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Since the beginning of this year, with plot and abetment of Li Hongzhi and the overseas Falun Gong headquarters, the underground Falun Gong organization in China secretly contact each other, purchase, equip and disseminate large quantity of "timely broadcasting systems", illegally propagate cult throughout China, undermine social order and endanger the stable and united political circumstances.

今年以来,在李洪志及海外"法轮功"组织总部的策划、唆使下,境内"法轮功"地下组织秘密串联,组织购买、装配、散发大量"定时播音装置",在全国范围内非法宣扬邪教,危害社会秩序,破坏安定团结的政治局面。

One hand mechanical obstruct led to the increase of veinous resistance and the obstacle of microcirculation, the other hand the adhesive PMN was activated in excess, the white blood cells released a lot of enzymes, in which PMN-elastase can decompose the components of cell and many albumens, inclusive of immunoglobulin、alexin and fibrication. These components induced the injury of the pancreatic capillary vessels and cell and lysosome enzy made the tissue protein hydrolyze and produced unsaturated fatty acids, which destroyed the structure and function of cellar membrane. The inflammatory cellar factors activate other immunocytes to produce the injury and necrosis of tissue, which aggravated the pathological injury and led to shock、pyaemia and MODS. So ICAM-1 and LFA-1 played an important role in SAP. Frossard found that the expression of ICAM-1 in the rat model, especially in serum、pancreas and lung. All these showed ICAM-1 is an important factor in AP and concomitant lung injury.

胰腺小叶组织局部血管EC首先被激活,ICAM-1表达升高,与被激活的PMN表面表达的LFA-1相结合,"PMN-EC"相互作用加剧,一方面机械性阻塞毛细血管导致静脉阻力增加、微循环障碍;另一方面粘附的PMN过度吞噬或激活,当白细胞吞噬的颗粒不能被封闭隔离,连同细胞内的酶被释放出来,其中的PMN-elastase能够降解细胞基质中各种成分,水解多种蛋白,加重胰腺的毛细血管内皮细胞和腺泡的损伤;释放的溶酶体酶使组织蛋白水解,产生的不饱和脂肪酸引发脂质过氧化方应,破坏细胞膜的结构和功能;释放的炎性细胞因子,激发其他的免疫细胞的功能,导致进一步的组织损伤和坏死,加重SAP的病理损伤,最终导致休克、脓毒血症及多器官功能障碍等严重后果。

After spores penetrated into the hemocoele, as the hyphae propagated and grew in the coelum, pathological changes occurred on all infected tissues and organs, including cuticle separating from epidermis, incompaet of fat body, disorganized digestive tract, crack in the muscle tissue and the disintegrated trachea tissue.

白僵菌侵入昆虫血腔后,随着菌丝在体腔内大量繁殖,受侵的各器官和组织均发生明显的病变,如皮细胞层和表皮分离、脂肪体松散、消化道被解体、肌肉组织出现裂缝、气管组织被解体破坏等。

Results Compared to those before treatment the volume of the implanted keloid of Group D began to decrease since 14 days after treatment time-dependently (all P 《 0.05), and the volumes of the other 3 groups continued to increase and peaked on days 21, 14, or 7 respectively (all P 《 0.05).Microscopy showed infiltration of a larger quantity of histiocyte in the keloid tissue, and more obvious collagen disorganization and apoptosis of fibroblasts in Group D than in the other 3 groups.The protein expression of Bcl-2 was more remarkable and the protein expression of BAX was less remarkable in Group D than iu the other 3 groups.

结果 用药前和用药后7、14、21、28、35 d,D组瘢痕疙瘩组织块体积(mm3)分别为173±5、172±5、147±5、125±6、112±7和84±9,从用药后14 d开始明显缩小(均P<0.05);而其他3组瘢痕疙瘩组织块体积均明显增大,从用约后7 d开始各时点测得的体积均明显大于D组(均P<0.05)。D组瘢痕疙瘩组织中有大量小鼠组织细胞浸润,胶原结构破坏和成纤维细胞凋亡明显重于其他3组,Bcl-2蛋门质表达明显弱于而Bax蛋白质表达明显强于其他3组。

Results (1) Osteitis and destruction of end-plate showed long T1 and long T2.(2) Anterior and lateral abscess had long T1 and long T2 presenting even and structure-less signal.(3) The intervertebral discs showed normal or only degeneration at the early stage, and destroyed at later stage.(4) With Gd-DTPA enhancement, fibrotic granulation around abscess and destroyed area was well demonstrated.(5) Abscess in the bone and/or paraspinal abscess, destruction of end-plate were the important evidence for early diagnosis.

