磷脂

But when the cholesterol was substituted by its oxide result, the membrane life time extended to nearly 60 hrs ;besides the diameter of the hole on which the membrane exists 、temperature、PH value cause great effects to the life time of the membrane, and the diameter was the most important element. When the diameter larger than 3mm it is almost no way to form a membrane on the hole under experiment condition. Between 1mm and 3mm it has a better result by change the prescription of the membrane. when it is 0.5mm all the membrane can last more than 40 hrs ,and the prescription like Och : phosphatidylcholine / cephalin＝4:1; 3:1; 2:1; 1:1; 2:3 need the request of the experiment basically, when cooperate with multi hole structure it is very to detect the concentration and found the model.

研究结果说明，成膜在普通胆固醇/磷脂配方下不能达到满意的结果，无论如何改变外界条件成膜始终超不过7小时，而将胆固醇换成氧化胆固醇再和磷脂按照一定配比的时候，成膜时间则大大延长，可达60h左右；另外除成膜配方，成膜孔径、温度、PH值等因素对成膜有着很重要的影响，成膜孔径对膜的影响最为关键，在3mm以上的孔径时成膜比较困难，在1－3mm适当提高成膜液浓度可以改善，孔径为0.5mm左右成膜比较稳定基本都能达到40h以上，合适的几个配方如卵/脑磷脂：氧化胆固醇＝4：1；3：1；2：1；1：1；2：3基本满足实验要求，配合一板多孔结构，可以保证单位时间的离子传输量的检测和计算。

It is postulated that IP induces the release of endogenous protective molecules that interact with cognate receptors to activate PLC through mediation of the chincough virus sensitive proteins. PLC hydrolyzes membrane phospholipid and produces diacylglyccerol,that,by working together with calcium hydronium,translocates PKC from cytoplasm to cell membrane and activates PKC.

推测缺血预处理细胞保护作用机制是：缺血预处理引起内源性保护物质释放，作用于细胞膜上的相应受体，通过百日咳病毒敏感性G蛋白的介导而激活磷脂酶，磷脂酶水解膜磷脂，产生二酰基甘油(diacylglyccerol，DAG)，DAG和Ca2+协同作用于PKC，使其从胞浆转位到细胞膜并且被活化，PKC在膜上使底物蛋白磷酸化，引发一系列相关信号转导通路的激活，调控保护性基因的表达，从而提高细胞对缺血再灌注损伤的耐受性。

The major phopholipids of egg yolk are phasphatidylcboline (73%) and phosphatidylethanolamine (15%).

蛋黄中主要的磷脂是卵磷脂(73%)和磷脂酰乙醇胺(15%)。

Both Phosphatidyl Choline and Phosphatidyl Ethanolamine are important sorts of phospholipids, being widely used in the filed of food, pharmacy, cosmetic and so on.

卵磷脂和脑磷脂是其中两种重要的磷脂，被广泛应用于食品、医药、化妆品等行业。

Soybean powder phosphatide was hydrolyzed by phospholipase A_1,and the optimum conditions were investigated.

利用磷脂酶 A1对大豆粉末磷脂进行水解，通过实验确定了大豆粉末磷脂酶解的最佳反应条件为：反应时间9 h，反应温度50℃，起始pH 5.1。

It was observed that sphingomyelin plays a negative regulatory role on the activity of 〓, resulting in a strong inhibition on 〓-induced cell death.

我们观察到，鞘磷脂对分泌型磷脂酶〓的活性起着负调控作用，它能有效的抑制分泌型磷脂酶〓对细胞的杀伤作用。

LB technique and atomic force microscopy were used to study the effect of 1,2-dioleoy-sn-glycero-3-phosphocholine, 1,2-dioleoy-sn-glycero-3-phosphoethanolamine and ceramide on the structure of sphingomyelin/Cholesterol LB bilayers.

用LB技术和原子力显微镜研究了1，2-二油酸甘油－3-磷脂酰胆碱、1，2-二油酸甘油－3-磷脂酰乙醇胺和神经酰胺对鞘磷脂/胆固醇结构的影响。

Itwas observed that sphingomyelin plays anegative regulatory role on the activity of sPLA 2, resulting in a strong inhibition on sPLA 2-induced cell death.

我们观察到，鞘磷脂对分泌型磷脂酶A 的活性起着负 2 调控作用，它能有效的抑制分泌型磷脂酶A 2对细胞的杀伤作用。

Aim To study the effect of phosphalipids on the cholesterol efflux from rat macrophages mediated by HDL3. Methods Measure the cholesterol content, cellular phospholipid content, and the balance between free cholesterol and cholesteryl ester when phosphatidylcholine or sphingomyelin was added to the mediun.

为了研究卵磷脂和鞘磷脂对高密度脂蛋白3介导大鼠腹腔巨噬细胞内胆固醇流出的影响，测定了细胞内胆固醇含量、磷脂含量及游离胆固醇－胆固醇酯平衡的变化。

ABSTRACT Aim To study the effect of phosphalipids on the cholesterol efflux from rat macrophages mediated by HDL3. Methods Measure the cholesterol content, cellular phospholipid content, and the balance between free cholesterol and cholesteryl ester when phosphatidylcholine or sphingomyelin was added to the mediun.

摘 要］为了研究卵磷脂和鞘磷脂对高密度脂蛋白3介导大鼠腹腔巨噬细胞内胆固醇流出的影响，测定了细胞内胆固醇含量、磷脂含量及游离胆固醇－胆固醇酯平衡的变化。

But we don't care about Battlegrounds.

但我们并不在乎沙场中的显露。

Ah! don't mention it, the butcher's shop is a horror.

啊！不用提了。提到肉，真是糟透了。