碳酸血症

For the aged critical illness patients with acute heart failure,MV could relieve anoxemia, hypercapnia and respiratory insufficiency effectively, and obviously increase success rate of emergency care.

机械通气能及时有效地缓解老年急性左心衰患者的低氧血症、高碳酸血症和呼吸功能障碍，明显提高老年急性左心衰患者的治疗抢救成功率。

Consider to discover, OSAHS patient appears because of relapsing in nightly Morpheus disease of blood of low oxygen, tall carbonic acid, consensual as disorder as parasympathetic nerve function, already made coronary heart disease (the cardiovascular disease such as CHD) sparks importantly element, can cause all sorts of rhythm of the heart wrong, aggravating cardiac muscle is short of blood, produce sudden death of heart source sex even. 1 O SAH S and hypertensive disease consider to discover, OSAHS patient's nightly small arousal can bring about sympathetic stimulant increase, adrenalin can nerve medium if adrenalin, go adrenalin secretes armour grow in quantity, heighten of material of phenolic amine of catechu of nightly make water.

探究发现，OSAHS患者夜间睡眠中因反复出现低氧、高碳酸血症、交感和副交感神经功能紊乱，已成为冠心病等心血管疾病重要的触发因素，可诱发各种心律失常、加重心肌缺血，甚至发生心源性猝死。1 O SAH S和高血压病探究发现，OSAHS患者夜间的微觉醒可导致交感神经兴奋性增强，肾上腺素能神经介质如肾上腺素、去甲肾上腺素分泌增多，夜间尿儿茶酚胺类物质增高。

The blood pressure of.1 of pathology physiology mechanism that SAS causes CVD breathes time-out to be able to bring about airframe in elevatory Morpheus anoxic, sympathetic is excessive and excited, element of serous catechu phenolic amine, kidney and hemal endodermis element are elevatory, bring about hemal easy of hemal convulsion; to shrink the function is disorder, can make blood-vessel flowing flesh happening reframes and fleshy, systemic blood-vessel obstruction adds element of the kidney when; is chronic and anoxic, system of hemal and nervous element is activationed, bring about blood pressure to lift. Obstruction of way of energy of life of patient of OSAS of disease of blood of 1.2 low oxygen increases, cause air current to interrupt, breath pauses, at the same time airframe gets used to low oxygen environment gradually, breathing centre drops to low oxygen and sensitivity of disease of blood of tall carbonic acid, breath suspends a frequency increasing, farther aggravating airframe is anoxic. 1.3 heads are self-adjusting the function drops normal person changes quickly in systematic circulation blood pressure when, the head can be passed adjust independently functional generation protects effect, make change of cerebral blood flow not big.

SAS诱发CVD的病理生理机制。1血压升高睡眠中呼吸暂停可导致机体缺氧，交感神经过度兴奋，血浆儿茶酚胺、肾素以及血管内皮素升高，导致血管痉挛；血管舒缩功能紊乱，可使血管平滑肌发生重构和肥厚，全身血管阻力增加；慢性缺氧时肾素—血管紧张素系统被激活，导致血压升高。1.2低氧血症OSAS患者气道阻力增加，造成气流中断、呼吸暂停，同时机体逐渐适应低氧环境，呼吸中枢对低氧和高碳酸血症敏感性下降，呼吸暂停次数增加，进一步加重机体缺氧。1.3脑自动调节功能下降正常人在体循环血压快速变化时，脑可通过自主调节功能产生保护效应，使脑血流量变化不大。

Because chronic obstructive inflammation of airway severely influences the ventilation and exchange function of COPD patients, The motor ability of patients is limited .Despnoea and fatigue limit patients to sedentary,because of this, skeletal muscle began to analosis,oxygen uptaking capacity of skeletal muscle descends,and concentration of chondriosome degrades.Function limitation of skeletal muscle causes oxygen needs increase,so vicious cycle emerges,and further to restrict patient's respiratory and motor function.

由于气道的慢性炎症性阻塞，严重影响了慢阻肺患者的通气及换气功能，使得患者的运动能力明显受限，因喘息和疲劳而限制了患者的运动能力，使患者出现久坐，由此出现骨骼肌的萎缩，骨骼肌摄氧能力下降，线粒体氧浓度降低，骨骼肌的功能受限又会引起通气量不足，进而引起低氧血症和高碳酸血症，使患者的摄氧需求增加，由此出现恶性循环，进一步制约了患者的呼吸和运动功能。2001年正式提出肺康复作为中重度COPD患者稳定期非药物治疗的首选管理方案。

Index of circulation in EVH group can maintain stable,but should guard against harmful effects on supply and demand equilibrium of oxygen by hypercapnia.

EVH组循环指标能够维持稳定，应防范高碳酸血症对氧供需平衡的不良影响。

Chronic obstructive pulmonary disease variously accompanied with hypersecretion and airway obstruction in acute period.

慢性阻塞性肺疾病急性期常发生痰多及气道阻塞，并因低氧血症、高碳酸血症和酸碱失衡而危及患者生命。

Hypercapnia, along with hyperventilation, may be used clinically to decrease emergence time from inhaled anesthesia.

在临高碳酸血症与过度通气一起可以被用於缩短吸入麻醉的苏醒时间。

Objective To evaluate the effects of tracheal gas insufflation on elimination of CO2 and improvement of ventilation efficiency.

目的 探讨气管内吹气对急性高碳酸血症家兔二氧化碳清除的效果及其对通气效率的改善作用。

Methods:Normal frequency ventilation of high frequency jet respirometer was used to control breath continuously for 31 cases without autonomous breath,the ventilation frequency:16～20 times/min with continuous ventilation time 12～72 hours.

结果：通气1h后PaO2（15.92±2.50）kPa，PaCO2（5.90±0.76）kPa；通气24h后PaO2（14.76±3.21）kPa，PaCO2（6.11±0.84）kPa。在Ⅱ型呼吸衰竭，常频与高频通气交替应用比单用高频通气辅助呼吸，其提高血氧分压的疗效优越（P＜0.001），且可防止高碳酸血症。

BiPAP aerates applicable AECOPD is amalgamative Ⅱ breathe out decline patient, disease of the disease of low oxygen blood that can improve a patient apparently, blood of tall carbonic acid and PH are worth and method of tradition of remedial effect excel, be worth clinical promotion application.

结论BiPAP通气适用AECOPD合并Ⅱ型呼衰患者，可明显改善患者的低氧血症、高碳酸血症及PH值且治疗效果优于传统方法，值得临床推广应用。

Breath, muscle contraction of the buttocks; arch body, as far as possible to hold his head, right leg straight towards the ceiling (peg-leg knee in order to avoid muscle tension).

呼气，收缩臀部肌肉；拱起身体，尽量抬起头来，右腿伸直朝向天花板（膝微屈，以避免肌肉紧张）。

The cost of moving grain food products was unchanged from May, but year over year are up 8%.

粮食产品的运输费用与5月份相比没有变化，但却比去年同期高8％。