结果 (1)椎体骨炎及终板破坏呈长T1长T2信号,(2)椎旁或椎前冷脓肿呈长T1长T2均匀无结构信号,且上下跨越范围较大,(3)早期椎间盘信号正常或只出现退变,晚期则被破坏,(4)Gd-DTPA增强扫描可清楚显示冷脓肿周围纤维肉芽组织及椎管内侵犯,(5)骨内小脓肿和/或椎旁脓肿、椎体终板破坏是MRI诊断早期脊柱结核的重要依据。

It is evident that the plerocercoids parasitize mainly in the subcutaneous tissues and muscles of mice, and mice experimentally infected with plerocercoids show different degree of histopathological changes.

小鼠感染裂头蚴后,多组织器官有裂头蚴寄生,其中以皮下肌肉处多见。裂头蚴寄生可引起组织不同程度的增生、变性、坏死,组织结构破坏及大量炎症细胞浸润等病理变化。

This report presents 2 cases of inadvertent injection of sodium hypochlorite into the periradicular tissue because of apical constriction destruction and canal perforation. Acute severe pain, gingival swelling, cellulites, and even parathesis over the circumscribed region of the upper lip were noted.

本病例报告是两例有关根管治疗过程中,因治疗过程不愼使根尖孔破坏与穿孔,并将根管冲洗溶液次氯酸钠挤压至牙根周围组织中,造成急性疼痛、牙龈肿胀、蜂窝组织炎,甚至有局部性的嘴唇麻痹现象等剧烈组织反应。

Foreign gene could destroy larvas mid-intestine, and reduce its appetite. Observation by microscope showed that the gaps between mid-intestine cells was extended in treated larvae, and the nucleus were destroyed, the gap between peritrophic membrane and mid-intestine cells was appeared, parts of peritrophic membrane was broken off from mid-intestine cells.

外源基因对舞毒蛾幼虫的杀虫机理在于破坏幼虫的中肠组织,影响幼虫的取食,使幼虫取食量减少,甚至终止取食;中肠细胞间隙增大,细胞核遭受破坏;围食膜与中肠细胞之间出现空隙,甚至脱落,围食膜不再完整;中肠组织脱落,成为一个严重变形的残架。

One hand mechanical obstruct led to the increase of veinous resistance and the obstacle of microcirculation, the other hand the adhesive PMN was activated in excess, the white blood cells released a lot of enzymes, in which PMN-elastase can decompose the components of cell and many albumens, inclusive of immunoglobulin、alexin and fibrication. These components induced the injury of the pancreatic capillary vessels and cell and lysosome enzy made the tissue protein hydrolyze and produced unsaturated fatty acids, which destroyed the structure and function of cellar membrane. The inflammatory cellar factors activate other immunocytes to produce the injury and necrosis of tissue, which aggravated the pathological injury and led to shock、pyaemia and MODS. So ICAM-1 and LFA-1 played an important role in SAP. Frossard found that the expression of ICAM-1 in the rat model, especially in serum、pancreas and lung. All these showed ICAM-1 is an important factor in AP and concomitant lung injury.

胰腺小叶组织局部血管EC首先被激活,ICAM-1表达升高,与被激活的PMN表面表达的LFA-1相结合,&PMN-EC&相互作用加剧,一方面机械性阻塞毛细血管导致静脉阻力增加、微循环障碍;另一方面粘附的PMN过度吞噬或激活,当白细胞吞噬的颗粒不能被封闭隔离,连同细胞内的酶被释放出来,其中的PMN-elastase能够降解细胞基质中各种成分,水解多种蛋白,加重胰腺的毛细血管内皮细胞和腺泡的损伤;释放的溶酶体酶使组织蛋白水解,产生的不饱和脂肪酸引发脂质过氧化方应,破坏细胞膜的结构和功能;释放的炎性细胞因子,激发其他的免疫细胞的功能,导致进一步的组织损伤和坏死,加重SAP的病理损伤,最终导致休克、脓毒血症及多器官功能障碍等严重后果。

It calls on them to pool their resources and increase their cooperative efforts,because many of these crimes are too complex for one group to handle,especially those requiting a vast network of fences.

有组织的犯罪团伙也直接参与:新技术为犯罪提供了无数的机会:如数据信息犯罪、偷窃服务项目、与财产有关的犯罪、工业破坏、与政治有关的破坏、对文化的破坏、针对个人的犯罪和与金融有关的罪行等。

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