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破骨细胞

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Results:Compared with control,the re were significant increase in bones length and length of their diaphyses after treated with 10-7 mol/L estrone.

16 d胎龄雌性小鼠尺骨在含有雌酚酮的BGJb培养基中旋转培养48 h后,测量骨总长和骨干长度;H.E染色,光镜下计数破骨细胞和肥大软骨细胞。

Methods Bone metabolism markers were measured and their correlations with thyroid hormones were analyzed in 49 patients with Graves' disease.

Graves病患者在过量甲状腺激素作用下,成骨细胞和破骨细胞的活性和数量发生变化,导致骨代谢紊乱。

Osteoblast and osteoclast activity in a malignant infantile osteopetrosis patient following bone marrow transplantation.

骨髓移植之后的恶性婴儿骨硬化病的成骨细胞和破骨细胞活性。

The osteoclast is a member of the monocyte/macrophage family, decreased or increased number and activity of osteoclasts respectively causes osteopetrosis or osteoporosis and other osteolytic diseases.

破骨细胞来源于单核/巨噬细胞谱系,其数量和活性的不足或增加分别会导致骨硬化症和骨质疏松及其他溶骨性疾病。

The promotion of osteoblastic differentiation of rat bone marrow stromal cells by a polyvalent plant mosaic virus.

多价植物的花叶病毒造成的鼠类骨髓基质造骨细胞的分化促生。 4。破骨细胞种植及再吸收分析===================尴尬。。

In this study, we investigated the expression of osteoblast differentiation and osteoclast differenation by adlay extract, benzopyrene and tributyltin, three environmental estogens.

在本实验中,我们观察三种环境雌激素样物质:薏苡仁水萃取物、苯骈芘及三丁基锡对造骨细胞和破骨细胞分化的影响。

LRP5 is expressed in the osteoblasts lining the endosteal and trabecular bone surfaces, but not in osteoclasts.

LRP5在骨膜内及骨小梁表面的成骨细胞表面表达,而不在破骨细胞中表达。

Results: Immunohistochemical staining revealed that expression of RANKL protein was detected in periodontal ligament cells and osteoclasts which mostly located in resorption lacunae.

结果:正畸牙移动压力侧组化结果显示,RANKL阳性表达位于牙周膜细胞和位于骨陷窝内的破骨细胞中,在正畸牙移动第3、5和7天时呈强阳性表达。

Results High homogenous hMSCs were obtained and after co-culture for 5 d with osteoblasts, they fused to form multinuclear cell clusters in micrograph and were postive in TRAP staining. Conclusion The co-culture system method is successful.

结果:体外分离、培养的hMSCs具有独特的细胞免疫学表型,即CD73和CD105阳性, CD34和CD45 阴性。hMSCs与成骨细胞共同培养5 d后,即可见单个核细胞融合成多核细胞,TRAP 染色可见多数细胞胞浆呈酒红色,为诱导成功的破骨细胞

We have, therefore, investigated the direct effects of adiponectin on primary cultures of osteoblastic and osteoclastic cells in vitro and determined its integrated effects in vivo by characterizing the bone phenotype of adiponectin-deficient mice.

为此,我们研究了脂联素对离体培养的原代成骨细胞和破骨细胞的直接影响,并通过对脂联素缺陷小鼠的骨基因表型的研究明确其在体内的综合作用。

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