病理学

Methods 42 death cases of cardiovascular system disease with trauma were autopsied systematically and the sections are observed by H.

方法42例心血管系统疾病伴损伤死亡案例均接受系统病理学解剖和组织学观察，且死因明确。

Methods 42 death cases of cardiovascular system disease with trauma were autopsied systematically and the sections are observed by H.

方法42例心血管系统疾病伴损伤死亡案例均接受系统病理学解剖和组织学观察，且死因明确。结果42例中40岁以上者占到64.28%，男性较女性死者多。擦伤和挫伤在损伤中比例最大共占到80.95%，冠心病和高血压病死亡者各占45.24%、16.67%。

The EI of different concentrations (7%vol, 8%vol, 9%vol) were iv. infused. The minimum induction dose, the dose of Eisol%, MAC, the cardiac anesthetisia index and the maximal tolerant concentration of end-tidal isoflurane were determined. The animals were autopsied after dead and the main organs were histo-pathologically diagnosed as well.

分别静脉注射7％vol、8％vol、9％volEI测定EI静脉给药的最小诱导剂量、使呼气末浓度达1％的累加用药量DE_(iso1％，MAC以及心脏麻醉指数和最大耐受末呼气浓度，动物死亡后即刻取重要脏器送病理学检查。

Some exudation and bleedings happen in the pulmonary alveoli, some inflammatory cells infiltrate into lung tissues; in the serum AMY, ALP, TNF-α, IL-6 increases obviously high, COX-2 expression increase; The expression of NF-κB in big rat lung of SAP increase, and the nucleus is thick dyed, it explain that the NF-κ moved into the nucleus; Stimulate the inflamatory reaction.

我们的实验中见到SAP组的大鼠肺脏组织细胞腔扩大、肺泡壁断裂，有的出现肺大泡，肺泡内伴有渗出及少量非出血，肺组织可见咽细胞浸润；血清中AMY、ALP、TNF-α、IL-6明显增高；在SAP组大鼠肝细胞、肾小管上皮细胞、肺脏小气管及肺泡上皮上COX-2表达增加；NF-κB在SAP大鼠肺脏中表达增加，并且出现核浓染，说明NF-κB前移入核；启动炎症反应等一系列反应SAP大鼠受损伤的肺组织中AQP-1无论在mRNA水平上、蛋白水平上还是免疫组化染色病理学检测上都明显下降。

Six and twelve boors after sepsis, leptin levels in liver homogenate were detected by radioimmunoassay serum alanine aminotransferase and 4 enzymes in liver homogenate. myeloperoxidase, glutathin-S-transferase, xanthine oxidase and superoxide dismutase, all related with synthesis of free radicals, detoxication and purine metabolism, were detected by 96 well spectrophotometry. H-E staining was used to examine the histopathologic changes of liver.

于脓毒症后6 h和12 h，采用放射免疫法测量肝组织匀浆液中Leptin水平，采用96孔分光光度法检测血清丙氨酸氨基转移酶及髓过氧化物酶、谷胱甘肽－S转移酶、黄嘌呤氧化酶、超氧化物歧化酶等4种与自由基合成、解毒和嘌呤生成代谢相关的酶的水平，同时以H-E染色法观察肝组织病理学改变。

Brain contusions and traumatic vessel lesions areimportant pathological foundations of cerebral hemorrhage and primary causes of DTICH.

脑挫伤和外伤性脑内血管病变是脑出血的重要病理学基础，是迟发性外伤性脑内血肿的主要原因。

W. . The levels of myocardial troponin I, creatin kinase, aspartate transamnaseand lactatede hydrogenasewere detected before and per 30 mins during the experiment, and the changes of rabbit myocardial pathology were analyzed.

分别于实验前和实验中每隔30分钟抽取血液，以肝素抗凝并分析心肌肌钙蛋白I浓度，肌酸激酶、门冬氨酸氨基转移酶和乳酸脱氢酶活性，同时观察心肌组织的病理学变化。

Methods The rats were divided into model group（only CCl4 hypodermic）,curcumine treatment group（CCl4 hypodermic and curcumine injected to abdominal cavity in low,medium and high doses）and normal group.The pathologic changes were observed by light and electron microscope.

将大鼠分为模型组（单纯皮下注射CCl4），低、中、高剂量姜黄素治疗组（皮下注射CCl4的同时，给予不同浓度的姜黄素腹腔注射）及正常对照组，应用光镜、电镜观察肝细胞的病理学变化。

Light microscope findings shown that the impairment in TyrphostinA1 groups was far less severe than that in model groups. In conclusion, Curcumine and TyrphostinA1 showed preferable effect on acute lung injury induced by oleic acid in rats.

结果表明，TyrphostinA1明显改善大鼠肺功能，减轻肺组织病理学损伤，降低肺指数及肺湿/干重比，降低肺渗透性和肺泡灌洗液中蛋白及TNF-α、IL-6的含量，提高肺泡灌洗液中IL-10的含量。

It delicates that it is necessary to improve the quality of illustration and strengthen its application in pathology teaching.

结论有必要提高教材插图的质量并增加其在病理学教学中的应用。

Do you know, i need you to come back

你知道吗，我需要你回来

Yang yinshu、Wang xiangsheng、Li decang,The first discovery of haemaphysalis conicinna.

1〕 杨银书，王祥生，李德昌。安徽省首次发现嗜群血蜱。