皮质

Research laryngeal mask and tracheal intubation for patients cortisol and ACTH (Adrenocorticotropic Hormone, ACTH) impact.

研究喉罩与气管插管对病人皮质醇（Cortisol，COR）和促肾上腺皮质激素(Adrenocorticotropic Hormone，ACTH)的影响。

Four rats of either group were sacrificed at the day of 2,4,6,8,10, separately; the level of blood glucose and the contents of adrenocorticotropic hormone,orticosterone and adrenaline were measured as the stress markers,as well as the relative weight of the thymus and spleen.

于第2、4、6、8和10天分批处死大鼠，检测应激指标血糖、促肾上腺皮质激素、皮质类固醇、肾上腺素含量，测量胸腺和脾脏重量与体重的相对比值。

Plasma adrenocorticotropic hormone, plasma, and salivary cortisolwere measured serially.

连续测量血浆促肾上腺皮质激素，血浆和唾液皮质醇。

Results of HE dye after cerebral alchemic reperfusion showed: Cortex was infiltrated by neutrophil; Cells of corpus striatum and cortex of parietal lobe were stained light; the number off cells obviously reduced; the feature of cells necrosis, such as cells swelling, nuclear fragmentation and lysis et al, would be observed.

HE染色结果显示：脑缺血再灌注后，皮质区中性白细胞浸润，纹状体外侧和顶叶皮质细胞染色淡，细胞数明显 NF－。B在脑缺血再灌注后脑组织局部炎症反应中的作用减少，出现细胞肿胀，胞核碎裂和溶解等细胞坏死特征。

Results: The changes following hydrocortisone intervention of rats could be identified by PCA, and the kidney deficiency syndrome rats entered a state of "hyperfunction", involving a series of changes in metabolic pathway and metabolic network. The response integral area of lactic acid (δ 1.37) increased, indicating the accumulation of metabolites of lactic acid. The amount of dimethylamine (δ 2.72) also increased, indicating that the disorder of kidney function was related to kidney-deficiency syndrome. The response of creatinine (δ 3.87), asparaginic acid (δ 2.83), taurine (δ 3.44, 3.28), and hippurate (δ 7.84, 7.56, 7.64, 3.97) all decreased significantly, indicating that the hydrocortisone might cause adrenal cortex excretion injury. The response integral area of succinate (δ 2.41) and citric acid (δ 2.53, 2.68) decreased, which was usually caused by disorder of the mitochondrial function.

结果：PCA方法处理肾虚证和空白对照大鼠尿液数据显示，两组大鼠的数据可以在得分图实现分类，与对照组比较肾虚组大鼠由于氢化可的松诱导，大鼠机体进入一个过消耗后衰弱的状态，相关代谢发生显著变化，乳酸(δ 1.37)代谢发生堆积；二甲胺(δ 2.72)的含量增加，提示肾虚与肾功能异常是密切相关的；天冬氨酸(δ 2.83)，牛磺酸(δ 3.44， 3.25)，马尿酸(δ 7.84， 7.56， 7.64， 3.97)，肌氨酸(δ 3.87)等的相对积分面积明显下降，这预示糖皮质激素可能引起了肾上腺皮质分泌功能的损害；琥珀酸(δ 2.41)和柠檬酸(δ 2.53， 2.68)是三羧酸循环能量代谢及糖酵解的中间产物，其含量的降低，通常是由于线粒体功能紊乱所引起。

MethodsNonheparinized autohemic blood of the rat was taken out from rat"s femoral artery and injected into its caudate nucleus immediately to establish a model of intracerebral hemorrhage.The changes of edema in the ipsilateral bassal ganglia , ipsilateral cortex, contralateral bassal ganglia and contralateral cortex were measured at 1,2,3,5 and 7 days respectively after intracerebral hemorrhage. The rat"s packed red blood cells, lysed red blood cells and hemoglobins at different concentrations were injected respectively into the rat"s caudate nucleus. The changes of edema in various regions of the brain at different time were measured as above. 5U thrombin was injected into the rat"s caudate nucleus and the changes of edema in various regions of the brain at different time were measured as above.

