症状学的

Perhaps most people are now persuaded that it is possible for a man to have volitional control of his muscles and also to know the physical character of his movements and their consequences for himself and others, and yet be unable to resist the urge or temptation to perform a certain act ; yet many think this incapacity exists only if it is associated with well-marked physiological or neurological symptoms or independently definable psychological disturbances .

也许，大部分人现在相信，对于一个人来说，这样的情况是可能的，即对其肌肉有自愿的控制，并且也知道他的运动及其对于他自身与他人的后果的物理特性，但却无法抵制实施某一特定的行为的迫切要求或诱惑，（这样的情况是可能的，即一个人可以对其肌肉进行自主的控制，并且也明白自己的动作的物理特征以及对于自己和他人所可能产生的后果，但就是抵制实施某一特定的行为的迫切要求或诱惑；）而且，许多人认为，只有其与得到了适当标示的生理学的或神经学上的症状或者可单独界定的生理错乱相联系，才存在这种无能。

Finally statistical analysis is conducted.Result: Orally taking Xuefu Zhuyu Tang and calming-wind medicines can ease stenocardia,improve ECG,enhance the stopped rate of nitroglycerin,better the heart contraction and dilation,regulate blood fat,promote blood rheology,lower CRP、ET、TXB2,raise NO、6-keto-PGF1αand so on,compired with the control group,it is obviously statically meaningful without obvius adverse reaction.

结果：血府逐瘀汤加治风活络药物内服治疗能有效地缓解心绞痛和临床症状；改善心电图；提高速效扩冠药的停减率；改善心脏的收缩及舒张功能；调节血脂；改善血液流变学；降低CRP、ET、TXB2的水平，升高NO、6-keto-PGF1α的水平等，与对照组相比，有显著的统计学意义，且未发现明显的不良反应。

There are many complex factors, such as age, environmental, social, psychological, personal factor and human diseases, etc., affecting the development of insomnia symptoms. Insomnia is one kind of neurology/psychiatry diseases, due to brain overstrains and stresses in the nerve center system, in Western Medicine, while there are approximately two factors that can cause the insomnia symptoms, including the anti-pathogenic- qi deficiency and the pathogenic-factors invasion in Traditional Chinese Medicine.

失眠的原因有年龄、环境、社会心理、身体疾病等繁多复杂因素所造成，西医学认为失眠是由大脑压力所造成脑与神经所支配命令的自律神经系统降低或失去其调整身体功能作用之中枢神经系统失调的疾病症状，然而，中医学的失眠病因病机可概括为正虚、邪扰两种，其中正气虚涉及心、脾、肝、胆、肾、胃等脏腑，邪气扰则以痰、热、火、饮、食为多，失眠的病因主要有脏腑虚损、精血不足、神魂失养、七情所伤、内邪滞扰、外邪所感、思虑劳倦太过或暴受惊恐，亦可因禀赋不足、房劳久病或年迈体虚等所致；失眠的主要病机在於阴阳及气血失和，脏腑功能失调，进而神明被扰、神不安舍所致。

This project not only illuminates involvement of iron in the etiology of PD, but also gives some clues for the cure of Parkinson''s disease by studying the effect of iron chelators. This may be helpful for us to fully understand PD.

本研究为脑内高铁参与PD的病因学提供了新的实验依据，而且通过应用螯合脑内特殊部位过量铁并改善症状的铁螯合剂，为神经退行性疾病的防治开拓一条新的研究领域和可能的干预措施。

The latedischarges decreased from 9.29 ± 0.97 to 6.71 ± 0.68 with the A-fiberconditioning stimulus increasing from 1 to 5 (n〓8, P〓0. 05).(7) The intervalbetween the conditioning stimulus and test stimulus (C-T interval) wasincreasing, the inhibition tended to plateau off. At shorter time intervalsthe inhibition became more effective. When C-T interval was limited in 50ms,the inhibitory effects was the strongest, here, the late discharges reducedfrom 12.57±1.21to 2.29±0.42 (n=11, P＜0. 01).(8) Behavior research showedthat the rat model of snake venom exhibited neuropathic pain with heathyperalgesia, cold and mechanical allodynia, which corresponding to the acuteelectrophysiological findings.

