vitro: Results:(1) The activity of the darnaged neurons was decreased, the survival rate was descent.(2) The transudation rate of LDH in cultured medium increased obviously.

体外模拟脑缺血再灌注造成了培养神经元的损伤：表现为：(1)受损神经元的活性降低，细胞存活率下降；(2)细胞培养液LDH的漏出率明显提高；(3)细胞死亡率显著上升；(4)NOS染色强阳性细胞数在缺血期和再灌注早期明显增多。

The FosB/△FosB positive staining was detected in nucleus accumbens septum, tuberculum olfactorium and dorsal caudate putamen, without little cFos positive staining. The results indicate the expression of cFos and FosB/△FosB display the differences in area and intensity, and the area intensively expressing FosB/△FosB is more nearly coincide with the area related to schizophrenia. So FosB/△FosB is a better mark to research the pathogenesis of schizophrenia.

同时大量研究证明，动物模型的行为及生理变化与中枢神经系统神经元的病理变化有关，因此，利用即早基因反应的灵敏性、功能多样性、检测方便性等，应用免疫组织化学的方法对模型小鼠脑内cFos和FosB/△FosB蛋白的表达进行检测，通过确定不同脑区cFos和FosB/△FosB蛋白的表达，来研究哪些脑区或神经元参与了反应，从而发现精神分裂症在脑区内所涉及的解剖结构，有助于对精神分裂症发病机制的认识。

Sections from frontal lobe, occipital lobe, striatum and hippocampus of normal subjects and sections from hippocampus of AD patients were used in hematoxylin eosin, Lox Fast blue, toluidine blue stains and ulex europaeus agglutinin immunostaining.

应用荆豆凝集素(ulex europaeus agglutinin， UEA)免疫组织化学及甲苯胺蓝等染色观察正常增龄病例额叶、枕叶、壳核、海马及AD患者海马的神经元、毛细血管形态改变，应用图像分析技术测算各部位神经元与毛细血管密度比值并分析其与年龄之间的相关性。

A dynamical model for unfully interconnected neural networks as associtive memory is proposed.

由此引出神经元集团的概念，并指出它和动态神经元的联系。

Among the 279 DMN neurons recorded, 173 (62.0%) responded to the stimulation of gastric vagal nerves.

在279个记录的DMN神经元中，有173（62％）个神经元对刺激胃迷走神经起反应。

Within 2 weeks of inflammation, the expression of TRPV1 in small and medium-sized TG neurons increased obviously. Also the TRPV1 stained terminals and fibers appeared more frequent and denser in both the vibrissal pad skin and throughout laminae I and the outer zone of laminae II of Vc.

正常大鼠TRPV1主要表达于TG的中小神经元，触须部皮肤以及三叉神经尾侧亚核含丰富的TRPV1阳性末梢；面部炎症痛后2周内，TG的中小神经元，触须部皮肤末梢以及Vc的Ⅰ和Ⅱ外层均可见明显的TRPV1表达增加。

Within 2 weeks of inflammation, the expression of TRPV1 in small and medium-sized TG neurons increased obviously. Also the TRPV 1 stained terminals and fibers appeared more frequent and denser in both the vibrissal pad skin and throughout laminae I and the outer zone of laminae Ⅱ of Vc.

正常大鼠TRPV1主要表达于TG的中小神经元，触须部皮肤以及三叉神经尾侧亚核含丰富的TRPV1阳性末梢；面部炎症痛后2周内，TG的中小神经元，触须部皮肤末梢以及Vc的Ⅰ和Ⅱ外层均可见明显的TRPV1表达增加。

The paired peptidergic buccal ganglion neurons RB 1 and LB 1 have dramatic modulatory effects on both the feeding motor program and the force of heart contraction (Wels- ford and Prior, 199 1). The B 1 neurons appear to contain the small cardioactive peptides.

配对肽能口神经节中的神经元RB1及LB1对摄食动力神经程序和的心脏收缩力都有很强的调节效果。B1神经元中有少量作用于心脏的缩氨酸出现。

We present the possible miRNA-mediated signaling pathways involved in the regulation of chemosensation and thermosensation by controlling the corresponding sensory neuron and interneuron functions.

我们勾勒出了可能存在的由miRNA介导的通过控制相应感觉神经元与中间神经元功能从而调控化学趋向性与温度趋向性的信号通路。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。