磷酸钠

Water,c12-20 acid peg-8 ester,octyldodecanol,propylene glycol,coco-caprylate/caprate,cetearyl isononanoate,prunus amygdalus dulcisoil,biosaccharide gum-1,phenoxyethanol,ethylhexyl methoxycinnamate,potassium cetyl phosphate,simmondsia chinensisseed oil,carbomer,glyceryl polymethacrylate,malva sylvestrisextract,glyceryl stearate,cetearyl alcohol,cetyl palmitate,cocoglycerides,c12-13 alkyl lactate,methylparaben,sorbic acid,disodium edta,sodium hydroxide,tocopheryl acetate,benzophenone-3,iris germanica root extract,retinyl palmitate,butylparaben,peg-8,propylparaben,ethylparaben,sodium hyaluronate,chlorhexidine digluconate,tocopherol,ascorbyl palmitate,citric acid,ascorbic acid

水， c12-20 acid peg-8 ester，辛基十二烷醇，丙二醇，coco-caprylate/caprate，鲸蜡硬脂醇异壬酸酯，甜杏仁油，多醣物质，苯氧乙醇，4-甲氧基肉桂酸-2-乙基己基酯，十六烷基磷酸钾，西蒙得木籽油，卡波姆，甘油聚甲基丙烯酸酯，锦葵萃取，甘油硬脂酸，棕榈醇，十六烷基棕榈酸酯，椰油脂酸甘油酯类，c12-13烷醇乳酸酯，对羟基苯甲酸甲酯，山梨酸，乙二胺四乙酸二钠，氢氧化钠，维生素E醋酸酯，二苯甲酮-3，鸢尾花萃取，维他命A酯，对羟基苯甲酸丁酯，聚乙二醇-8，羟苯丙酯，羟苯乙酯，透明质酸钠，洗必泰葡萄糖酸盐，维生素E ，维生素C棕榈酸酯，柠檬酸，抗坏血酸

Water,c12-20 acid peg-8 ester,caprylic/capric triglyceride,proplylene glycol,ppg-15 stearyl ether,chondrus crispus,iris germanica(orris extract/cells),centaurea cyanus,macadamia ternifolia,buxus chinensis,triethanolamine,potassium cetyl phosphate,butyrospermum parkii,c12-13 alkyl lactate,carbomer,phenoxyethanol,methylparaben,imidazolidinyl urea,glyceryl hydroxystearin,tocopheryl acetate.retinyl palmitate,sodium hyaluronate,biosaccharide gum-1,propylparaben,disodium edta,ethylparaben,pentadecalactone,hydroxystearic acid,tetradecyloctadecanoic acid,tetradecyleicosanoic acid,hexadecyleicosanoic acid,butylparaben,isobutylparaben,chlorhexidine digluconate,tocopherol,lecithin,ascorbyl palmitate,glyceryl stearate,glyceryl oleate,BHT,citric acid.

水，C12-20 酸 PEG-8酯，辛酸/癸酸三酸甘油酯，丙二醇，PPG-15 硬脂基醚，卡拉胶萃取，德国鸢尾花提取物/细胞，矢车菊提取物，澳洲坚果油，荷荷巴油，三乙醇胺，十六烷基磷酸钾，牛油果，c12-13烷醇乳酸酯，卡波姆，苯氧乙醇，羟苯甲酯，尿素醛，甘油羟基硬脂酸酯，醋酸盐维他命E，维他命A酯，透明质酸钠，多醣物质，羟苯丙酯，乙酸乙二胺二钠，羟苯乙酯，环十五内酯，羟基硬脂酸，十四烷基十八烷酸，十四烷基二十烷酸，十六烷基二十烷酸，羟苯丁酯，对羟基苯甲酸异丁酯，洗必泰葡萄糖酸盐，维生素E，卵磷脂，抗坏血酸棕榈酸酯，甘油硬脂酸，甘油油酸酯，BHT，柠檬酸。水：几乎所有护肤品成分第一位都是水。辛酸/癸酸三酸甘油酯：润滑剂、扩散剂，棕榈油/椰子油衍生物

When proceeding the research of drug identification by ultraviolet spectrophotometry, this paper adopts the approaches below: to identify Metronidazole Buccal Tablets and Potassium Sodium Dehydroandroan Drographolide Succinate by ulviolet maximum absorbent wavelength; to identify Metronidazole Disodium Phosphate for Injection by ulviolet maximum absorbent wavelength and minimum absorbent wavelength; to identify Ceftriaxone Sodium for Injection and Clarithromycin Tablets by retention time of high performance liquid chromatography- ultraviolet spectrophotometry.