此时轻刷WDR神经元的感受野不能引起其活动改变，但伤害性齿镊夹捏仍可引起WDR神经元放电增多；〓5〓晚成分放电的潜伏期缩短，即宁静期的时程变短，由给蛇毒前的118.83〓3.67ms降至50.72〓1.36ms〓n〓32，P〓0.01〓；〓6〓在正常动物，如果预先给予只激活A纤维的弱条件电刺激〓mA，100μs〓可抑制随后的伤害性检验刺激所诱发的WDR神经元的晚成分放电，当条件刺激个数从1增加至5时，每次伤害性检验刺激所诱发的晚成分放电数从9.29〓0.97个降至6.71〓0.68个〓n〓8，P〓0.05〓；〓7〓固定条件刺激数为1个，当条件刺激与检验刺激之间的间隔增大时，A纤维条件刺激对WDR神经元晚成分放电的抑制作用逐渐减弱，当条件刺激与检验刺激之间的间隔在50 ms以内时，抑制效应最为显著，此时，晚成分放电数由正常时的12.57〓1.21个降至2.29〓0.42个〓n〓11，P〓0.01〓；〓8〓与急性研究中的WDR神经元电活动的变化结果相匹配，利用蛇毒制备的大鼠模型在行为学上表现为热痛觉过敏、冷觉的痛性感觉异常及机械痛觉过敏等慢性痛症状。

At the same time, most WDR neurons failedto respond to the light brush applied to the receptive fields, but they couldbe intensively excited by the noxious pinch.(5) The latency of the latedischarges was shortened from 118.83 ± 3.67ms to 50.72 ± 1.36ms (n〓32, P〓0. 01).(6) Preceding graded number of A〓fiber conditioning inputs (〓mA, 100 μs) delayed the C-activity evoked by the following nociceptive teststimulus activating both A- and C-fiber applied to the sciatic nerve. The latedischarges decreased from 9.29 ± 0.97 to 6.71 ± 0.68 with the A-fiberconditioning stimulus increasing from 1 to 5 (n〓8, P〓0. 05).(7) The intervalbetween the conditioning stimulus and test stimulus (C-T interval) wasincreasing, the inhibition tended to plateau off. At shorter time intervalsthe inhibition became more effective. When C-T interval was limited in 50ms,the inhibitory effects was the strongest, here, the late discharges reducedfrom 12.57±1.21to 2.29±0.42 (n=11, P＜0. 01).(8) Behavior research showedthat the rat model of snake venom exhibited neuropathic pain with heathyperalgesia, cold and mechanical allodynia, which corresponding to the acuteelectrophysiological findings.

此时轻刷WDR神经元的感受野不能引起其活动改变，但伤害性齿镊夹捏仍可引起WDR神经元放电增多；〓5〓晚成分放电的潜伏期缩短，即宁静期的时程变短，由给蛇毒前的118.83〓3.67ms降至50.72〓1.36ms〓n〓32，P〓0.01〓；〓6〓在正常动物，如果预先给予只激活A纤维的弱条件电刺激〓mA，100μs〓可抑制随后的伤害性检验刺激所诱发的WDR神经元的晚成分放电，当条件刺激个数从1增加至5时，每次伤害性检验刺激所诱发的晚成分放电数从9.29〓0.97个降至6.71〓0.68个〓n〓8，P〓0.05〓；〓7〓固定条件刺激数为1个，当条件刺激与检验刺激之间的间隔增大时，A纤维条件刺激对WDR神经元晚成分放电的抑制作用逐渐减弱，当条件刺激与检验刺激之间的间隔在50 ms以内时，抑制效应最为显著，此时，晚成分放电数由正常时的12.57〓1.21个降至2.29〓0.42个〓n〓11，P〓0.01〓；〓8〓与急性研究中的WDR神经元电活动的变化结果相匹配，利用蛇毒制备的大鼠模型在行为学上表现为热痛觉过敏、冷觉的痛性感觉异常及机械痛觉过敏等慢性痛症状。

In the United States, chronic alcoholism and hepatitis C are the most common ones.

在美国，慢性酒精中毒，肝炎是最常见的。

If you have any questions, you can contact me anytime.

如果有任何问题，你可以随时联系我。