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白血病

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High expression of cellular asparagine synthetase is a causative factor for the resistance of leukemic cell to Lasparaginase therapy.

细胞内门冬酰胺合成酶(asparagines synthetase, AS)的表达水平与白血病细胞对左旋门冬酰胺酶耐药有关。

BACKGROUND: Invasive aspergillosis is a leading cause of mortality in patients with acute leukemia.

背景:侵袭性曲霉菌病是导致急性白血病患者死亡的重要原因。

In the present study, the mechanisms of how EGCG enhanced ATO cytotoxicity in leukemia cells were investigated.

结果显示EGCG和三氧化二砷并用可以显著地诱导白血病细胞中过氧化氢和细胞凋亡的发生。

Science,1993,262:1274[8] Baffy G,Miyashita T,Williamson JR,et al.Apoptosis induced by withdrawal of interleukin-3 (IL-3) from an IL-3-dependent hematopoietic cell line is associated with repartitioning of intracellular calcium and is blocked by enforced BCL-2 oncoprotein production.

本实验观察到11例未缓解、难治复发急性白血病均呈BCL-2mRNA表达阳性,而9例完全缓解患者中仅2例为BCL-2mRNA表达阳性(P.01),结果支持BCL-2介导使细胞逃脱生理性凋亡、抵抗机体自然防御与临床治疗的机制,并与Campos等的报道类似[3]。

BAL in the diagnosis and treatment are different from a single type of leukemia.

BAL的诊断和治疗均不同于单一类型的白血病

At present, we have found about the same period of BAL in patients with the diagnosis of acute leukemia of 9.6%.

目前,我们发现BAL患者约占同期诊断的急性白血病的9.6%。

This little boy developed leukeamia and turned out bald as a coot for the side-effect of chemotherapy.

这个小男孩得了白血病,他因为化疗的副作用掉光了头发。

The boy developed leukaemia,and he turned out a bald as a coot because of the side-effect of cnemotherapy.

这个小男孩得了白血病,他因为化疗的副作用掉光了头发。

Objective To investigate the effect of oleanolic acid on the expression of bcl-2 and bax gene in HL60 cells.

目的 探讨齐墩果酸对人急性早幼粒细胞白血病HL60细胞bcl-2和bax基因表达的影响。

To investigate the clinical significance and the expression of bcl-2 and bax gene in acute leukemia.

目的:探讨抗凋亡基因Bcl-2及其拮抗基因Bax在急性白血病中的表达及临床意义。

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This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。

There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.

双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。