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癫痫

与 癫痫 相关的网络例句 [注:此内容来源于网络,仅供参考]

Although patients who were stuporous or confused (vs. comatose) improved more often on AEDs, they were less often realized to be in SE before the EEG.

虽然那些昏睡或混淆患者抗癫痫药物治疗往往有效,但是在脑电图诊断之前很少会意识到他们已经处于癫痫持续状态。

Objective To evaluate the effect of a new localization mode and the effects of lesionectomy and/or multiple subpial transectionunder monitoring of intra-operative ECoG recordings for treatment of intractable epilepsy.

目的评价难治性癫痫的致痫灶定位方法和皮层电极监测下致痫灶切除,加行多处软脑膜下横纤维切断术治疗癫痫的疗效。

Syncope must be differentiated from epileptic seizures, although seizures due to brain hypoxia can occur in a syncopal episode.

晕厥必须与癫痫发作相鉴别,尽管在晕厥发作时也会发生脑缺氧引起的癫痫发作。

ACET inhibit the descending of Bax,prolong its descending period andthen promote its expression,in order to weaken the inhibition of the apoptosis during 4 to 10 days after seizure and then promote the apoptosis during 10 to 14 days after.

埋线治疗通过抑制Bax蛋白表达的下降、延长其降至低峰值的时程,致使在癫痫发作后的中期(4d~10d)抑制大鼠海马神经元凋亡的作用减弱;继而通过促进Bax蛋白表达的回升,致使在癫痫发作后的远期(10d~14d)出现促进凋亡的作用。

The dynamic changes of the the neuron apoptosis and correlative genes in hippocampus of ACET-groups and twice-ACET-groups suggested that ACET can slower the apoptosis by inhibiting p~(53)and prolong the ascending period of p~(53) in 4 days after the seizure,this effect would weaken after 4 days.ACET may inhibit the descending of Bax,prolong its descending period and then promote its expression,in order to weaken the inhibition of the apoptosis during 4 to 10 days after seizure and then promote the apoptosis during 10 to 14 days after.This effect of ACET can be enhanced after treating again within 7 to 14 days later.

埋线组和二次埋线组大鼠海马神经元凋亡及相关基因表达的动态变化结果提示:埋线治疗在癫痫发作后的早期(4d内)可通过抑制促凋亡基因p~(53)蛋白表达、延缓其达峰值的时程,从而减缓神经元凋亡的发生,此作用在4d后减弱;埋线治疗抑制大鼠海马神经元凋亡的作用在癫痫发作后的中期(4d~10d)减弱,甚至在远期(10d~14d)出现促进凋亡的作用,可能是通过抑制Bax蛋白表达的下降、延长其降至低峰值的时程,促进Bax蛋白表达的回升的结果;在中远期(7d~14d)内予二次埋线治疗对该效应有续效叠加的作用。

The epileptic model has been successfully established by kainic acid microinjected into nucleus amygdalae in Wistar male rats, this model is applicable for temporal lobeepilepsy.

采用杏仁核海人酸微量注射方法成功建立了Wistar大鼠点燃癫痫模型,该模型适用于颞叶癫痫的相关研究。

Objective To observe the morphology of neuronal death in nucleus amygdalae of rats after status epilepticus induced by lithium-pilocarpine, and correlate this with markers of DNA fragmentation that has been associated with cellular apoptosis. MethodsSE was induced for 3 hours with LPC.24 or 48 hours later the rats were killed.

目的 观察锂-匹鲁卡品致痫鼠杏仁核神经元损伤的形态学改变,并检测细胞凋亡相关的DNA碎片标志物方法用LPC诱发大鼠癫痫持续状态(status epilepticus,SE)3 h,于癫痫中止后第24小时和第48小时处死动物。

She suffers from epilepsy , and doctors ' on are monitoring her seizures in a the hope of preforming performing an operation to minimize them.

她饱受癫痫之苦,医生希望通过手术来控制癫痫的发作,使之降到最低。

7 The mechanism is not known, but a brief convulsion neither presages epilepsy nor requires the administration of anticonvulsant medication.

其机制还不清楚,但是一次短暂的抽搐既不是癫痫的先兆,也无需要使用抗癫痫药。

Ammonium oxalate may obviously inhibit the frequency, scope and duration of the epileptic discharge. It has obviously effects on protecting neurons in hippocampus of epilesy rats caused by penicillin.

草酸铵能抑制癫痫大鼠的痫样放电频率、幅度和总时间,能明显抑制海马区锥体层细胞坏死,对癫痫所致的脑部损伤有缓解作用。

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