神经元

The characteristics of CaBP-LI positive rubrospinal neurons in the red nucleus showed that these neurons was not homogeneous. Rubrospinal neurons had their subpopulation in term of CaBP like immunoreactivity (from strongly posotive to negative) and special distribution features.

大鼠红核-脊髓投射神经元的CaBP-LI阳性和CaBP-LI阴性两种亚型及其分布特点说明，这些神经元的性质并非均一，并可能与红核-脊髓投射神经元对钙超载具有不同的耐受性有关。

Rubrospinal neurons in the rostral portion still showed CaBP-LI negative. Conclusion The characteristics of CaBP-LI positive rubrospinal neurons in the red nucleus showed that these neurons was not homogeneous. Rubrospinal neurons had their subpopulation in term of CaBP like immunoreactivity (from strongly posotive to negative) and special distribution features.

大鼠红核-脊髓投射神经元的CaBP-LI阳性和CaBP-LI阴性两种亚型及其分布特点说明，这些神经元的性质并非均一，并可能与红核-脊髓投射神经元对钙超载具有不同的耐受性有关。

The characteristics of CaBP-LI positive rubrospinal neurons in the red nucleus showed that these neurons was not homogeneous. Rubrospinal neurons had their subpopulation in term of CaBP like immunoreactivity (from strongly posotive to negative) and special distribution features.Key words: calbindin-D28k; immunocytochemistry; red nucleus; rubrospinal tract; rat

大鼠红核-脊髓投射成年人神经元的CaBP-LI阳性和CaBP-LI阴性两种亚型及其分布特点说明，这些成年人神经元的性质并非均一，并可能与红核-脊髓投射成年人神经元对钙超载具有不同的耐受性有关。

Results The rubrospinal neurons were retrogradely identified by cholera toxin subuint B coulpled with colloidal gold (CB-Au,prepared by our laboratory).No CaBP-LI positive neurons identified by CB-Au was present in the rostral level of the red nucleus.

结果 红核-脊髓投射成年人神经元表现为两种亚型：一种为CaBP-LI阴性成年人神经元；另一种为CaBP-LI阳性成年人神经元。

Methods Employing cholera toxin B-subunit combined horseradish peroxidase retrograde tracing technique and cholera toxin B-subunit combined colloid goldretrograde tracing with calcitonin gene-related peptideimmunohistochemical double-labeled staining technique,the innervation of musculus sacrococcygeus ventralis medialisin rats was studied.

大鼠骶尾腹内侧肌又称荐尾腹侧中肌(musculus sacrococcygeus ventralis medialis)或屈尾短肌，是位于盆腔后壁、骶椎前方的一对梭形肌肉，属尾肌群，因其功能似乎仅为屈尾，故至今尚未引起人们的注意，而与该肌位置关系较密切的会阴肌因在某些运动神经元疾患时仍可保持其正常生理功能而一直受到神经工作者的关注，尤其是近年来有诸多文献报道了会阴肌的神经支配、相关神经元的细胞构筑、神经递质和神经调质的分布以及纤维联系特点［1～10］，藉以探讨运动神经元疾病的神经病理学。

Correction of scoliotic cures with the use of a computerassisted tool allowing 3-dimensional isualization of the spinal cures and the external shape of the trunk.

在病理情况下，在TNF存在的情况下，NGF敏感神经元，包括支配椎间盘的神经元，比GDNF敏感神经元具有更大的伸展其轴突的功能。

The present results showed that both cytoplasm and membrane of neuronal soma and dendrite expressed NKCC1, while KCC2 only expressed in membrane of soma and dendrite. The results also indicated that the dendrites rather than the somata of neurons expressed more NKCC1 in adult rat neocortex, while the level of KCC2 expression in the dendrite membrane was similar to that in the membrane of somata.

结果显示：成年大鼠皮质神经元的胞浆和细胞膜均有NKCC1的表达，而KCC2主要表达在神经元胞体和树突膜上，其中NKCC1在神经元树突上的表达水平比胞体高，而KCC2的表达水平在树突和胞体膜上没有明显差异。

These results demonstrated an important convergence of dopaminergic and 5-hydroxytryptaminergic activities in the same DRN neurons, dopaminergic and 5-hydroxytryptaminergic activities did not tonically inhibit the DRN neurons.

DA、5-HT抑制大鼠DRN神经元的自发放电活动，并在同一DRN神经元有重要会聚作用，DA、5-HT对DRN神经元无紧张性抑制作用。

Electron-microscopic observation: In stroke-prone spontaneous hypertension group, electron density was found increased in necrotic neurons; moreover, some nuclear membrane lost double-layer structure with ridges broken, even reduced or disappeared, displaying vacuolated changes. In losartan 30 mg/ group and losartan 10 mg/ group, most of neurons displayed basically normal morphology, with neuron chromatin evenly distributed and nuclear envelops regular, but there were still some neurons that had dense chromatin, with ridges broken and reduced.

3组大鼠脑组织电镜观察：易感型自发性高血压组大鼠坏死神经元电子密度增高，有的核膜失去双层结构，嵴断裂、减少甚至消失，呈空泡样改变；氯沙坦30mg/组和氯沙坦10 mg/组大多数神经元染色质分布均匀，核膜规整，细胞形态基本正常；少数神经元核染色质较致密，部分嵴断裂、减少。

The dynamic changes of the the neuron apoptosis and correlative genes in hippocampus of ACET-groups and twice-ACET-groups suggested that ACET can slower the apoptosis by inhibiting p~(53)and prolong the ascending period of p~(53) in 4 days after the seizure,this effect would weaken after 4 days.ACET may inhibit the descending of Bax,prolong its descending period and then promote its expression,in order to weaken the inhibition of the apoptosis during 4 to 10 days after seizure and then promote the apoptosis during 10 to 14 days after.This effect of ACET can be enhanced after treating again within 7 to 14 days later.

埋线组和二次埋线组大鼠海马神经元凋亡及相关基因表达的动态变化结果提示：埋线治疗在癫痫发作后的早期(4d内)可通过抑制促凋亡基因p~(53)蛋白表达、延缓其达峰值的时程，从而减缓神经元凋亡的发生，此作用在4d后减弱；埋线治疗抑制大鼠海马神经元凋亡的作用在癫痫发作后的中期(4d～10d)减弱，甚至在远期(10d～14d)出现促进凋亡的作用，可能是通过抑制Bax蛋白表达的下降、延长其降至低峰值的时程，促进Bax蛋白表达的回升的结果；在中远期(7d～14d)内予二次埋线治疗对该效应有续效叠加的作用。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。