白细胞形成

Beijing 100034,China(The School of Oncology,Beijing Institute for Cancer Research,Beijing Medical University,Beijing 100034,China)(The School of Oncology,Beijing Institute for Cancer Research,Beijing Medical University,Beijing 100034,China)Abstract : Objective In order to study the effects of ATP on U937 cells. Methods Cell culture,flow cytometry,HE staining,Nigrosine staining and electron microscopy were used.

应用ATP(0.23g/L)作用于人白血病细胞U937，(1)用白细胞计数器每日计数细胞和计算抑制率；(2)用流式细胞分析仪文献检测 ATP对U937细胞周期改变和凋亡的影响；(3)苏木精-伊红染色和电子显微镜文献检测凋亡小体的形成过程；(4)用苯胺黑染色文献检测凋亡小体的生命本性。

Methods The formation of mouse-tail scale epidermis were used as the epidermis parakeratosis model,different groups of mouse were treated with rhIL-11or methotrexate.

采用小鼠尾部鳞片表皮颗粒层形成模型，不同组别的小鼠给以白细胞介素-11或甲氨蝶呤，观察其对鼠尾鳞片表皮颗粒层和血清IL-2含量的影响。

BACKGROUND: The natural killer cells at the site of placentation express killer-cell immunoglobulin-like receptors that can bind to human leukocyte antigen-C molecules on trophoblast cells.

背景：自然杀伤细胞在胎盘形成时表达伤细胞免疫球蛋白样受体，它可以在滋养层细胞上与人白细胞抗原-C分子结合。

Fibrin exudation, pulmonary interstitial incrassation, thrombosis of pulmonary artery, hemorrhage and gore were found. Conclusion: MR enhanced pulmonary perfusion can diagnose pulmonary embolism.

病理显示：梗死区可见大量的纤维素渗出，肺间质增厚，动脉腔内可见红细胞和血栓形成，其周围白细胞浸润伴肺淤血，部分可见点状出血及肺淤血。

Leucocytic differentiation antigen 40 sign-al access participates in regulation of inflammatory reaction accommodation of major cell component (vascular endothelial cell、vascular smooth muscle cell and macrophage), it is playing critical role in AS process, participates ather-osclerotic occurrence and development ,reside in atherosclerotic plaque cell thro-ugh CD40-CD40L interaction, induce activate by itself , express and secrete profit to happen immune response、inflammatory reaction、hemagglutination t-hrombogenic proteo-cytokine.

白细胞分化抗原40 (CD40)信号通路参与了动脉粥样硬化斑块内主要细胞成分(血管内皮细胞、血管平滑肌细胞以及巨噬细胞)炎症反应的调节，其在AS进程中扮演着关键性的角色，参与了动脉粥样硬化的发生和发展；存在于AS斑块内的细胞通过CD40-CD40L相互作用，导致自身活化，表达和分泌了有利于免疫应答发生、炎症反应、血凝和血栓形成的蛋白质细胞因子，如激活在AS斑块中的关键性细胞成分如黏附分子、炎症因子、基质金属蛋白酶等的产生，导致斑块的不稳定和破裂，而最终导致粥样硬化斑块病变的恶化。

Methods Male Sprague Dawley rats were induced by ligating the anterior descending coronary artery and were divided into the 2nd, 4th, 8th, 12th and 21st week groups. Elastic fibers were evaluated in the infarct region using Masson trichrome staining, Uana orcein staining and image analysis. The expressions of TNFα and IL-1β were also detected by immunohistochemistry.

SD大鼠冠状动脉前降支结扎形成心肌梗死模型，于术后2、4、8、12、21周时，用Masson染色、Uana地衣红染色技术和免疫组织化学方法，分别观察梗死区弹性纤维和白细胞介素1β(IL-1β)、肿瘤坏死因子的变化，计算机图像分析系统作半定量分析。

The necrotic epithelium becomes embedded in exuding fibrin and red and white cells,so that a grayish pseudomembrane is formed-commonly over the tonsils,pharynx or larynx.

坏死的上皮细胞被纤维蛋白、红细胞和白细胞凝聚在一起形成灰白色的假膜，通常覆盖在扁桃腺、咽或喉。

Bisexual worms were perfused out from the liver of infected mice between 10 minutes and 48 hours after medication. Tegumental surface alternations were observed and became rather apparent 1～2 hours after medication. Major surface alterations included pronounced swelling, wrinkling, tough junction and fu...

结果表明，干给小鼠药后10分钟，两性血吸虫皮层即有变化，1～2小时后已十分明显，主要变化为水肿，皮层褶嵴紧密连接、融合，形成大量球状物或小泡，感觉器肿大、破溃，以及皮层糜烂、剥落和白细胞附着于皮层损害处等。

In the experiment cell viability of HUVEC and PMBC were assessed by MTT assay, survival rate of PMN was assessed by trypan blue staining.

其中白细胞与内皮细胞的粘附在血栓形成过程中具有重要意义，是启动缺血后炎性损伤的关键。

It also reduced the formation of platelet-leukocyte conjugates,both in unstimulated and adenosine diphosphate-stimulated bloodsamples at 3%–4% end-expiratory sevoflurane concentrationsused to maintain anesthesia.

七氟醚呼末浓度为3％－4％时无论用还是不用二磷酸腺苷刺激的血标本中血小板－白细胞结合物的形成均减少。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。