自体股动脉取血，并采用自体未肝素化新鲜血回注法建立大鼠尾状核出血模型，观察脑出血后1，2，3，5，7天血肿同侧基底节区、血肿同侧对应的皮质区、血肿对侧对应的基底节区及血肿对侧对应的皮质区脑组织水分含量的变化；分别以自体浓缩红细胞(PRBC，50％)、溶解红细胞(LRBC，50％)、不同浓度的大鼠血红蛋白注入大鼠尾状核，并观察不同时间不同部位脑组织水分含量的变化；以5U凝血酶注入大鼠尾状核，观察不同时间不同部位的脑组织水分含量的变化。

Results showed:(1) The airway responsiveness of smoke control group was higher than that of normal control group, the airway responsiveness of smoke + exercise group was lower than that of smoke control group;(2) Plasma level of cortisol determined immediately after exercise was higher than that determined before exercise, no significant difference occurs between that determined next day morning after all exercise finished in contrast to that determined before exercise;(3) HE staining showed, there was severe chronic pulmonary inflammatory response in smoke control group, which was reduced in the smoke + exercise group;(4) chronic cigarette smoke downregulated the protein and mRNA expression of BKca in smooth muscles of bronchi and bronchioli, exercise increased the mRNA expression of BKca;(5) chronic cigarette smoke downregulated the protein and mRNA expression of Kv1. 5 in smooth muscles of bronchi and bronchioli, exercise increased both the protein and mRNA expression of Kv1. 5 in contrast to smoke control group in bronchioli and did not effect the expression of Kv1. 5 in bronchi;(6) chronic cigarette smoke induced upregulation of FIZZ1/RELMα in bronchial smooth muscle cells, exercise decreased these effect.

结果：(1)吸烟对照组的气道反应性明显增高，吸烟加运动应激可明显降低大鼠的气道反应性；(2)吸烟运动组运动后血浆皮质醇浓度明显高于运动前，但全部运动结束后次日晨测定的血浆皮质醇浓度与运动前无明显差异；(3)HE染色显示，吸烟对照组肺组织出现明显的慢性炎症反应，吸烟加运动应激组比吸烟对照组炎症反应轻；(4)慢性吸烟可降低大鼠大气道和小气道BKca mRNA和蛋白表达，而吸烟加运动应激可使BKca mRNA表达上调，但对小气道的蛋白表达无影响；(5)慢性吸烟可降低大鼠大气道和小气道Kv1.5蛋白和mRNA表达，在小气道吸烟加运动应激可减轻这种作用，大气道则无作用；(6)吸烟使支气管平滑肌的FIZZ1/RELMα蛋白表达明显增强，而吸烟加运动组的RELMα蛋白及mRNA均明显减弱。

Low signal was also seen in the motor cortex on T2-weighted images in three of the eight normal volunteers and four of the 16 disease controls: one with a bulbospinal muscular atrophy, two with leukoencephalopathy and one with Binswanger's disease.

在8例正常人对照者中有3例、16例其他神经疾病患者中有4例（1例脊髓延髓性肌萎缩症患者、2例表现为下肢痉挛性轻瘫的不明原因的白质脑病患者、1例表现为痴呆和下肢痉挛性轻瘫的皮质下动脉硬化性脑病患者）在T2像上于运动皮质区也可见低信号。

Cortical nephrocalcinosis is seen as tiny calcific foci in the cortex Fig.

肾皮质钙质沉着表现为皮质内的微小钙化点（图5）。

Other major features include: partial or complete callosal agenesis, cortical malformations periventricular and subcortical heterotopias, cysts around the 3rd ventricle and/or choroid plexuses, papillomas of choroid plexuses, hypoplastic cerebellum, retrobulbar cysts, microophthalmia and colobomas.

其他主要表现包括：部分或完全的胼胝体发育不良，皮质畸形，脑室周，皮质下异位，环绕三脑室和脉络丛囊肿，脉络丛乳头状瘤，小脑发育不良，眼球后囊肿

According to the clear water experiment, aeration performance of the new equipment is good with high total oxygen transfer coefficient and oxygen utilization ratio.

曝气设备的动力效率在叶轮转速为120rpm～150rpm时取得最大值，此时氧利用率和充氧能力也具有较高值。

The environmental stability of that world - including its crushing pressures and icy darkness - means that some of its most famous inhabitants have survived for eons as evolutionary throwbacks, their bodies undergoing little change.

稳定的海底环境─包括能把人压扁的压力和冰冷的黑暗─意谓海底某些最知名的栖居生物已以演化返祖的样态活了万世，形体几无变化。