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硬化的细胞

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Osteoblast and osteoclast activity in a malignant infantile osteopetrosis patient following bone marrow transplantation.

骨髓移植之后的恶性婴儿骨硬化病的成骨细胞和破骨细胞活性。

The osteoclast is a member of the monocyte/macrophage family, decreased or increased number and activity of osteoclasts respectively causes osteopetrosis or osteoporosis and other osteolytic diseases.

破骨细胞来源于单核/巨噬细胞谱系,其数量和活性的不足或增加分别会导致骨硬化症和骨质疏松及其他溶骨性疾病。

Nodes of Ranvier; saltatory conduction; Schwann cells; oligodendrocytes; myelin basic protein; demyelination; remyelination; myelinopathy; myelinogenesis; myelin-associated protein; myelin sheaths; demyelinating neuropathy; myelin proteolytic protein; chronic inflammatory demyelinating polyradiculoneuropathy; multiple sclerosis; MS

郎飞氏结;跳跃传导;施旺细胞;少突神经胶质细胞;髓鞘碱性蛋白脱髓鞘;髓鞘再生;髓鞘质病;髓鞘形成;髓磷脂-关联的蛋白质;髓鞘;脱髓鞘神经病;髓磷脂溶解蛋白的蛋白质;慢性炎症性脱髓鞘多神经根神经病;多发性脑硬化;MS

However, zona hardening could occur during in vitro maturation of oocytes in a simply defined medium which reduced the fertilization rate.

采用无蛋白培养基对卵母细胞进行体外成熟培养,并防止透明带硬化,是研究影响卵母细胞成熟过程因素的基础。

According to the above results, we discuss the effective mechanism of Wenxin capsule anti- atherosclerosis atherosis in detail which is protecting vessel endolethial cell, regulating angiotasis, anti-platleo aggregation and inhibiting proliferation of smooth muscle cell.

依据上述结果对温心胶囊抗动脉粥样硬化作用机理进行了详细论述,认为其作用主要是通过保护血管内皮细胞、调节血管张力、抗血小板聚集及抑制平滑肌细胞增殖而实现的。

Theurinary IL-6 level positively correlated with density of glomerular matrix membrane, global sclerosis, fiber or fibrocellular crescents and interstitial fibrosis (p. 05). According to the degree of density of glomerular matrix membrane and interstitial fibrosis, urinary Col-IV level had better correlation than urinary TGF-betal and IL-6 levels.In IgAN, Col-IV showed increased expression in diseased renal tissue whereas the site of expression of TGF-betal was mainly localized within the cytoplasm of tubular epithelial cells. Interstial expressionwas also present but glomerular TGF-betal expression was found only in patients with heavy mesangial proliferation. There was a significant correlation between glomerular positivity for Col-IV and severity of histological damage. There was also a significant correlation between positivity for TGF-betal and Col-IV in the tubular epithelial and interstitial lesions. In contrast, there was no ralationship between glomerular positivity for TGF-betal and severity of histological damage.The urinary TGF-betal level paralleled tubular TGF-betal expression.

结果 ①IgAN患者尿TGF-β1、IL-6、Col-Ⅳ水平较健康人明显增高(P<0.01),该变化与血中的浓度无关(P>0.05);②尿TGF-β1水平与小管间质TGF-β1阳性表达呈正相关(P=0.000),而与小球TGF-β1阳性表达无关(P>0.05),尿Col-Ⅳ水平与小球和小管间质Col-Ⅳ阳性表达均呈良好的相关性(P<0.01),还与小管间质TGF-β1阳性表达呈正相关(P<0.05):③小球Col-Ⅳ阳性表达与肾组织慢性病变密切相关(P<0.05),小管间质Col-Ⅳ和TGF-β1阳性表达均与肾小管间质病变呈良好的相关性(P<0.01),而小球TGF-β1阳性表达与肾组织损伤无关(P>0.05);④尿TGF-β1、Col-Ⅳ水平与肾小球基质基底膜面密度、小管间质病变呈正相关(P<0.01),与小球内细胞数呈负相关(P<0.05),该结果与其在组织中的表达一致;尿IL-6水平浙江大学硕士学位论文尿TGF一B一、IL一6和Col一IV在IgA肾病中的应用价值与基质基底膜面密度、球性硬化、纤维或细胞纤维新月体所占肾小球百分数及小管间质病变均有显著的相关性(F.05);在轻度肾病理损伤时,尿'l'G卜pl、I卜6、Col一IV水平即升高,而尿Col一W在反映细胞外基质积聚和间质纤维化程度上比尿TGF一pl和IL一6有更好的相关性。

While symptoms may seem to improve almost immediately after surgery, it takes approximately 6-12 months before new vessels develop sufficiently. Diseases associated with moyamoya include sickle cell anemia, atherosclerosis, prior radiation therapy to the skull base in children, and less commonly neurofibromatosis, tuberous sclerosis, meningitis, retinitis pigmentosa, fibromuscular dysplasia, Down's syndrome and Fanconi's anemia.

手术之后几乎立即可以看到症状的好转,新的血供充分发展起来大概要花6-12月时间。moyamoya相关的疾病包括镰状细胞性贫血、动脉粥样硬化、颅底部放疗,不太常见的有神经纤维瘤病,结节性硬化、脑膜炎、色素性视网膜炎、纤维性肌发育不良、Down氏综合症和Fanconi's 贫血症

Results Microscopically, the tumor cells were epithelioid or short spindled and special nests of tumor cells separated by arboring fibrovascular septa. Such morphological variants of CCSK as myxoid degeneration, microcysts formation and sclerosing pattern of extracellular hyalined collagen simulating osteoid were present in some regions.

结果 镜下见瘤细胞为上皮样或短梭形,被分枝状纤维血管间质分隔成巢团状,部分区域见黏液样变性微囊肿和细胞外胶原玻璃样变类似骨样组织的硬化型等形态变异。

FIZZ1 had no effect on the expression of scavenger receptor CD36 in cultured macrophages. FIZZ1 can promote the development of AS by the up-regulation of the expression of SR-A and the phagocytose of fatty substance in VSMCs.

FIZZ1不影响体外培养的巨噬细胞清道夫受体CD36的表达,FIZZ1能促进ox-LDL诱导的体外培养的平滑肌细胞SR-A表达,从而可能加速平滑肌细胞吞噬脂质,促进动脉粥样硬化进展。

METHODS: Rabbits were fed with either high cholesterol or standard chows for 10 weeks.

日本大耳白兔分为对照组和高胆固醇饮食组,饲养10 wk后,检测血脂水平,心电图和室颤阈值,并利用全细胞膜片钳技术记录心室肌细胞的ICa,L,组织化学技术检测动脉粥样硬化发生情况。

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