通过测定紫外最大吸收波长λ_(max鉴别甲硝唑口颊片和炎琥宁、测定紫外最大吸收波长λ_(max和最小吸收波长λ_(min鉴别注射用甲硝唑磷酸二钠，结合高效液相色谱(high performance liquid chromatography-ultraviolet detector，HPLC-UV)中保留时间的测定鉴别注射用头孢曲松钠和克拉霉素片，本文建立了药物鉴别的紫外分光光度法。

The research indicates that:(1) detecting λmax (277nm) can identify Metronidazole Buccal Tablets prepared by Chongqing Medical College;(2) detecting λmax (321nm) and λmin (265nm) can identify Metronidazole Disodium Phosphate for Injection prepared by Chongqing Medical College;(3) detecting tr (6.7min) in HPLC-UV spectrum can identify Ceftriaxone Sodium for Injection prepared by Chongqing Keru Pharmacy Co.;(4) detecting tR (10.2min) 6.7min in HPLC-UV spectrum can identify Clarithromycin Tablets prepared by Harebin Yida Pharmacy Co.;(5) detecting λmax (254nm) can identify Potassium Sodium Dehydroandroan Drographolide Succinate prepared by Hong Kong Yaohen Co..

结果表明：①测定紫外最大吸收波长λ_(max277nm可鉴别重庆医科大学医药研究所制备的甲硝唑口颊片；②测定紫外最大吸收波长λ_(max321nm和最小吸收波长λ_(min265nm可鉴别重庆医科大学医药研究所制备的注射用甲硝唑磷酸二钠；③测定HPLC-UV中的保留时间(t_R6.7min)可鉴别重庆科瑞制药股份有限公司制备的注射用头孢曲松钠；④测定高效液相色谱的保留时间(t_R10.2min)可鉴别哈尔滨伊达制药厂提供的克拉霉素片；⑤测定紫外最大吸收波长(又max254nm)可鉴别香港耀恒公司提供的炎琉宁。

It is a method most in use to analyze drugs.This paper chooses the antibiotics intending to be registered as research topics to study their identification, tests and assay by ultraviolet spectrophotometry. These antibiotics includes Metronidazole Buccal Tablets, Metronidazole Disodium Phosphate for Injection, Ceftriaxone Sodium for Injection, Clarithromycin Tablets, Potassium Sodium Dehydroandroan Drographolide Succinate.

本文选择拟注册的抗生素类药物甲硝唑口颊片、注射用甲硝唑磷酸二钠(Metronidazole Disodium Phosphate for Injection)、注射用头孢曲松钠(Ceftriaxone Sodium for Injection)、克拉霉素片和炎琥宁(Potassium Sodium Dehydroandroan Drographolide Succinate)为研究对象，应用紫外分光光度法进行鉴别、检查和含量测定研究。

Based on laboratory research,the feasible analysis on process technology of producing sodium dichromate from used chromic residue phosphate produced by reused sodium hydroger sulfate in chromate producing system is made,together with its potential market analysis.

铬盐生产厂含铬硫酸氢钠返回系统中再利用所产生的废渣———磷酸铬渣，通过实验室研究，可生产重铬酸钠。

Then the contents of brain tissue water, malonyl diadehyde and the activities of Na+-K+-adenosine triphosphatase (Na+-K+-ATPase), Ca2+-Mg2+-adenosine triphosphatase (Ca2+-Mg2+-ATPase), CuZn-superoxide dismutase and mitochondrial Mn-superoxide dismutase were measured.

测定脑组织含水量、丙二醛含量、钠钾三磷酸腺苷酶(Na~+-K~+-ATPase)、钙镁三磷酸腺苷酶Ca~(++-Mg~-ATPase、铜锌超氧化物歧化酶以及线粒体锰超氧化物歧化酶的活力。

Artificial acidification can be achieved by the addition of lactic, acetic , tartaric or citric acid to rye dough or by adding acidic forms of sodium and calcium salts of ortho- and\0r pyrophosphoric acids.

通过向黑麦面团中添加乳酸、醋酸、酒石酸或柠檬酸或添加酸性正磷酸和\或焦磷酸的钠盐和钙盐就可以实现人工酸化面团的目的。

MethodWith natural polymer KGMas main materials,trisodium trimetaphosphate was taken as the crosslinking agent to prepare a kind of biodegradable phosphorylated KGM hydrogel.

