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No structural change was found in the 4th group. Conclusion The test prove that cholestasis would occure during TPN and become serious with time prolonging. Integrity and function of gallbladder-wall tissue cell could be defended and sustained by applying GLN, but there is no direct preventing action. There is apparent cholecy stokinetic and cholagogic fundations by applying CCK. But CCK would lose its function if gallbladder-wall was damaged. The test prove that TPN+GLN+CCK is the best way to prevent cholestasis during TPN.

TPN时间越长,胆汁淤积越重; GLN虽无直接收缩胆囊作用,但它可保护胆囊壁组织结构完整,而CCK虽有收缩胆囊、减轻胆汁瘀积作用,但晚期当胆囊壁组织结构破坏时,其作用减弱;联合应用GLN和CCK可能是预防TPN期间胆汁瘀积的一种方法。

Results After lengthing, obviously histological variety was observed on the sections of condyloid process and intercondylar disc,but it could be repaired under 1mm/d distraction rate.

结果:牵张后大体观察可见关节盘无显著形态异常,髁状突有不同程度适应性改建;组织学主要表现为关节盘不同程度中带变薄,纤维走行改变,血管侵入,后带变厚;髁状突表现为纤维软骨组织形态学的改变和软骨、骨的改建活动,2mm/日组可见破坏现象发生。

Effects of MAP on intrinsic quality of strawberry(Fragaria ananassa Duch.) during shelf-life extention storage were studied in this paper.

植物组织或器官在衰老过程中常伴随细胞膜结构的破坏,导致组织结构和细胞区域化的丧失。

By numerical method,the relationship of the temperature with irradiated time is obtained.

具有一定强度的激光辐照皮肤组织时可造成组织的破坏。

Results:(1) FSN can inhibit secondary joints swelling and multi-arthritis evidently, improve the whole condition of rats; at the same time, it can also lighten the synovial inflammation and hyperplasia of lesion joints distinctly, and prevent the joint cartilage and bone from destruction; the collective efficiency of FSN is better than TWP.(2) FSN can raise AA rats low LTT of spleen to normal nearly, remedy the disorder of Th / Ts and Thl / Th2 cells balance in peripheral blood, restrain exorbitant TNF- a ? IL-1 produced by PM O , thereof exert anti-inflammatory and immunoregulation effect.(3) Inside the lesion joints, FSN can depress abnormal hyper-expression of TNF- a mRNA and NF-kB in synovial tissues, as well as advance the expression of Caspase-3 (a proteolytic enzyme of apoptosis), reduce synovial immflamination and proliferation.(4) FSN can lower the expression of VEGF in synovial tissue,reduce neogenetic veins, so inhibit the growth of pannus tissue and the damage of cartilage and bone by that.(5) The above effect of FSN are in proportion to its dosage.Conclusions: FSN has inhibitory effects on symptom and condition of experimental RA, which is better than TWP as a whole.

结果:(1)FSN有明显的抗炎作用,可显著抑制AA大鼠足跖肿胀与多关节炎,改善大鼠的全身情况,同时明显减轻病变关节滑膜炎症与增生,防止关节软骨及骨质的破坏,其综合作用优于TWP;(2)FSN还能使AA大鼠低下的脾LTT恢复至接近正常,纠正外周血中T细胞亚群Th/Ts及Th1/Th2平衡紊乱,抑制大鼠PHφ过高的TNF-α、IL-1分泌,而发挥抗炎和免疫调节作用;(3)在病变关节局部,FSN能显著抑制AA大鼠滑膜细胞异常增高的TNF-αmRNA的表达,降低滑膜组织中NF-kB表达,并增加凋亡蛋白酶Caspase-3表达,从而抑制滑膜的炎症与增殖;(4)FSN还可下调滑膜组织中VEGF表达,减少血管新生,由此抑制血管翳的形成及其对软骨和软骨下骨的侵蚀;(5)FSN的上述作用皆呈现一定的量效依赖关系,高剂量组作用优于低剂量组。

FSN can raise AA rats low LTT of spleen to normal nearly, remedy the disorder of Th/Ts and Th1/Th2 cells balance in peripheral blood, restrain exorbitant TNF-α、IL-1 produced by PMφ, thereof exert anti-inflammatory and immunoregulation effect.(3) Inside the lesion joints, FSN can depress abnormal hyper-expression of TNF-αmRNA and NF-kB in synovial tissues, as well as advance the expression of Caspase-3 (a proteolytic enzyme of apoptosis), reduce synovial immflammation and proliferation.(4) FSN can lower the expression of VEGF in synovial tissue, reduce neogenetic veins, so inhibit the growth of pannus tissue and the damage of cartilage and bone by that.(5) The above effect of FSN are in proportion to its dosage. Conclusions: FSN has inhibitory effects on symptom and condition of experimental RA, which is better than TWP as a whole.

