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白血病

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The less of E-cadherin in leukemia cells may result in β-catenin translocating to the nu-clear and transcriptional activation of its target genes.

白血病细胞中E-cadherin表达降低或缺失可引起β'-catenin从细胞膜释放并进入细胞核,最终可能会引起β-catenin靶基因的转录。

Methods: The effect of ICA on the telomerase activity and proliferation and differentiation of HL-60 cells was compared with ATRA and valuated in vivo and in vitro.

采用ICA与全反式维甲酸对比试验,观察ICA对白血病细胞端粒酶活性及增殖分化的影响。

Other studies winnowed out abnormal DNA in lung adenocarcinoma tumors and acute myeloid leukemia.

其它研究把肺腺癌肿瘤和急性骨髓性白血病中的异常DNA挑了出来。

Other studies winnowed out abnormal DNA in lung adenocarcinoma tumors and acute myeloid leukemia

其他的研究筛选出了肺腺癌肿瘤和急性髓细胞性白血病中的异常DNA。

About 50 percent of all blood cancers develop in patients older than 60 years.

约有50%的白血病患者在60岁以后发病。

Objective: To determine effects of berbamine on the growth of leukemia cell line NB4 and explore its possible mechanisms.

目的:探讨小檗胺对白血病细胞系NB4生长的影响及其机制。

To explore the mechanism of berbamine inducing apoptosis in human leukemia Jurkat cells.

研究小檗胺诱导人白血病Jurkat细胞凋亡的机制。

[Purpose] To explore the mechanism of berbamine inducing apoptosis in human leukemia Jurkat cells.

导读:[目的]研究小檗胺诱导人白血病Jurkat细胞凋亡的机制。

So, berbamine could significantly induce apoptosis in those four kinds of leukemic cells in a time-and-concentration-dependent manner in vitro.

以上结果表明,小檗胺除对已报道的HL〓细胞外,还能显著地诱导其它类型的白血病细胞凋亡(如K〓、Jurkat、NB〓细胞),并呈明显的浓度-时间依赖关系。

Berbamine can selectively induce apoptosis of human leukemia Jurkat cells in the manner of dose and time dependence through up-regulation expression level of caspase-3 protein.

结论小檗胺能激活caspase-3蛋白表达,诱导人白血病Jurkat细胞凋亡且呈时效关系。

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This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。

There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.

双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。