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硬化

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Meanwhile, the concentration of Pb,Zn and other heavy metals is rather high in the oxidation zone and decreasesdownward to levels that are unbelievably low for Pb-Zn mine tailings. Theseobservations suggest that heavy metals should be re-immobilized by secondaryminerals, rather than primary minerals, in the oxidation zone, and could be leachedout from suboxic environment due to low rate oxidative dissolution which preventsthe precipitation of secondary minerals in significant amount. Thus, cover scenarioswhich establish a suboxic environment beneath the cover layer might not always beeffective in preventing heavy metal release from mine tailings.Sequential extraction studies indicate that, in addition to heavy metal leaching,oxidation of the mine tailings increased the mobility of residual Zn, and Cu, while Pb remains immobile due to the formation of anglesite (PbSO4). Using the seven-step extraction technique developed by Dold (J. Geochem. Explor., 2003, 80: 55-68), galena dissolves in significant amount at step 2 most probably due to the strong complexing of Pb2+ by acetate, while other metal sulfides dissolve at step 6, which suggest that this extraction technique could not be used to evaluate the mobility of lead in natural environment

重金属含量均低于新鲜尾矿,但以硬化层中的含量最高(Pb、Zn含量分别为0.47%和0.93%),往下大幅度降低,表明尾矿风化过程中形成的次生矿物对Pb、Zn等重金属元素有较强的固定作用,而次氧化带的淋溶作用造成了金属元素的大量流失;(4)与新鲜尾矿相比,硬化层中铅的可迁移性大大降低,硬化层之下尾矿样品中铅的可迁移性也不同程度降低,这是由于硫化物氧化产生的硫酸根与尾矿中的铅反应形成铅矾(PbSO_4)的缘故;(5)氧化作用使尾矿中的zn、Cu等重金属的可迁移性不同程度地增大,其中对锌的可迁移性影响最大,铜的可迁移性与铁比较相似,铅则容易与硫化物氧化形成的SO_4~(2-)反应形成铅矾(PbSO_4);(6)为了减少尾矿污染和修复矿山环境,人们提出了各种不同的尾矿覆盖方案,本文的研究结果表明,这些方法不一定能够有效地阻滞污染元素的迁移。

PART ONEInvestigation on Hp CagA+ strain infection condition and pathogenesis risk analyze in patients of carotid atherosclerosisObjective To observate pathological changes of carotid atherosclerosis as well as Hp infection situation, examinate blood fats, blood serum hs-CRP, blood plasma YKL-40, blood serum sCD14, analyze relations between Hp or Hp CagA+ strain and carotid atherosclerosis, and explore possible mechanism of carotid atherosclerosis caused by infection.

然后进行基础动物实验,应用Hp CagA+菌株干预高脂血症大兔,建立Hp CagA+菌株感染致兔颈动脉粥样硬化模型,进行血清Hp抗体、hs-CRP、血浆YKL-40水平检测、斑块内Hp DNA,进一步求证Hp CagA+菌株感染和颈动脉粥样硬化形成的相关性,Hp CagA+菌株感染是否可促进颈动脉粥样硬化的形成,深入探讨其可能机制,以期为颈动脉粥样硬化形成的病因学研究提供实验基础,为临床治疗颈动脉粥样硬化提供新的策略,从而完善其防治模式。

As a result of chemical contains what hard water softeners, so that water to soften the water, flacherie self-aggregation intracelluar intenerated because the loss of water, when huaguang PD developering is water dilution of 1: 4, press ideal calculation, the working fluid anionic surfactant concentration, in its critical micelle concentration.

由于显影洋含有硬火不硬化剂何使火不硬化,火虽不纬不硬化,可火洋的离子含度并未因火的不硬化而减众,当华平PD显影液被火按1:4浓缩后,按理想状况筹算,处事液洋阴离子暗不天活性剂的浓度,在它的临界胶束浓度边。

Result:all the cases were diagnosed as early anfh by x-ray film in 35 sites,stage i showed osteoporosis and osteosclerosis in 15 sites,stage ii showed osteosclerosis and cystic hyalomere in 20 sites.56 cases of early anfh were discovered by ct in 43 sites.stage i stelliform sign distortion and osteoporosis and osteosclerosis in 17 sites,stage ii showed meniscus sign was 26 sites.

结果:全部患者x线平片诊断早期anfh共35个部位,其中ⅰ期占15个,x线表现为骨质疏松及骨硬化;ii期20个,表现为骨硬化和囊状低密度区。ct发现早期anfh共43个部位,其中i期占17个,表现为组成骨小梁星芒结构消失,骨质疏松变和骨硬化;ii期26个,表现为囊状低密度区及骨硬化

There are the normal group,the AS group,the pure swimming group,the swimming plus AS group,the simvastatin group(simvastatin plus AS of overstrain),the large dose Tongxinluo group,the small dose Tongxinluo group,the bulbus allii macrostemonis four drugs group and the ginseng group.

