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硝化作用

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But, this kind of condition would only occurred when a great deal of nitrobacteria are added into the water.

1去氨硝化菌能将鱼类所排放的氨转成毒性较小的亚硝酸盐,而去亚硝酸硝化菌则负责将亚硝酸盐再转成毒性更小的硝酸盐,硝酸盐必须由换水的方式移除,像硝化细菌这类的细菌再作用时需要消耗水中的氧气所以也称好氧菌。

The results indicate that excellent nitration of NH,-N need the sufficient removal of COD to prevent the restrain of toxicant on the nitrobacteria.

指出NH_3-N完全硝化的前提是废水中COD的充分降解,以消除有毒物质对硝化菌的抑制作用。

Long running performance showed that due to the membrane interception,the nitrobacter is enriched in reactor in the interest of improving the nitrification rate;the maximum ammonia nitrogen loading can be 0.19 kg/(m3·d) with effluent ammonia nitrogen<1 mg/L (removal rate 99%).

采用浸没式膜生物反应器处理焦化废水的试验结果表明:膜的截留作用可使硝化菌在反应器内富集而有利于提高系统的硝化能力,其去除氨氮的最高负荷为0.19kg/(m3·d),出水氨氮<1mg/L(去除率为99%);泥龄长可能使微生物的代谢产物或其他大分子物质积累,从而

A submerged membrane sequencing batch bioreactor was used to treat coke wastewater.Long running performance showed that due to the membrane interception,the nitrobacter is enriched in reactor in the interest of improving the nitrification rate;the maximum ammonia nitrogen loading can be 0.19 kg/(m3·d) with effluent ammonia nitrogen<1 mg/L (removal rate 99%).Long sludge retention time may result in the accumulation of metabolic products and high molecular materials,and thus inhibiting activity of nitrate bacteria and causing a ccumulation of NO2-,which is beneficial to the running of short-cut denitrif ication.However,too long retention time will affect the activity of nitrite bacteria,detrimental to the treatment effect of ammonia nitrogen.

采用浸没式膜生物反应器处理焦化废水的试验结果表明:膜的截留作用可使硝化菌在反应器内富集而有利于提高系统的硝化能力,其去除氨氮的最高负荷为0.19kg/(m3·d),出水氨氮<1mg/L(去除率为99%);泥龄长可能使微生物的代谢产物或其他大分子物质积累,从而抑制硝酸盐细菌的活性,导致NO2-积累而有利于短程脱氮的进行,但泥龄过长也会影响亚硝酸盐细菌的活性,从而影响对氨氮的处理效果。

A submerged membrane sequencing batch bioreactor was used to treat coke wastewater.Long running performance showed that due to the membrane interception,the nitrobacter is enriched in reactor in the interest of improving the nitrification rate;the maximum ammonia nitrogen loading can be 0.19 kg/(m3·d) with effluent ammonia nitrogen<1 mg/L (removal rate 99%).Long sludge retention time may result in the accumulation of metabolic products and high molecular materials,and thus inhibiting activity of nitrate bacteria and causing a ccumulation of NO2-,which is beneficial to the running of short-cut denitrif ication.However,too long retention time will affect the activity of nitrite bacteria,detrimental to the treatment effect of ammonia nitrogen.

简介: 采用浸没式膜生物反应器处理焦化废水的试验结果表明:膜的截留作用可使硝化菌在反应器内富集而有利于提高系统的硝化能力,其去除氨氮的最高负荷为0.19kg/(m3·d),出水氨氮<1mg/L(去除率为99%);泥龄长可能使微生物的代谢产物或其他大分子物质积累,从而抑制硝酸盐细菌的活性,导致NO2-积累而有利于短程脱氮的进行,但泥龄过长也会影响亚硝酸盐细菌的活性,从而影响对氨氮的处理效果。

According to the correlation of the inhibiting concentration to photobacterium and nitrifying bacterium by nitrification substrates and products, the change of luminous intensity of photobacterium can indicate the inhibition from nitrification substrates and products.

根据硝化基质和产物对发光细菌和硝化细菌抑制浓度的相关性,可用发光细菌发光强度的变化指示硝化基质和产物的抑制作用。

The mechanisms of simultaneous nitrification and denitrification,short-cut nitrification and denitrification,and denitrifying phosphorus removal are discussed.

膜生物反应通过膜的高效截留作用,可维持反应器内高浓度的生物量,满足硝化菌的生长增殖需求,强化活性污泥的硝化能力。

To determine whether nitration of Trx may alter its cardioprotective effect,adult mice were subjected to 30 minutes myocardial ischemia and 3 hours or 24 hours of reperfusion and treated with vehicle, native or nitrated human Trx1 (10 min before reperfusion).

为进一步确定TRX硝化是否改变其心肌保护作用,采用缺血30 min,再灌注3 h或24h小鼠缺血/再灌注模型,分别于再灌前10 min给予磷酸盐缓冲液,TRX或硝化TRX。

Oxidative and nitrative injury in human umbilical vein endothelial cells (ECV304) induced by high glucose and exogenous nitrating agent were detected by cell biological and chemical methods.

采用细胞生物学及化学方法,检测了高浓度的糖和外源性硝化试剂作用于人脐静脉内皮细胞(ECV304)后所导致的细胞蛋白氧化损伤及硝化损伤。

At higher concentrations hydrogen bondings formed due to the residual hydoxyl groups of nitrocelluloses lead to viscoities only a weak function of shear rates.

在较高浓度时,硝化纤维素分子链上未硝化的羟基所形成分子间的氢键作用,使黏度不因剪切率升高而急遽降低。

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