皮质

Electrocorticogram was recorded with silver electrodes positioned through the burr holes on both sides, and at same time, myoelectricity activity induced by clonic jerks of the extremities was recorded with the electrode inserted into the quadriceps femoris muscle of the left posterior limb.

同时在左右两侧钻开的颅骨里放置银制电极，记录皮质脑电图；在大鼠左后肢股四头肌插入肌电记录电极，记录肢体抽搐引起的肌电活动。

Methods Detailed preoperative evaluation was performed electrocorticogram monitoring was used during the microsurgery. The operations included the resection of the epileptogenic foci, anterior temporal lobectomg, anterior temporal lobectomg and hippocampectomy, corpus callosotomy.

全部病例术前进行详细评估，术中采用皮质脑电监测，手术方式采用显微手术方法，包括致痫灶切除术，前颞叶切除术，前颞叶及海马切除术和胼胝体切开术。

The changes of SEP contents of cerebral cortex NO and SOD, and the changes of the ultrastrcture were recorded by evoked potential electrograph before and after MCAO.

利用诱发电位肌电图测试仪，测各组MCAO前后SEP的变化，脑皮质NO、SOD含量，及电镜超微结构的改变，观察三七皂苷对MCAO性脑损伤的保护作用。

AIM: To investigate the modification by glucocorticoid on CD4 CD25 regulatory T cells in mice with experimental autoimmune encephalomyelitis.

目的：研究糖皮质激素在实验性变态反应性脑脊髓炎治疗中对调节性T细胞的调变作用。

Taken together, PIASy represses the expression of CYP11A1 gene in H295 and may participate in regulating steroidogenesis in endocrine organs like adrenal gland.

我们的结论是，PIASy可以抑制CYP11A1基因的表现，因此可能参与肾上腺皮质等内分泌器官中固醇类荷尔蒙生成的调控。

Objective To observe the effect of Dexamethasone combined with Ornidazole on treatment of endodontic interappointment emergencies.

目的 观察联合应用糖皮质激素与奥硝唑治疗根管治疗期间急症的效果。

RESULTS On the experimental side, there was new bone formation in the bone medullary cavity, the endosteum and the section surface of the cortex at 2 weeks. At 4 weeks, at the center of the blood clot in the tube there was new bone formation and bone defect was completely healed at 8 weeks.

结果 实验侧术后2 周即在骨断端髓腔、骨内膜及皮质断面处有新骨形成，并长入管内血肿，术后4周新骨长入血肿中心，8周完全骨愈合。

Results: 1 The capacity of engulfment of A549 cells started enhancing at the 1h of preincubation with dexamethasone, reached the highest at the 4h of preincubation,At the 8h、24h of preincubation the capacity of engulfment evidently reduced. 2 Glucocorticoid cytosolic receptors antagonist RU38486 inhibited the improved engulfment ability by dexamethasone. 3Phagocytosing apoptotic eosinophils did not change the release of IL-6 and IL-8,but can inhibited the release of IL-6 and IL-8 from A549 cells stimulated by lipopolysaccharide. 4Increasely uptaking by dexamethasone also did not agitate the release of IL-6 and IL-8 from A549 cells. Conclusion: 1Dexamethasome improve the phagocyte capacity for apoptotic eosinophils of A549 cells by glucocorticoid cytosolic receptors, and the effect presents time-dependently. 2Phagocytosis of apoptotic eosinphils can degrade inflammatory reflection, so it is likely to be critical in the control of asthma inflammation.

结果： 1 A549细胞在地塞米松诱导后1小时吞噬能力开始升高，4小时吞噬能力达最高峰，8小时、24小时吞噬能力明显下降 2加入RU38486后抑制了地塞米松所增强的吞噬能力 3凋亡嗜酸粒细胞的吞噬并不能改变A549细胞释放IL-6和IL-8，但能抑制脂多糖刺激A549细胞后IL-6、IL-8的分泌 4地塞米松增加的吞噬也不激发A549细胞释放IL-6和IL-8 结论： 1地塞米松促进A549细胞吞噬凋亡的嗜酸粒细胞是通过A549细胞糖皮质激素胞浆受体发挥作用的，并且具有时间依赖性 2 A549细胞吞噬凋亡的嗜酸粒细胞能降低炎症反应，对哮喘炎症的控制具有重要意义

Methods 2: 25 New Zealand long-eared rabbits, equability quantity of male and female supplied by Xinjiang Medical University, were randomly divided into 5 groups, as following (n=5): control group, OA 30, 60, 90, 120min group, observe the activity of Na~+-K~+-ATP ase, LDH.

随机分为5组：(1)对照组(n=5)：从耳缘静脉缓慢注入生理盐水0.1ml／kg。(2)实验组(n=20)：从耳缘静脉缓慢(2min内)注入OA 0.1ml／kg体重，以30、60、90和120min为时限又分为4组(每组5只)，检测脑皮质组织中Na~+。K~+-ATP酶活性和乳酸脱氢酶活性。

It indicates the improvement of ERG following steroids treatment may due to the direct effect of the drug on retina.

提示糖皮质类固醇治疗后ERG的改善可能是药物对视网膜的直接作用引起。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。