方法］以天然高分子KGM 为主要原料，采用三偏磷酸三钠作为交联剂来制备1种可降解磷酸酯化KGM水凝胶。

The effects and mechanism of GABAergic neurons, NOergic neurons, opioid peptide and cyclic adenosine monophosphate in the nucleus reticularis thalami on sleep-wakefulness cycle of rats and the effects and mechanism of the 5-HTergic nerve fibers project from the nucleus raphes dorsalis to RT on sleep-wakefulness cycle of rats were investigated with the methods of brain stereotaxic, nucleus spile, microinjection and polysomngraphy.1. The effects of GABAergic neurons in RT on sleep-wakefulness cycle of rats1.1 Microinjection of 3-mercaptopropionic acid (3-MP, a kind of glutamate decarboxylase inhibitor) into RT. On the day of microinjection, sleep only decreased a litter. On the second day, sleep marked decreased and wakefulness marked increased. On the third and fourth day, sleep and wakefulness stages resumed to normal.1.2 Microinjection of gamma-amino butyric acid (GABA 1.0μg) into RT enhanced sleep and reduced wakefulness compared with control; while microinjection of L-glutamate (L-Glu, 0.2μg) decreased sleep and increased wakefulness; microinjection of bicuculline (BIC, 1.0μg), a GABAA receptor antagonist, enhanced wakefulness and reduced sleep; microinjection of baclofen (BAC, 1.0μg), GABAB receptor agonist, had the same effects as GABA.2. The effects of NOergic neurons in RT on sleep-wakefulness cycle of rats2.1 Microinjection of L-arginine (L-Arg, 0.5μg) into RT decreased sleep compared with control, but there were on statistaical difference between L-Arg group and control; while microinjection of sodium nitroprusside (SNP, 0.2μg), a NO donor into RT, sleep marked decreased and wakefulness marked increased. Microinjection of nitric oxide synthase inhibitor, N-nitro-L-arginine (L-NNA, 2.0μg) into RT enhanced sleep and reduced wakefulness.2.2 After simultaneous microinjection of L-NNA (2.0μg) and SNP (0.2μg) into RT, SNP abolished the sleep-promoting effect of L-NNA compared with L-NNA group; after simultaneous microinjection of L-NNA (2.0μg) and L-Arg(0.5μg) into RT, we found that L-NNA could not blocked the wakefulness-promoting effect of L-Arg.3. The effects of opioid peptide in RT on sleep-wakefulness cycle of rats3.1 Microinjection of morphine sulfate (MOR, 1.0μg) into RT increased wakefulness and decreased sleep compared with control; while microinjection of naloxone hydrochloride (NAL, 1.0μg), the antagonist of opiate receptors, into RT, enhanced sleep and reduced wakefulness.3.2 After simultaneous microinjection of MOR (1.0μg) and NAL (1.0μg) into RT, the wakefulness-promoting effect of MOR and the sleep-promoting effect of NAL were not observed compared with control.4. The effects of cAMP in RT on sleep-wakefulness cycle of rats Microinjection of cAMP (1.0μg) into RT increased sleep and decreased wakefulness compared with control; microinjection of methylene blue (MB,1.0μg) into RT enhanced sleep and reduced wakefulness compared with control.5. The effects of the 5-HTergic nerve fibers project from DRN to RT on sleep-wakefulness cycle of rats5.1 When L-Glu (0.2μg) was microinjected into DRN and normal sodium (NS,1.0μg) was microinjected into bilateral RT. We found that sleep was decreased and wakefulness was increased compared with control; when L-Glu (0.2μg) was microinjected into DRN and methysergide (MS,1.0μg), a non-selective 5-HT antagonist, was microinjected into bilateral RT, We found that sleep was enhanced and wakefulness was reduced compared with L-Glu group.5.2 When p-chlorophenylalanine (PCPA, 10μg) was microinjected into DRN and NS (1.0μg) was microinjected into bilateral RT, We found that sleep was increased and wakefulness was decreased compared with control; microinjection of 5-hydroxytryptaphan (5-HTP, 1.0μg), which can convert to 5-HT by the enzyme tryptophane hydroxylase and enhance 5-HT into bilateral RT, could block the effect of microinjection of PCPA into DRN on sleep-wakefulness cycle.