结果:(1)FSN有明显的抗炎作用,可显著抑制AA大鼠足跖肿胀与多关节炎,改善大鼠的全身情况,同时明显减轻病变关节滑膜炎症与增生,防止关节软骨及骨质的破坏,其综合作用优于TWP;(2)FSN还能使AA大鼠低下的脾LTT恢复至接近正常,纠正外周血中T细胞亚群Th/Ts及Th1/Th2平衡紊乱,抑制大鼠PMφ过高的TNF-α、IL-1分泌,而发挥抗炎和免疫调节作用;(3)在病变关节局部,FSN能显著抑制AA大鼠滑膜细胞异常增高的TNF-αmRNA的表达,降低滑膜组织中NF-kB表达,并增加凋亡蛋白酶Caspase-3表达,从而抑制滑膜的炎症与增殖;(4)FSN还可下调滑膜组织中VEGF表达,减少血管新生,由此抑制血管翳的形成及其对软骨和软骨下骨的侵蚀;(5)FSN的上述作用皆呈现一定的量效依赖关系,高剂量组作用优于低剂量组。

NB can make liver, brain, spleen and nephridial tissue pathology damage, to refrain theactivity of liver mixed function oxidase, induce oxidative stress, effect tissue enzymatic activity,destroy organism antioxygen defence system and function of clean up free radical, to cause proteinoxidation damage, to educe toxic action.

综上所述,NB可以造成肝、脑、脾、肾组织的病理学损伤,抑制肝内混合功能氧化酶的活性,诱导氧化应激,影响组织酶活性,破坏机体抗氧化防御系统及清除自由基功能,引起机体蛋白质氧化损伤,进而发挥其毒性作用。

The cell processes showed up from the edge of explants and neuroglial cell migration was observed at the early stage.

培养早期上丘脑组织块边缘出现细胞突起,胶质细胞迁移,随着培养时间延长,细胞出现凋亡,组织结构破坏。

The treatment to periodontal disease is mainly combined therapy,which control inflammation by periodontal medicine and operation;adjust interlocking,eliminate dental wound,relieve the burden of peridental supporting tissue by recovery treatment;fix the loose teeth,disperse the bite force,control the patho-loose and displacement,elevate the masticatory efficiency,make the troubled teeth to rest,is benefit to the control to periodontal inflammation and the healing of destructed periodontium.

牙周病治疗主要是综合治疗,通过牙周药物治疗、手术治疗来控制炎症;通过修复治疗调整咬合,消除牙创伤,减轻牙周支持组织的负担;固定松牙,分散力,控制病理性松动和移位,提高咀嚼效能,使患牙得到生理性休息,利于牙周炎症的控制和已破坏的牙周组织的愈合。

In order to analyze the association of the periodontitis severity with changes of the human dental pulp, one hundred caries-free and restoration-free periodontally involved teeth with different periodontic destruction were studied histologically.

为了分析牙周炎的病变程度与牙髓组织改变的关系,对100个牙体完好的具有不同牙周破坏程度的牙周炎患牙进行了组织学观察,分析牙根暴露量、牙周袋深度和牙齿松动度与牙髓病变的关系。

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With Death guitarist Schuldiner adopting vocal duties, the band made a major impact on the scene.

随着死亡的吉他手Schuldiner接受主唱的职务,乐队在现实中树立了重要的影响。

But he could still end up breakfasting on Swiss-government issue muesli because all six are accused of nicking around 45 million pounds they should have paid to FIFA.

不过他最后仍有可能沦为瑞士政府&议事餐桌&上的一道早餐,因为这所有六个人都被指控把本应支付给国际足联的大约4500万英镑骗了个精光。

Closes the eye, the deep breathing, all no longer are the dreams as if......

关闭眼睛,深呼吸,一切不再是梦想,犹如。。。。。。