白血病的
- 与 白血病的 相关的网络例句 [注:此内容来源于网络,仅供参考]
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Immunology and cytogenetics can improve the correct rate of diagnosis , and be benefical to personalized therapy.
细胞遗传学检查已成为急性白血病的诊断、个体化治疗、治疗监测、预后判断和发病机制研究的不可缺少的重要手段。
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The establishment of this method can help and guide to the clinical diagnoses of leukemia.
此法的建立为白血病的临床研究提供了一种有效的手段。
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For example, a vaccine made against chronic myelogenous leukemia might be used for any patient with the disease.
例如,慢性粒细胞白血病的疫苗就可以用于任何患有该病的病人身上。
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Objective:To evaluate the antileukemic activity and side effects of topotecan in patients with refractory and relapsed acute myelogenous leukemia.
目的:探讨以拓扑替康为主的联合化疗方案对复发难治性急性髓系白血病的疗效及不良反应。
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The purpose of this study was to investigate the mechanism of effects of interferon-alpha on chronic myeloid leukemia.
本研究探讨干扰素治疗慢性粒细胞性白血病的可能新机制,为临床治疗CML提供新的思路。
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Objective To investigate the clinic effects of standard and improved FLAG regimen in treatment of refractory and relap sed acute myeloid leukemia.
目的研究标准和改良FALG方案治疗急性难治性髓系白血病的临床效果。方法选择中国医科大学附属一院血液科内科2005-06~2007-11间,收治的27例难治性AML患者。
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Immunophenotype and dna ploidy could give a scientific laboratory basis for diagnosis, selection of treatment and prediction of prognosis.
dna倍体分析可为急性白血病的临床诊断提供客观的实验室依据。
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Mercaptopurine has been a standard component of long-term continuing treatment for childhood lymphoblastic leukaemia, whereas 6-thioguanine has been mainly used for intensification courses.
背景6-巯基嘌呤是治疗儿童淋巴细胞白血病的一种标准的长期维持用药,而 6-硫鸟嘌呤则主要用于强化治疗。
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The pathogenesis of CMPD is not clear except chronic myeloid leukemia associated with the bcr/abl fusion gene.
除慢性髓系白血病的发病与bcr-abl融合基因有关外,其他CMPD的发病机制仍不清楚。
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Jude Children's Research Hospital in Memphis, Tenn., for acute lymphoblastic leukemia.
Jude 小孩研究医院里2169名接受治疗的患急性成淋巴细胞白血病的儿童和青少年。
- 推荐网络例句
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This one mode pays close attention to network credence foundation of the businessman very much.
这一模式非常关注商人的网络信用基础。
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Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.
扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。
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There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.
双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。