本研究采用脑立体定位、核团插管、微量注射、多导睡眠描记等方法，研究丘脑网状核(nucleus reticularis thalami，RT)中γ-氨基丁酸(gamma-amino butyric acid ，GABA)能神经元、一氧化氮(nitrogen monoxidum，NO)能神经元、阿片肽类神经递质、环一磷酸腺苷(cyclic adenosine monophosphate，cAMP)及中缝背核(nucleus raphes dorsalis，DRN)至RT的5-羟色胺(5-hydroxytryptamine，5-HT)能神经纤维投射对大鼠睡眠-觉醒周期的影响及其作用机制。1 RT内GABA能神经元对大鼠睡眠-觉醒周期的影响1.1大鼠RT内微量注射GABA合成关键酶抑制剂3-巯基丙酸(3-MP，5μg)，注射当天睡眠时间略有减少，第二日睡眠时间显著减少，觉醒时间明显增多，第三、四日睡眠和觉醒时间逐渐恢复至正常。1.2大鼠RT内微量注射GABA受体激动剂GABA( 1.0μg)后，与生理盐水组比较，睡眠时间增加，觉醒时间减少；而RT内微量注射L-谷氨酸(glutamic acid， L-Glu， 0.2μg)后，睡眠时间减少，觉醒时间增加；RT内微量注射GABAA受体阻断剂荷包牡丹碱(bicuculline，BIC，1.0μg)后，睡眠时间减少，觉醒时间增加；RT内微量注射GABAB受体激动剂氯苯氨丁酸(baclofen，BAC，1.0μg)后，产生了与GABA相似的促睡眠效果。2 RT内NO能神经元对大鼠睡眠-觉醒周期的影响2.1大鼠RT内微量注射NO的前体L-精氨酸(L-Arg，0.5μg)后，与生理盐水组对比，睡眠时间略有减少，但无显著性意义；而RT内微量注射NO的供体硝普钠(Sodium Nitroprusside，SNP，0.2μg)后可明显增加觉醒时间，缩短睡眠时间；微量注射一氧化氮合酶抑制剂L-硝基精氨酸(L-arginine，L-NNA，2.0μg)后，引起睡眠时间增多，觉醒时间减少。2.2大鼠RT内同时微量注射L-NNA(2.0μg)和SNP(0.2μg)后与L-NNA组比较发现SNP逆转了L-NNA的促睡眠作用；RT内同时微量注射L-NNA(2.0μg)和L-Arg(0.5μg)后，与L-NNA(2.0μg)组比较发现L-Arg可以增加觉醒而缩短睡眠，其促觉醒作用未能被NOS的抑制剂L-NNA所逆转。3 RT内阿片肽对大鼠睡眠-觉醒周期的影响3.1大鼠RT内微量注射硫酸吗啡(morphine sulfate，MOR，1.0μg)后与生理盐水组对比，睡眠时间减少而觉醒时间增加； RT内微量注射阿片肽受体拮抗剂盐酸纳洛酮(naloxone hydrochloride，NAL，1.0μg)后与生理盐水组比较，睡眠时间增加而觉醒时间减少。3.2大鼠RT内同时微量注射MOR(1.0μg)和NAL(1.0μg)后，与生理盐水组对比，原有的MOR促觉醒效果和NAL的促睡眠效果都没有表现。4 RT内环一磷酸腺苷信使对大鼠睡眠-觉醒周期的影响大鼠RT内微量注射cAMP(1.0μg)后与NS(1.0μg)组比较，睡眠时间增多而觉醒时间减少；RT内微量注射亚甲蓝(methylene blue，MB，1.0μg)后，与NS组比较，睡眠时间增多而觉醒时间减少。5中缝背核投射到丘脑网状核的5-羟色胺能神经纤维对大鼠睡眠-觉醒周期的影响5.1大鼠DRN内微量注射L-Glu(0.2μg)，同时在双侧RT内微量注射NS (1.0μg)后，与对照组(DRN和双侧RT注射NS， 0.2μg)比较，睡眠时间减少，觉醒时间增多；大鼠DRN内微量注射L-Glu(0.2μg)，同时在双侧RT内微量注射二甲基麦角新碱(methysergide， MS， 1.0μg )后，与对照组(DRN注射L-Glu 0.2μg，双侧RT注射NS 1.0μg)比较，睡眠时间增多，觉醒时间减少。5.2大鼠DRN内微量注射对氯苯丙氨酸(p-chlorophenylalanine，PCPA，10μg)，同时在双侧RT内微量注射NS (1.0μg)后，与对照组(DRN和双侧RT注射NS， 1.0μg)比较，睡眠时间增多，觉醒时间减少；大鼠DRN内微量注射PCPA(10μg)，产生睡眠增多效应后，在双侧RT内微量注射5-羟色胺酸(5-hydroxytryptaphan ， 5-HTP， 1.0μg )后，与对照组(DRN注射PCPA 10μg，双侧RT注射NS 1.0μg)比较，睡眠时间减少，觉醒时间增多。

On the other hand, the more important thing is because the urban housing is a kind of heterogeneity products.

另一方面，更重要的是由于城市住房是一种异质性产品。

Climate histogram is the fall that collects place measure calm value, cent serves as cross axle for a few equal interval, the area that the frequency that the value appears according to place is accumulated and becomes will be determined inside each interval, discharge the graph that rise with post, also be called histogram.

气候直方图是将所收集的降水量测定值，分为几个相等的区间作为横轴，并将各区间内所测定值依所出现的次数累积而成的面积，用柱子排起来的图形，也叫做柱状图。