目的对络气虚滞型动脉粥样硬化模型中9个编号组即正常组、动脉粥样硬化组、单纯游泳组、游泳加动脉粥样硬化组、辛伐他汀组(即辛伐他汀+过劳加动脉粥样硬化,下同)、通心络大剂量组、通心络小剂量组、薤白四味组、人参组进行综合评价。

Methods: 1. Rats in the study were divided into six groups: normal control group, hyperlipidemia group (fed with high-cholesterol diet), atherosclerosis group (vitamin D〓 intramuscular injection, endothelium denudate and fed with high-cholesterol diet with vitamin D〓), L-arginine group (besides treatment as atherosclerosis group, 2.25% L-arginine was added in the drinking water), chitosan group (besides treatment as atherosclerosis group, 5% chitosan was added in the diet), simvastatin group (besides treatment as atherosclerosis group, 0.03% simvastatin was added in the diet). The study lasted 90 days.

1。实验大鼠分为6组:正常对照组、高脂血症组、动脉粥样硬化组(维生素D〓肌肉注射加动脉球囊损伤手术加含维生素D〓的高脂饲料饲养)、L-精氨酸组(动脉粥样硬化组基础上饮水中加入2.25%L-精氨酸)、几丁糖组(动脉粥样硬化组基础上饲料中加入5%几丁糖)、辛伐他汀组(动脉粥样硬化组基础上饲料中加入0.03%辛伐他汀),90天后取标本检测。2。

All rats were killed twelve weeks later, the right kidneies were used for observing pathological change, and the expression of MCP-1 in nephridial tissue were detected by immunohistochemical method. Results (1) Change of morphology: the structure of renal tubule was normal in control group, most glomcruluses were segmented sclerosis in GS group, the renal lesions in valsartan group was minorer than this in GS group.

结果 (1)形态学改变:对照组大鼠肾小管结构正常;肾小球硬化组多数肾小球呈节段性硬化,许多肾小球及肾小管受累;缬沙坦治疗组肾脏病变轻于肾小球硬化组,且肾小球硬化指数明显低于肾小球硬化组(P.01);(2)MCP-1的表达:对照组肾小管中有少量MCP-1的表达,在肾小球内几乎不表达,在肾小球硬化组和缬沙坦治疗组肾小球及肾小管内表达皆强于正常对照组(P.01)。

The results were compared among 4 groups. Results:①OCB positive rate was 37% in MS patients, which was lower than other reports, 24% in central nervous system infectious diseases group,5% in central nervous system non-infectious diseases group and 0 in peripheral nervous system diseases group,respectively.②OCB positive rate in conventional MS was 56%, which was higher than that in neuromyelitis optica.

结果①多发性硬化组OCB阳性率为37%,中枢神经系统感染组为24%,中枢神经系统非感染性疾病组为5%,外周神经系统疾病组为0;多发性硬化组的OCB阳性率比其他各组高,但低于国外报道的阳性率;②多发性硬化患者中传统型多发性硬化组OCB阳性率为56%,高于视神经脊髓炎组的11%。

objective to study the relationships between smoking,atherosclerosis and c-reactive protein.methods to detect the serum crp and serum lipid while in quiring the smoking status for278patients with cardio-vascular disease.then analyed the crp level among different groups.results crp level had significant difference between smoking and non-smoking groups,atherosclerosis and non-atherosclerosis groups;but it seems no relationˉship with high lipid.in differentsmoking amount groups,crp level had significant difference in atherosclerosis paˉtients,but no significant difference in non-atherosclerosis patients.conclusion smoking,high lipid and crp were all very good prognostic mark in atherogenesis.crp was a significant prognostic mark in atherosclerosis influenced by smoking.

目的 初步探讨c反应蛋白crp与吸烟和动脉粥样硬化三者之间的关系。方法通过对临床278例动脉粥样硬化和非动脉粥样硬化病人血清crp和血脂的检测及吸烟情况的调查,应用spss统计软件进行差异分析,比较不同组间crp水平。结果 crp在吸烟与不吸烟组、动脉粥样硬化和非动脉粥样硬化组差异有显著性。而在血脂正常组和高血脂组差异无显著性。在不同吸烟量组间:非动脉粥样硬化组间crp水平差异无显著性,而动脉粥样硬化组差异有极显著性。结论吸烟、血脂升高、crp升高是动脉粥样硬化较好的预示指标;在吸烟人群中crp升高者更易患动脉粥样硬化

DSC thermo-analysis showed that both of epoxy resins and blended resins had an exothermic peak during the period of thermal-scanning due to the curing reaction. Comparison between different kinds of liquefied wood, blending resins that using the liquefied Japanese cedar of LW-C as the raw material would undergo more degree of cross-linking reaction. They released more reaction heat, and formed a more complete cross-linking structure. However, heat treatment could promote an advanced cross-linking reaction of room temperature cured resins. DSC isothermosetting analyses showed that the curing reaction of both epoxy resins and blended resins conformed to Arrhenius kinetic model. The time needed to achieve 95% of curing reaction was shorter for blending resin than that of epoxy resin.

由DSC热分析显示环氧树脂与掺合树脂在热扫描过程中均出现硬化反应放热峰,其中以液化柳杉LW-C为原料之掺合树脂在硬化过程可进行较多之架桥反应,其反应放热量较大,硬化树脂之架桥结构较完整,而加热可促进常温硬化树脂进一步之架桥反应;由DSC等温硬化性分析显示环氧树脂与掺合树脂之硬化反应均符合Arrhenius动力学模式,添加液化柳杉之掺合树脂达95%硬化率所需时间较环氧树脂短。

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